Pages that link to "Q24682108"
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The following pages link to Tipin and Timeless form a mutually protective complex required for genotoxic stress resistance and checkpoint function (Q24682108):
Displaying 50 items.
- Human single-stranded DNA binding proteins are essential for maintaining genomic stability (Q21263027) (← links)
- Cdc7-Dbf4 and the human S checkpoint response to UVC (Q24672815) (← links)
- The human Tim/Tipin complex coordinates an Intra-S checkpoint response to UV that slows replication fork displacement (Q24683336) (← links)
- The Mcm2-7 replicative helicase: a promising chemotherapeutic target (Q26861045) (← links)
- Timeless links replication termination to mitotic kinase activation (Q27342850) (← links)
- Differences in the DNA replication of unicellular eukaryotes and metazoans: known unknowns (Q27694684) (← links)
- The Implications of Multiple Circadian Clock Origins (Q27909963) (← links)
- The Causes and Consequences of Topological Stress during DNA Replication (Q28077125) (← links)
- Tim/Timeless, a member of the replication fork protection complex, operates with the Warsaw breakage syndrome DNA helicase DDX11 in the same fork recovery pathway (Q28114770) (← links)
- Human Timeless and Tipin stabilize replication forks and facilitate sister-chromatid cohesion (Q28118451) (← links)
- Emerging models for the molecular basis of mammalian circadian timing (Q28249506) (← links)
- Modulation of ATR-mediated DNA damage checkpoint response by cryptochrome 1 (Q28506381) (← links)
- Timeless preserves telomere length by promoting efficient DNA replication through human telomeres (Q29041627) (← links)
- Contribution of sleep to the repair of neuronal DNA double-strand breaks: evidence from flies and mice (Q30828167) (← links)
- SUMOylation of ATRIP potentiates DNA damage signaling by boosting multiple protein interactions in the ATR pathway (Q31171264) (← links)
- Network architecture of signaling from uncoupled helicase-polymerase to cell cycle checkpoints and trans-lesion DNA synthesis (Q33474752) (← links)
- Systematic analysis of circadian genes in a population-based sample reveals association of TIMELESS with depression and sleep disturbance (Q33533337) (← links)
- Tipin-replication protein A interaction mediates Chk1 phosphorylation by ATR in response to genotoxic stress (Q33541382) (← links)
- Mammalian TIMELESS is required for ATM-dependent CHK2 activation and G2/M checkpoint control (Q33661490) (← links)
- Checkpoint-Dependent and -Independent Roles of Swi3 in Replication Fork Recovery and Sister Chromatid Cohesion in Fission Yeast (Q33725551) (← links)
- New insights into checkpoint kinase 1 in the DNA damage response signaling network (Q34129155) (← links)
- Swi1 associates with chromatin through the DDT domain and recruits Swi3 to preserve genomic integrity (Q34406008) (← links)
- Sister acts: coordinating DNA replication and cohesion establishment (Q34411870) (← links)
- Coordinated degradation of replisome components ensures genome stability upon replication stress in the absence of the replication fork protection complex (Q34561912) (← links)
- Survival of the replication checkpoint deficient cells requires MUS81-RAD52 function (Q35034362) (← links)
- Timeless functions independently of the Tim-Tipin complex to promote sister chromatid cohesion in normal human fibroblasts (Q35079332) (← links)
- The fork and the kinase: a DNA replication tale from a CHK1 perspective (Q35205026) (← links)
- Architecture and ssDNA interaction of the Timeless-Tipin-RPA complex (Q35369529) (← links)
- The Human RNA Polymerase I Transcription Terminator Complex Acts as a Replication Fork Barrier That Coordinates the Progress of Replication with rRNA Transcription Activity (Q35516592) (← links)
- Swi1Timeless Prevents Repeat Instability at Fission Yeast Telomeres. (Q35962436) (← links)
- Cardiac physiologic and genetic predictors of hyperoxia-induced acute lung injury in mice (Q35987300) (← links)
- Tipin is required for stalled replication forks to resume DNA replication after removal of aphidicolin in Xenopus egg extracts (Q35989104) (← links)
- Fork rotation and DNA precatenation are restricted during DNA replication to prevent chromosomal instability (Q35989991) (← links)
- A comparative proteomic study identified LRPPRC and MCM7 as putative actors in imatinib mesylate cross-resistance in Lucena cell line. (Q35991845) (← links)
- And-1 coordinates with Claspin for efficient Chk1 activation in response to replication stress (Q36000849) (← links)
- Replication Protein A (RPA) deficiency activates the Fanconi anemia DNA repair pathway (Q36072502) (← links)
- Plx1 is required for chromosomal DNA replication under stressful conditions (Q36491053) (← links)
- Chk1 and Claspin potentiate PCNA ubiquitination (Q36585485) (← links)
- Separation of intra-S checkpoint protein contributions to DNA replication fork protection and genomic stability in normal human fibroblasts (Q36618648) (← links)
- The human Tim-Tipin complex interacts directly with DNA polymerase epsilon and stimulates its synthetic activity (Q36812407) (← links)
- Emerging links between the biological clock and the DNA damage response (Q36816885) (← links)
- A prognostic signature of G(2) checkpoint function in melanoma cell lines (Q36823687) (← links)
- Integrating S-phase checkpoint signaling with trans-lesion synthesis of bulky DNA adducts (Q36891684) (← links)
- DNA damage tolerance: a double-edged sword guarding the genome (Q37187991) (← links)
- Replicating damaged DNA in eukaryotes (Q37340387) (← links)
- Dual functions of DNA replication forks in checkpoint signaling and PCNA ubiquitination (Q37374118) (← links)
- Tim-Tipin dysfunction creates an indispensible reliance on the ATR-Chk1 pathway for continued DNA synthesis (Q37387886) (← links)
- Tipin/Tim1/And1 protein complex promotes Pol alpha chromatin binding and sister chromatid cohesion (Q37418052) (← links)
- Mammalian RAD52 Functions in Break-Induced Replication Repair of Collapsed DNA Replication Forks (Q37528453) (← links)
- Regulation of DNA replication by the S-phase DNA damage checkpoint (Q37540336) (← links)