Mechanism of NF-kappaB inactivation induced by survival signal withdrawal in cerebellar granule neurons (Q44963281)

7 December 2017

title

Mechanism of NF-kappaB inactivation induced by survival signal withdrawal in cerebellar granule neurons (English)

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7 December 2017

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Attila D Kovács

1

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7 December 2017

Shikha Chakraborty-Sett

2

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7 December 2017

Servio H Ramirez

3

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7 December 2017

Lynn F Sniderhan

4

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7 December 2017

Andrea L Williamson

5

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7 December 2017

Sanjay B Maggirwar

6

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7 December 2017

language of work or name

English

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publication date

1 July 2004

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European Journal of Neuroscience

7 December 2017

published in

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7 December 2017

volume

20

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7 December 2017

issue

2

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7 December 2017

page(s)

345-352

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Control of apoptosis by Rel/NF-kappaB transcription factors

7 December 2017

cites work

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The Rel/NF-kappaB family directly activates expression of the apoptosis inhibitor Bcl-x(L).

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Duration of nuclear NF-kappaB action regulated by reversible acetylation

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Acetylation of RelA at discrete sites regulates distinct nuclear functions of NF-kappaB.

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Missing pieces in the NF-kappaB puzzle

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Nuclear factor-kappaB mediates the cell survival-promoting action of activity-dependent neurotrophic factor peptide-9

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The anti-apoptotic activities of Rel and RelA required during B-cell maturation involve the regulation of Bcl-2 expression

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NF-kappaB is involved in the survival of cerebellar granule neurons: association of IkappaBbeta [correction of Ikappabeta] phosphorylation with cell survival

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Selective and AMPA receptor-dependent astrocyte death following prolonged blockade of glutamate reuptake in rat cerebellar cultures

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Autoradiographic localization and depolarization-induced release of acidic amino acids in differentiating cerebellar granule cell cultures

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Tumor necrosis factor induces Bcl-2 and Bcl-x expression through NFkappaB activation in primary hippocampal neurons

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NFkappaB activation is required for the neuroprotective effects of pigment epithelium-derived factor (PEDF) on cerebellar granule neurons

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21 January 2018

Identifiers

DOI

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7 December 2017

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