Increased urinary Smad3 is significantly correlated with glomerular hyperfiltration and a reduced glomerular filtration rate and is a new urinary biomarker for diabetic nephropathy (Q36142232)

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Language	Label	Description	Also known as
English	Increased urinary Smad3 is significantly correlated with glomerular hyperfiltration and a reduced glomerular filtration rate and is a new urinary biomarker for diabetic nephropathy	scientific article

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scholarly article

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

Increased urinary Smad3 is significantly correlated with glomerular hyperfiltration and a reduced glomerular filtration rate and is a new urinary biomarker for diabetic nephropathy (English)

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

0 references

author name string

Kaifeng Guo

1

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

Junxi Lu

2

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

Jingxin Kou

3

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

Mian Wu

4

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

Lei Zhang

5

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

Haoyong Yu

6

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

Mingliang Zhang

7

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

Yuqian Bao

8

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

Haibing Chen

9

1 reference

Europe PubMed Central

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12 August 2017

Weiping Jia

10

1 reference

Europe PubMed Central

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12 August 2017

publication date

8 October 2015

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

16

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

159

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

New insights into molecular mechanisms of diabetic kidney disease

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4599589

2 October 2017

Molecular mechanisms in the pathogenesis of diabetic nephropathy: an update

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4599589

2 October 2017

Transforming growth factor-β/Smad signalling in diabetic nephropathy.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4599589

2 October 2017

A glimpse of various pathogenetic mechanisms of diabetic nephropathy

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4599589

2 October 2017

Hypertension in diabetic nephropathy: epidemiology, mechanisms, and management

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4599589

2 October 2017

Blockade of endothelial-mesenchymal transition by a Smad3 inhibitor delays the early development of streptozotocin-induced diabetic nephropathy

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4599589

2 October 2017

Essential role for Smad3 in angiotensin II-induced tubular epithelial-mesenchymal transition.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4599589

2 October 2017

The clinical significance of hyperfiltration in diabetes

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4599589

2 October 2017

Advanced glycation end-products induce tubular CTGF via TGF-beta-independent Smad3 signaling

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4599589

2 October 2017

Urinary peptidome may predict renal function decline in type 1 diabetes and microalbuminuria

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4599589

2 October 2017

CTGF and chronic kidney fibrosis

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4599589

2 October 2017

Nonalbuminuric renal impairment in type 2 diabetic patients and in the general population (national evaluation of the frequency of renal impairment cO-existing with NIDDM [NEFRON] 11).

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4599589

2 October 2017

Is hyperfiltration associated with the future risk of developing diabetic nephropathy? A meta-analysis.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4599589

2 October 2017

Insights into the regulation of renal hemodynamic function in diabetic mellitus

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4599589

2 October 2017

The differential role of Smad2 and Smad3 in the regulation of pro-fibrotic TGFbeta1 responses in human proximal-tubule epithelial cells.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4599589

2 October 2017

TGF-beta induces proangiogenic and antiangiogenic factors via parallel but distinct Smad pathways

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4599589

2 October 2017

Mediators of diabetic renal disease: the case for tgf-Beta as the major mediator

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4599589

2 October 2017

Smad pathway is activated in the diabetic mouse kidney and Smad3 mediates TGF-beta-induced fibronectin in mesangial cells

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4599589

2 October 2017

Prevention of diabetic renal disease with special reference to microalbuminuria

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4599589

2 October 2017

Predominance of hemodynamic rather than metabolic factors in the pathogenesis of diabetic glomerulopathy

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4599589

2 October 2017

Prevention of diabetic glomerulopathy by pharmacological amelioration of glomerular capillary hypertension

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4599589

2 October 2017

Correlation of high urinary Smad1 level with glomerular hyperfiltration in type 2 diabetes mellitus

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4599589

23 September 2018

Glomerular hyperfiltration: a new marker of metabolic risk.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4599589

23 September 2018

BCL-2: pro-or anti-oxidant?

1 reference

https://api.crossref.org/works/10.1186%2FS12882-015-0156-8

21 January 2018

Course of renal function in type 2 diabetic patients with abnormalities of albumin excretion rate

1 reference

https://pubmed.ncbi.nlm.nih.gov/26449625

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Advanced glycation end products activate Smad signaling via TGF-beta-dependent and independent mechanisms: implications for diabetic renal and vascular disease

1 reference

https://pubmed.ncbi.nlm.nih.gov/26449625

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Renal renin-angiotensin system in diabetes: functional, immunohistochemical, and molecular biological correlations

1 reference

https://pubmed.ncbi.nlm.nih.gov/26449625

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Sequential alterations in glomerular prostaglandin and thromboxane synthesis in diabetic rats: relationship to the hyperfiltration of early diabetes

1 reference

https://pubmed.ncbi.nlm.nih.gov/26449625

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Urinary MCP-1 and RBP: independent predictors of renal outcome in macroalbuminuric diabetic nephropathy

1 reference

https://pubmed.ncbi.nlm.nih.gov/26449625

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

[Change and significance of urinary smad3 in type 2 diabetic nephropathy]

1 reference

https://pubmed.ncbi.nlm.nih.gov/26449625

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Urinary albumin excretion rate and glomerular filtration rate in the prediction of diabetic nephropathy; a long-term follow-up study of childhood onset type-1 diabetic patients

1 reference

https://pubmed.ncbi.nlm.nih.gov/26449625

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1186/S12882-015-0156-8

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

ResearchGate publication ID

0 references

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