Pages that link to "Q35685695"
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The following pages link to Pathogenic natural antibodies propagate cerebral injury following ischemic stroke in mice (Q35685695):
Displaying 32 items.
- Complement in the Homeostatic and Ischemic Brain (Q26796057) (← links)
- B-1 Cell Heterogeneity and the Regulation of Natural and Antigen-Induced IgM Production (Q28078102) (← links)
- Deficiency of complement receptors CR2/CR1 in Cr2⁻/⁻ mice reduces the extent of secondary brain damage after closed head injury (Q33729953) (← links)
- Natural IgM antibodies that bind neoepitopes exposed as a result of spinal cord injury , drive secondary injury by activating complement (Q33814826) (← links)
- Membrane lipid interactions in intestinal ischemia/reperfusion-induced Injury (Q33902557) (← links)
- Oxidative stress sensitizes retinal pigmented epithelial (RPE) cells to complement-mediated injury in a natural antibody-, lectin pathway-, and phospholipid epitope-dependent manner (Q34620560) (← links)
- Targeting pathogenic postischemic self-recognition by natural IgM to protect against posttransplantation cardiac reperfusion injury (Q35246399) (← links)
- The Fifth Domain of Beta 2 Glycoprotein I Protects from Natural IgM Mediated Cardiac Ischaemia Reperfusion Injury (Q35974974) (← links)
- A Rich-Club Organization in Brain Ischemia Protein Interaction Network (Q35999554) (← links)
- IgM exacerbates glomerular disease progression in complement-induced glomerulopathy (Q36016859) (← links)
- The alternative complement pathway propagates inflammation and injury in murine ischemic stroke (Q36339354) (← links)
- Natural IgM Blockade Limits Infarct Expansion and Left Ventricular Dysfunction in a Swine Myocardial Infarct Model (Q36394858) (← links)
- Targeted inhibition of complement using complement receptor 2-conjugated inhibitors attenuates EAE. (Q36401237) (← links)
- Method parameters' impact on mortality and variability in mouse stroke experiments: a meta-analysis (Q36579398) (← links)
- New therapeutic and diagnostic opportunities for injured tissue-specific targeting of complement inhibitors and imaging modalities (Q37180522) (← links)
- Lung ischemia reperfusion injury: a bench-to-bedside review (Q38050047) (← links)
- A Special Connection between γδ T Cells and Natural Antibodies? (Q38846636) (← links)
- Transplantation and Damage-Associated Molecular Patterns (DAMPs). (Q38898214) (← links)
- Injury site-specific targeting of complement inhibitors for treating stroke (Q38991032) (← links)
- Anti-Inflammatory Targets for the Treatment of Reperfusion Injury in Stroke (Q41314097) (← links)
- Clinical hypothermia temperatures increase complement activation and cell destruction via the classical pathway. (Q41869157) (← links)
- Targeting mechanisms at sites of complement activation for imaging and therapy (Q43194836) (← links)
- Therapeutic hypothermia modulates complement factor C3a and C5a levels in a rat model of hypoxic ischemic encephalopathy (Q45049394) (← links)
- Natural IgM initiates an inflammatory response important for both hepatic ischemia reperfusion injury and regeneration. (Q46310923) (← links)
- Inhibition of Complement Drives Increase in Early Growth Response Proteins and Neuroprotection Mediated by Salidroside After Cerebral Ischemia. (Q47338117) (← links)
- Thromboembolic Model of Cerebral Ischemia and Reperfusion in Mice. (Q48041553) (← links)
- Natural Autoantibodies: An Undersugn Hero of the Immune System and Autoimmune Disorders—A Point of View. (Q55303901) (← links)
- Autoimmunity After Ischemic Stroke and Brain Injury (Q64084557) (← links)
- Acute Complement Inhibition Potentiates Neurorehabilitation and Enhances tPA-Mediated Neuroprotection. (Q64882444) (← links)
- Targeted complement inhibition salvages stressed neurons and inhibits neuroinflammation after stroke in mice (Q88698065) (← links)
- Neuroinflammation as a target for treatment of stroke using mesenchymal stem cells and extracellular vesicles (Q90081477) (← links)
- Therapeutic Modulation of the Complement Cascade in Stroke (Q92650436) (← links)