Pages that link to "Q35888648"
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The following pages link to Oxidative stress and successful antioxidant treatment in models of RYR1-related myopathy (Q35888648):
Displaying 47 items.
- Swimming into prominence: the zebrafish as a valuable tool for studying human myopathies and muscular dystrophies. (Q27028125) (← links)
- The role of oxidative stress in skeletal muscle injury and regeneration: focus on antioxidant enzymes (Q28076877) (← links)
- Review of RyR1 pathway and associated pathomechanisms (Q28078420) (← links)
- Pathophysiological concepts in the congenital myopathies: blurring the boundaries, sharpening the focus (Q28087567) (← links)
- The transcription coactivator ASC-1 is a regulator of skeletal myogenesis, and its deficiency causes a novel form of congenital muscle disease (Q28508101) (← links)
- N-acetyl-L-cysteine prevents stress-induced desmin aggregation in cellular models of desminopathy (Q28533968) (← links)
- Recent advances using zebrafish animal models for muscle disease drug discovery (Q28551915) (← links)
- Cross-tissue and cross-species analysis of gene expression in skeletal muscle and electric organ of African weakly-electric fish (Teleostei; Mormyridae). (Q30663602) (← links)
- Whole exome sequencing of a patient with suspected mitochondrial myopathy reveals novel compound heterozygous variants in RYR1. (Q33718496) (← links)
- Fluoxetine prevents dystrophic changes in a zebrafish model of Duchenne muscular dystrophy (Q33991780) (← links)
- Analysis of oxidative stress in zebrafish embryos. (Q34419696) (← links)
- Revealing the complexity of a monogenic disease: rett syndrome exome sequencing (Q34611497) (← links)
- Malignant hyperthermia and the clinical significance of type-1 ryanodine receptor gene (RYR1) variants: proceedings of the 2013 MHAUS Scientific Conference (Q34770934) (← links)
- Triadopathies: an emerging class of skeletal muscle diseases (Q35340211) (← links)
- Oxidative stress, mitochondrial damage, and cores in muscle from calsequestrin-1 knockout mice (Q35727433) (← links)
- Emerging applications for zebrafish as a model organism to study oxidative mechanisms and their roles in inflammation and vascular accumulation of oxidized lipids (Q36252515) (← links)
- Analysis of Zebrafish Larvae Skeletal Muscle Integrity with Evans Blue Dye. (Q36408382) (← links)
- Effects of aerobic training on exercise-related oxidative stress in mitochondrial myopathies. (Q36479248) (← links)
- Novel deletion of lysine 7 expands the clinical, histopathological and genetic spectrum of TPM2-related myopathies. (Q36611392) (← links)
- Genotype-phenotype correlations in recessive RYR1-related myopathies (Q37114746) (← links)
- Force measurement during contraction to assess muscle function in zebrafish larvae. (Q37354921) (← links)
- Selenoprotein W redox-regulated Ca2+ channels correlate with selenium deficiency-induced muscles Ca2+ leak (Q37628909) (← links)
- Analysis of embryonic and larval zebrafish skeletal myofibers from dissociated preparations (Q37709161) (← links)
- Oxidative stress and pathology in muscular dystrophies: focus on protein thiol oxidation and dysferlinopathies. (Q38075201) (← links)
- Zebrafish as a model system for mitochondrial biology and diseases (Q38140444) (← links)
- Dietary proteins and amino acids in the control of the muscle mass during immobilization and aging: role of the MPS response. (Q39125369) (← links)
- The HDAC Inhibitor TSA Ameliorates a Zebrafish Model of Duchenne Muscular Dystrophy (Q41823347) (← links)
- Excitation-Contraction Coupling Alterations in Myopathies (Q42046918) (← links)
- A novel late-onset axial myopathy associated with mutations in the skeletal muscle ryanodine receptor (RYR1) gene (Q45800786) (← links)
- When best evidence is rarer than the disease (Q46541105) (← links)
- Congenital myopathies: clinical phenotypes and new diagnostic tools (Q47134873) (← links)
- Muscle dysfunction in a zebrafish model of Duchenne muscular dystrophy. (Q48053828) (← links)
- Congenital myopathies: disorders of excitation-contraction coupling and muscle contraction (Q49388884) (← links)
- Treating pediatric neuromuscular disorders: The future is now. (Q52756417) (← links)
- Inositol trisphosphate receptor-mediated Ca2+ signalling stimulates mitochondrial function and gene expression in core myopathy patients. (Q55397765) (← links)
- 6-minute walk test as a measure of disease progression and fatigability in a cohort of individuals with RYR1-related myopathies. (Q55498109) (← links)
- Ryanodine Receptor 1-Related Myopathies: Diagnostic and Therapeutic Approaches (Q58600210) (← links)
- Real-time visualization of oxidative stress-mediated neurodegeneration of individual spinal motor neurons in vivo (Q58711477) (← links)
- Recent advances in understanding congenital myopathies (Q60959783) (← links)
- Fishing in the Cell Powerhouse: Zebrafish as A Tool for Exploration of Mitochondrial Defects Affecting the Nervous System. (Q64926601) (← links)
- Functional Electrical Stimulation: A Possible Strategy to Improve Muscle Function in Central Core Disease? (Q64963922) (← links)
- Mixed methods analysis of Health-Related Quality of Life in ambulant individuals affected with RYR1-related myopathies pre-post-N-acetylcysteine therapy (Q89630438) (← links)
- Identification of drug modifiers for RYR1 related myopathy using a multi-species discovery pipeline (Q90730108) (← links)
- Discovery of Novel Therapeutics for Muscular Dystrophies using Zebrafish Phenotypic Screens (Q91697849) (← links)
- Cored in the act: the use of models to understand core myopathies (Q92238728) (← links)
- A Novel Amino Acid Composition Ameliorates Short-Term Muscle Disuse Atrophy in Healthy Young Men (Q92243171) (← links)
- Preclinical model systems of ryanodine receptor 1-related myopathies and malignant hyperthermia: a comprehensive scoping review of works published 1990-2019 (Q94544834) (← links)