Pages that link to "Q36082853"
Jump to navigation
Jump to search
The following pages link to Investigating Nonalcoholic Fatty Liver Disease in a Liver-on-a-Chip Microfluidic Device (Q36082853):
Displaying 18 items.
- Computationally Informed Design of a Multi-Axial Actuated Microfluidic Chip Device (Q33906036) (← links)
- Development of an in vitro human liver system for interrogating nonalcoholic steatohepatitis (Q39109576) (← links)
- Mitochondrial carnitine palmitoyl transferase-II inactivity aggravates lipid accumulation in rat hepatocarcinogenesis (Q41969545) (← links)
- Non-alcoholic fatty liver disease (NAFLD) models in drug discovery (Q46251147) (← links)
- Organ-on-a-Chip Technology for Reproducing Multiorgan Physiology (Q47637355) (← links)
- Translating scientific discovery: the need for preclinical models of nonalcoholic steatohepatitis (Q49331156) (← links)
- Influence of different concentrations of uric acid on oxidative stress in steatosis hepatocytes. (Q52653039) (← links)
- Tissue engineering toward organ-specific regeneration and disease modeling. (Q55316142) (← links)
- Human hepatic 3D spheroids as a model for steatosis and insulin resistance (Q58700518) (← links)
- A glass-based, continuously zonated and vascularized human liver acinus microphysiological system (vLAMPS) designed for experimental modeling of diseases and ADME/TOX. (Q64886066) (← links)
- Harnessing the Potential of Stem Cells for Disease Modeling: Progress and Promises (Q89633106) (← links)
- Novel defatting strategies reduce lipid accumulation in primary human culture models of liver steatosis (Q89864969) (← links)
- Liver Bioreactor Design Issues of Fluid Flow and Zonation, Fibrosis, and Mechanics: A Computational Perspective (Q89976263) (← links)
- Advanced Organ-on-a-Chip Devices to Investigate Liver Multi-Organ Communication: Focus on Gut, Microbiota and Brain (Q90381944) (← links)
- Engineered Liver-on-a-Chip Platform to Mimic Liver Functions and Its Biomedical Applications: A Review (Q90574693) (← links)
- HIF-2α upregulation mediated by hypoxia promotes NAFLD-HCC progression by activating lipid synthesis via the PI3K-AKT-mTOR pathway (Q91655677) (← links)
- Liver-on-a-Chip Models of Fatty Liver Disease (Q92467382) (← links)
- Organ-On-Chip Technology: The Future of Feto-Maternal Interface Research? (Q97644327) (← links)