Pages that link to "Q24311308"

The following pages link to Inactivation of the CYLD deubiquitinase by HPV E6 mediates hypoxia-induced NF-kappaB activation (Q24311308):

Displaying 50 items.

• tumor protein p53 (Q283350) (← links)
• DUBs, New Members in the Hypoxia Signaling clUb (Q26750882) (← links)
• Hypoxia Induced NF-κB (Q26752556) (← links)
• Squamous cell carcinomas of the lung and of the head and neck: new insights on molecular characterization (Q26765060) (← links)
• Oncogenes and RNA splicing of human tumor viruses (Q26830643) (← links)
• High-risk human papillomavirus targets crossroads in immune signaling (Q28084802) (← links)
• Papillomavirus E6 oncoproteins (Q30410553) (← links)
• The helicase protein DHX29 promotes translation initiation, cell proliferation, and tumorigenesis (Q33564231) (← links)
• Activating transcription factor 3 activates p53 by preventing E6-associated protein from binding to E6. (Q33800021) (← links)
• Human papillomavirus deregulates the response of a cellular network comprising of chemotactic and proinflammatory genes (Q33851664) (← links)
• Serum response factor controls CYLD expression via MAPK signaling pathway (Q33900624) (← links)
• The repressing function of the oncoprotein BCL-3 requires CtBP, while its polyubiquitination and degradation involve the E3 ligase TBLR1. (Q34045748) (← links)
• BCL-3 degradation involves its polyubiquitination through a FBW7-independent pathway and its binding to the proteasome subunit PSMB1. (Q34056023) (← links)
• Hypoxia suppresses cylindromatosis (CYLD) expression to promote inflammation in glioblastoma: possible link to acquired resistance to anti-VEGF therapy. (Q34227712) (← links)
• Viral oncogenes, noncoding RNAs, and RNA splicing in human tumor viruses (Q34394771) (← links)
• NF-κB-induced KIAA1199 promotes survival through EGFR signalling (Q34563758) (← links)
• Human papillomavirus (HPV) upregulates the cellular deubiquitinase UCHL1 to suppress the keratinocyte's innate immune response (Q34743768) (← links)
• Integrative and comparative genomic analysis of HPV-positive and HPV-negative head and neck squamous cell carcinomas (Q35008028) (← links)
• HPV16E6-dependent c-fos expression contributes to AP-1 complex formation in SiHa cells (Q35138105) (← links)
• NF-kappaB protects human papillomavirus type 38 E6/E7-immortalized human keratinocytes against tumor necrosis factor alpha and UV-mediated apoptosis. (Q35193057) (← links)
• Activation of NF-κB by human papillomavirus 16 E1 limits E1-dependent viral replication through degradation of E1. (Q35488924) (← links)
• Expression of enterovirus 71 virus-like particles in transgenic enoki (Flammulina velutipes). (Q35625893) (← links)
• Productive Lifecycle of Human Papillomaviruses that Depends Upon Squamous Epithelial Differentiation (Q35908359) (← links)
• Hypoxia attenuates the proinflammatory response in colon cancer cells by regulating IκB (Q36299669) (← links)
• Cellular transformation by human papillomaviruses: lessons learned by comparing high- and low-risk viruses (Q36989890) (← links)
• The ubiquitin-specific peptidase USP15 regulates human papillomavirus type 16 E6 protein stability (Q37333809) (← links)
• IκB kinase b Mediating the Downregulation of p53 and p21 by Lipopolysaccharide in Human Papillomavirus 16+ Cervical Cancer Cells (Q37447488) (← links)
• CYLD: a tumor suppressor deubiquitinase regulating NF-kappaB activation and diverse biological processes (Q37448141) (← links)
• The biological properties of E6 and E7 oncoproteins from human papillomaviruses. (Q37617163) (← links)
• Deubiquitinases in the regulation of NF-κB signaling (Q37814068) (← links)
• Expanding role of ubiquitination in NF-κB signaling (Q37816941) (← links)
• CYLD: a deubiquitination enzyme with multiple roles in cancer. (Q37846231) (← links)
• TNF and ubiquitin at the crossroads of gene activation, cell death, inflammation, and cancer (Q37948115) (← links)
• Genetic and microbial factors modulating the ubiquitin proteasome system in inflammatory bowel disease (Q38149113) (← links)
• Hypoxia activates IKK-NF-κB and the immune response in Drosophila melanogaster (Q38978198) (← links)
• Manipulation of the innate immune response by human papillomaviruses (Q39003287) (← links)
• The functional interplay between the HIF pathway and the ubiquitin system - more than a one-way road. (Q39185765) (← links)
• Mechanism of hypoxia-induced NF-kappaB. (Q39669340) (← links)
• The Interaction Between Human Papillomaviruses and the Stromal Microenvironment (Q40444191) (← links)
• HPV16 E2-mediated potentiation of NF-κB activation induced by TNF-α involves parallel activation of STAT3 with a reduction in E2-induced apoptosis (Q42218400) (← links)
• NF-κB potentiates tumor growth by suppressing a novel target LPTS. (Q42374167) (← links)
• Transcriptional downregulation of microRNA-19a by ROS production and NF-κB deactivation governs resistance to oxidative stress-initiated apoptosis (Q42378496) (← links)
• NFX1 plays a role in human papillomavirus type 16 E6 activation of NFkappaB activity (Q42411008) (← links)
• HPV E6 oncoprotein prevents recovery of stalled replication forks independently of p53 degradation (Q42734252) (← links)
• Pomegranate extract inhibits EMT in clear cell renal cell carcinoma in a NF-κB and JNK dependent manner. (Q52979075) (← links)
• [CYLD deubiquitinase as a recurrent target in oncogenic processes]. (Q53285080) (← links)
• Molecular mechanisms in progression of HPV-associated cervical carcinogenesis (Q64097188) (← links)
• HPV Oncoproteins and the Ubiquitin Proteasome System: A Signature of Malignancy? (Q89834860) (← links)
• DUBs, Hypoxia, and Cancer (Q91208644) (← links)
• The Role of Deubiquitinases in Oncovirus and Host Interactions (Q92511672) (← links)