Pages that link to "Q28300280"
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The following pages link to Production of biologically active complement factor H in therapeutically useful quantities (Q28300280):
Displaying 28 items.
- Atypical hemolytic uremic syndrome (Q21202864) (← links)
- Complement regulation: physiology and disease relevance (Q26795441) (← links)
- Membranoproliferative glomerulonephritis and C3 glomerulopathy: resolving the confusion (Q27008508) (← links)
- Solution Structure of CCP Modules 10–12 Illuminates Functional Architecture of the Complement Regulator, Factor H (Q27673696) (← links)
- Complement Evasion Mediated by Enhancement of Captured Factor H: Implications for Protection of Self-Surfaces from Complement (Q28268485) (← links)
- Plasmodium falciparum uses a key functional site in complement receptor type-1 for invasion of human erythrocytes (Q30425504) (← links)
- Complement-mediated injury and protection of endothelium: lessons from atypical haemolytic uraemic syndrome (Q33396792) (← links)
- Novel use of intravenous immunoglobulin G in complement factor H missense mutation: a case report (Q33410111) (← links)
- Moss-Produced, Glycosylation-Optimized Human Factor H for Therapeutic Application in Complement Disorders (Q33437488) (← links)
- Lack of Evidence from Studies of Soluble Protein Fragments that Knops Blood Group Polymorphisms in Complement Receptor-Type 1 Are Driven by Malaria (Q34233738) (← links)
- Affinity purification of human factor H on polypeptides derived from streptococcal m protein: enrichment of the Y402 variant (Q35053992) (← links)
- Dense deposit disease. (Q35124165) (← links)
- Characterization of Inhibitors and Monoclonal Antibodies That Modulate the Interaction between Plasmodium falciparum Adhesin PfRh4 with Its Erythrocyte Receptor Complement Receptor 1. (Q36283289) (← links)
- Comparative Analysis of Novel Complement-Targeted Inhibitors, MiniFH, and the Natural Regulators Factor H and Factor H-like Protein 1 Reveal Functional Determinants of Complement Regulation. (Q36447625) (← links)
- Manipulating the mediator: modulation of the alternative complement pathway C3 convertase in health, disease and therapy. (Q36463266) (← links)
- Selectivity of C3-opsonin targeted complement inhibitors: A distinct advantage in the protection of erythrocytes from paroxysmal nocturnal hemoglobinuria patients. (Q36563619) (← links)
- Rational engineering of a minimized immune inhibitor with unique triple-targeting properties (Q37300691) (← links)
- Factor h: a complement regulator in health and disease, and a mediator of cellular interactions. (Q38223669) (← links)
- Biologic agents in the treatment of glomerulonephritides (Q38514894) (← links)
- Understanding the complement-mediated glomerular diseases: focus on membranoproliferative glomerulonephritis and C3 glomerulopathies (Q38880897) (← links)
- Protection of host cells by complement regulators. (Q38991019) (← links)
- Disease-linked mutations in factor H reveal pivotal role of cofactor activity in self-surface-selective regulation of complement activation (Q41377869) (← links)
- Identification of factor H-like protein 1 as the predominant complement regulator in Bruch's membrane: implications for age-related macular degeneration. (Q41705490) (← links)
- Complement factor H mutation associated with membranoproliferative glomerulonephritis with transformation to atypical haemolytic uraemic syndrome (Q41875130) (← links)
- An extended mini-complement factor H molecule ameliorates experimental C3 glomerulopathy. (Q42625961) (← links)
- An Engineered Complement Factor H Construct for Treatment of C3 Glomerulopathy. (Q51730313) (← links)
- Two distinct conformations of factor H regulate discrete complement-binding functions in the fluid phase and at cell surfaces (Q58738012) (← links)
- Biophysical analysis of sialic acid recognition by the complement regulator Factor H. (Q64981056) (← links)