Pages that link to "Q24629897"
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The following pages link to Physical exercise stimulates autophagy in normal skeletal muscles but is detrimental for collagen VI-deficient muscles (Q24629897):
Displaying 50 items.
- The beneficial role of proteolysis in skeletal muscle growth and stress adaptation (Q26750990) (← links)
- Mitochondrial Quality Control and Muscle Mass Maintenance (Q26770420) (← links)
- Interactions between Autophagy and Bacterial Toxins: Targets for Therapy? (Q26801464) (← links)
- Running forward: new frontiers in endurance exercise biology (Q26824141) (← links)
- Autophagy is essential to support skeletal muscle plasticity in response to endurance exercise (Q27000625) (← links)
- Autophagy modulation as a potential therapeutic target for diverse diseases (Q27010076) (← links)
- Cellular and molecular mechanisms of muscle atrophy (Q27012762) (← links)
- Role of autophagy in COPD skeletal muscle dysfunction (Q27015002) (← links)
- Intestinal Autophagy Improves Healthspan and Longevity in C. elegans during Dietary Restriction (Q27308697) (← links)
- Autophagy is required for exercise training-induced skeletal muscle adaptation and improvement of physical performance (Q30410425) (← links)
- Exercise training-induced regulation of mitochondrial quality (Q30418478) (← links)
- How to explain exercise-induced phenotype from molecular data: rethink and reconstruction based on AMPK and mTOR signaling (Q30729568) (← links)
- Activating Autophagy by Aerobic Exercise in Mice (Q33613673) (← links)
- Exercise-induced skeletal muscle remodeling and metabolic adaptation: redox signaling and role of autophagy (Q33722890) (← links)
- Haptoglobin is required to prevent oxidative stress and muscle atrophy (Q33800519) (← links)
- S-nitrosoglutathione reductase deficiency-induced S-nitrosylation results in neuromuscular dysfunction. (Q33862264) (← links)
- Mitochondrial dysfunction and defective autophagy in the pathogenesis of collagen VI muscular dystrophies. (Q34338854) (← links)
- Heat shock response and autophagy--cooperation and control (Q34464470) (← links)
- Muscle wasting and cachexia in heart failure: mechanisms and therapies (Q34681621) (← links)
- Can endurance exercise preconditioning prevention disuse muscle atrophy? (Q35167280) (← links)
- Role of PGC-1α during acute exercise-induced autophagy and mitophagy in skeletal muscle (Q35572598) (← links)
- Mitochondrial quality control: Easy come, easy go (Q35850525) (← links)
- Autophagy is not required to sustain exercise and PRKAA1/AMPK activity but is important to prevent mitochondrial damage during physical activity (Q35852789) (← links)
- Autophagy impairment in muscle induces neuromuscular junction degeneration and precocious aging. (Q35947145) (← links)
- Effects of long-term resistance exercise training on autophagy in rat skeletal muscle of chloroquine-induced sporadic inclusion body myositis (Q36218623) (← links)
- Acute Exercise-Induced Mitochondrial Stress Triggers an Inflammatory Response in the Myocardium via NLRP3 Inflammasome Activation with Mitophagy (Q36386652) (← links)
- Transcriptional Regulation by Nuclear Corepressors and PGC-1α: Implications for Mitochondrial Quality Control and Insulin Sensitivity (Q36470524) (← links)
- PGC-1α promotes exercise-induced autophagy in mouse skeletal muscle (Q36594146) (← links)
- The regulation of autophagy during exercise in skeletal muscle (Q36698649) (← links)
- Lysosomal calcium signalling regulates autophagy through calcineurin and TFEB. (Q36709792) (← links)
- The emerging role of skeletal muscle oxidative metabolism as a biological target and cellular regulator of cancer-induced muscle wasting (Q36901945) (← links)
- Autophagy is induced in the skeletal muscle of cachectic cancer patients (Q37128604) (← links)
- The influence of skeletal muscle on systemic aging and lifespan (Q37337242) (← links)
- Nutrient ingestion increased mTOR signaling, but not hVps34 activity in human skeletal muscle after sprint exercise (Q37344096) (← links)
- 212th ENMC International Workshop: Animal models of congenital muscular dystrophies, Naarden, The Netherlands, 29-31 May 2015. (Q37555754) (← links)
- "Get the Balance Right": Pathological Significance of Autophagy Perturbation in Neuromuscular Disorders (Q37610585) (← links)
- AMP-activated protein kinase: new regulation, new roles? (Q38019034) (← links)
- Mitochondrial biogenesis and fragmentation as regulators of protein degradation in striated muscles (Q38035636) (← links)
- Protein breakdown in muscle wasting: role of autophagy-lysosome and ubiquitin-proteasome (Q38105829) (← links)
- The role of mitochondrial dysfunctions due to oxidative and nitrosative stress in the chronic pain or chronic fatigue syndromes and fibromyalgia patients: peripheral and central mechanisms as therapeutic targets? (Q38120080) (← links)
- Skeletal muscle, autophagy, and physical activity: the ménage à trois of metabolic regulation in health and disease. (Q38165741) (← links)
- Autophagic cellular responses to physical exercise in skeletal muscle. (Q38189267) (← links)
- The many roles of PGC-1α in muscle--recent developments (Q38190176) (← links)
- Autophagy--a key player in cellular and body metabolism (Q38198952) (← links)
- Autophagy in skeletal muscle homeostasis and in muscular dystrophies (Q38202870) (← links)
- Instructive roles of extracellular matrix on autophagy. (Q38224428) (← links)
- Mitochondria, muscle health, and exercise with advancing age. (Q38453882) (← links)
- Proteotoxicity and cardiac dysfunction (Q38497014) (← links)
- Autophagy as a Potential Target for Sarcopenia. (Q38637495) (← links)
- Role of p66shc in skeletal muscle function. (Q38663521) (← links)