Pages that link to "Q36353924"
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The following pages link to Bax, but not Bcl-xL, decreases the lifetime of planar phospholipid bilayer membranes at subnanomolar concentrations (Q36353924):
Displaying 50 items.
- tBID, a membrane-targeted death ligand, oligomerizes BAK to release cytochrome c (Q24290118) (← links)
- Pore-forming activity of BAD is regulated by specific phosphorylation and structural transitions of the C-terminal part (Q24307486) (← links)
- Bax activates endophilin B1 oligomerization and lipid membrane vesiculation (Q24322581) (← links)
- Identification of novel in vivo phosphorylation sites of the human proapoptotic protein BAD: pore-forming activity of BAD is regulated by phosphorylation (Q24323069) (← links)
- Spatial and temporal association of Bax with mitochondrial fission sites, Drp1, and Mfn2 during apoptosis (Q24337576) (← links)
- Voltage-induced nonconductive pre-pores and metastable single pores in unmodified planar lipid bilayer (Q24537440) (← links)
- PB1-F2, an influenza A virus-encoded proapoptotic mitochondrial protein, creates variably sized pores in planar lipid membranes (Q24563255) (← links)
- The pro-apoptotic Bcl-2 family member tBid localizes to mitochondrial contact sites (Q24794830) (← links)
- A new view of the lethal apoptotic pore (Q27000479) (← links)
- Mitochondrial apoptosis and BH3 mimetics (Q28069552) (← links)
- Essential role of voltage-dependent anion channel in various forms of apoptosis in mammalian cells (Q28206500) (← links)
- The structure of the C-terminal domain of the pro-apoptotic protein Bak and its interaction with model membranes. (Q28366855) (← links)
- Lytic and non-lytic permeabilization of cardiolipin-containing lipid bilayers induced by cytochrome C (Q28385986) (← links)
- Bax activation initiates the assembly of a multimeric catalyst that facilitates Bax pore formation in mitochondrial outer membranes (Q28483986) (← links)
- The BCL-2 protein family: opposing activities that mediate cell death (Q29547380) (← links)
- Mitochondria and cell death: outer membrane permeabilization and beyond (Q29615459) (← links)
- Pro-apoptotic Bax molecules densely populate the edges of membrane pores (Q30388868) (← links)
- BCL2 and CASP8 regulation by NF-kappaB differentially affect mitochondrial function and cell fate in antiestrogen-sensitive and -resistant breast cancer cells. (Q30432099) (← links)
- Bax/Bak promote sumoylation of DRP1 and its stable association with mitochondria during apoptotic cell death (Q30480587) (← links)
- Differential cellular regulation of the mitochondrial permeability transition in an in vitro model of 1,3-dinitrobenzene-induced encephalopathy (Q31431906) (← links)
- Polycations induce the release of soluble intermembrane mitochondrial proteins. (Q31690013) (← links)
- Unravelling the bcl-2 apoptosis code with a simple model system (Q33344919) (← links)
- Oblimersen for the treatment of patients with chronic lymphocytic leukemia (Q33379565) (← links)
- Apoptosis: definition, mechanisms, and relevance to disease (Q33777704) (← links)
- Bcl-2 proteins: regulators of apoptosis or of mitochondrial homeostasis? (Q33787932) (← links)
- Mitochondria and apoptosis: HQ or high-security prison? (Q33816824) (← links)
- Ca(2+)-mediated mitochondrial dysfunction and the protective effects of Bcl-2. (Q33836188) (← links)
- Mitochondrial membrane perturbations in cholestasis. (Q33836633) (← links)
- BAX-dependent transport of cytochrome c reconstituted in pure liposomes (Q33912996) (← links)
- Variations in the rheostat model of apoptosis: what studies of retinal ganglion cell death tell us about the functions of the Bcl2 family proteins (Q33914559) (← links)
- Mechanisms underlying hypoxia-induced neuronal apoptosis (Q33935219) (← links)
- Bax, Bid and the permeabilization of the mitochondrial outer membrane in apoptosis (Q33954487) (← links)
- Mitochondria as the central control point of apoptosis (Q33994978) (← links)
- Apoptosis: implications for inflammatory bowel disease (Q34015892) (← links)
- Pro-apoptotic cascade activates BID, which oligomerizes BAK or BAX into pores that result in the release of cytochrome c. (Q34139321) (← links)
- VDAC regulation by the Bcl-2 family of proteins (Q34139328) (← links)
- The 'harmless' release of cytochrome c. (Q34139341) (← links)
- Enlargement and contracture of C2-ceramide channels (Q34182656) (← links)
- Apoptosis and liver diseases: recent concepts of mechanism and significance (Q34242758) (← links)
- Mitofusins and the mitochondrial permeability transition: the potential downside of mitochondrial fusion (Q34277591) (← links)
- The mitochondrion in apoptosis: how Pandora's box opens (Q34286386) (← links)
- Study of PTPC composition during apoptosis for identification of viral protein target. (Q34307368) (← links)
- Apoptotic pore formation is associated with in-plane insertion of Bak or Bax central helices into the mitochondrial outer membrane (Q34314503) (← links)
- The role of the mitochondrial apoptosis induced channel MAC in cytochrome c release (Q34451529) (← links)
- Involvement of mitochondria in apoptosis (Q34490979) (← links)
- A reversible component of mitochondrial respiratory dysfunction in apoptosis can be rescued by exogenous cytochrome c. (Q34582647) (← links)
- Mitochondria: regulating the inevitable. (Q34648793) (← links)
- And all of a sudden it's over: mitochondrial outer-membrane permeabilization in apoptosis (Q34648801) (← links)
- The adenine nucleotide translocator in apoptosis (Q34648850) (← links)
- Sphingosine, a product of ceramide hydrolysis, influences the formation of ceramide channels. (Q34984615) (← links)