Pages that link to "Q34266732"
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The following pages link to Monoamine oxidase inhibition prevents mitochondrial dysfunction and apoptosis in myoblasts from patients with collagen VI myopathies (Q34266732):
Displaying 11 items.
- Oxidative Stress-Mediated Skeletal Muscle Degeneration: Molecules, Mechanisms, and Therapies (Q26769962) (← links)
- Pharmacology and Clinical Drug Candidates in Redox Medicine (Q26785525) (← links)
- Melanocytes from Patients Affected by Ullrich Congenital Muscular Dystrophy and Bethlem Myopathy have Dysfunctional Mitochondria That Can be Rescued with Cyclophilin Inhibitors (Q34547398) (← links)
- Novel pathogenic variants and genes for myopathies identified by whole exome sequencing (Q35907744) (← links)
- The Mitochondrial Permeability Transition Pore: Channel Formation by F-ATP Synthase, Integration in Signal Transduction, and Role in Pathophysiology. (Q36146320) (← links)
- Rasagiline and selegiline suppress calcium efflux from mitochondria by PK11195-induced opening of mitochondrial permeability transition pore: a novel anti-apoptotic function for neuroprotection (Q38888524) (← links)
- Denervated muscle fibers induce mitochondrial peroxide generation in neighboring innervated fibers: Role in muscle aging. (Q42704750) (← links)
- Melatonin prevents mitochondrial dysfunctions and death in differentiated skeletal muscle cells (Q48350062) (← links)
- Contribution of monoamine oxidases to vascular oxidative stress in patients with end-stage renal disease requiring hemodialysis. (Q53137131) (← links)
- Drug Repurposing for Duchenne Muscular Dystrophy: The Monoamine Oxidase B Inhibitor Safinamide Ameliorates the Pathological Phenotype in Mice and in Myogenic Cultures From DMD Patients (Q58781307) (← links)
- Teaching an Old Molecule New Tricks: Drug Repositioning for Duchenne Muscular Dystrophy (Q91693351) (← links)