Pages that link to "Q34352305"

The following pages link to The ATM cofactor ATMIN protects against oxidative stress and accumulation of DNA damage in the aging brain (Q34352305):

Displaying 31 items.

• ATM substrate Chk2-interacting Zn2+ finger (ASCIZ) Is a bi-functional transcriptional activator and feedback sensor in the regulation of dynein light chain (DYNLL1) expression (Q24299748) (← links)
• ATMIN is a transcriptional regulator of both lung morphogenesis and ciliogenesis (Q28507428) (← links)
• Atmin mediates kidney morphogenesis by modulating Wnt signaling. (Q34211767) (← links)
• ATM signalling and cancer (Q34357377) (← links)
• Neuronal c-Jun is required for successful axonal regeneration, but the effects of phosphorylation of its N-terminus are moderate. (Q35817090) (← links)
• All stressed out without ATM kinase (Q35841601) (← links)
• The Zinc-finger protein ASCIZ regulates B cell development via DYNLL1 and Bim. (Q36193554) (← links)
• Digitor/dASCIZ Has Multiple Roles in Drosophila Development (Q36197355) (← links)
• DNA Repair Cofactors ATMIN and NBS1 Are Required to Suppress T Cell Activation (Q36257283) (← links)
• Mechanisms and consequences of ATMIN repression in hypoxic conditions: roles for p53 and HIF-1. (Q36580272) (← links)
• The ATM signaling network in development and disease (Q36712849) (← links)
• Mobile phone signal exposure triggers a hormesis-like effect in Atm(+/+) and Atm(-/-) mouse embryonic fibroblasts. (Q37421853) (← links)
• The novel zinc finger protein dASCIZ regulates mitosis in Drosophila via an essential role in dynein light-chain expression (Q37557450) (← links)
• ATM-dependent phosphorylation of MEF2D promotes neuronal survival after DNA damage (Q37663170) (← links)
• Functional interplay between ATM/ATR-mediated DNA damage response and DNA repair pathways in oxidative stress. (Q38221703) (← links)
• ATM activation in hypoxia - causes and consequences (Q38866639) (← links)
• Multiple functions of long non-coding RNAs in oxidative stress, DNA damage response and cancer progression. (Q39368869) (← links)
• Inactivation of the ATMIN/ATM pathway protects against glioblastoma formation (Q39634834) (← links)
• ASCIZ/ATMIN is dispensable for ATM signaling in response to replication stress. (Q41562968) (← links)
• ATMIN is required for maintenance of genomic stability and suppression of B cell lymphoma (Q41578158) (← links)
• Perturbed hematopoiesis in mice lacking ATMIN. (Q41919687) (← links)
• ATMIN: a new tumor suppressor in developing B cells (Q42742811) (← links)
• Temporal analysis of hippocampal CA3 gene co-expression networks in a rat model of febrile seizures (Q47381761) (← links)
• Atmin modulates Pkhd1 expression and may mediate Autosomal Recessive Polycystic Kidney Disease (ARPKD) through altered non-canonical Wnt/Planar Cell Polarity (PCP) signalling (Q58592668) (← links)
• An emerging regulatory network of NHEJ via DYNLL1-mediated 53BP1 redistribution (Q90428408) (← links)
• The relationship between plasma lipids, oxidant-antioxidant status, and glycated proteins in individuals at risk for atherosclerosis (Q92696258) (← links)
• Aging Neurovascular Unit and Potential Role of DNA Damage and Repair in Combating Vascular and Neurodegenerative Disorders (Q92799305) (← links)
• The genomics of oxidative DNA damage, repair, and resulting mutagenesis (Q93014579) (← links)
• ATMIN Is a Tumor Suppressor Gene in Lung Adenocarcinoma (Q93085618) (← links)
• Unilateral intranigral administration of β-sitosterol β-D-glucoside triggers pathological α-synuclein spreading and bilateral nigrostriatal dopaminergic neurodegeneration in the rat (Q93160682) (← links)
• Breaking up with ATM (Q94020354) (← links)