Pages that link to "Q36768116"
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The following pages link to Enhanced jun gene expression is an early genomic response to transforming growth factor beta stimulation (Q36768116):
Displaying 50 items.
- Smads bind directly to the Jun family of AP-1 transcription factors (Q24685193) (← links)
- Transforming growth factor-beta and breast cancer: Mammary gland development (Q24793194) (← links)
- c-Jun (AP-1) activates BMP-4 transcription in Xenopus embryos (Q28142023) (← links)
- The activity of guanine exchange factor NET1 is essential for transforming growth factor-beta-mediated stress fiber formation (Q28207920) (← links)
- SMIF, a Smad4-interacting protein that functions as a co-activator in TGFbeta signalling (Q28506547) (← links)
- BMP-2-induced Runx2 expression is mediated by Dlx5, and TGF-beta 1 opposes the BMP-2-induced osteoblast differentiation by suppression of Dlx5 expression (Q28570736) (← links)
- Smad3-Smad4 and AP-1 complexes synergize in transcriptional activation of the c-Jun promoter by transforming growth factor beta (Q28608955) (← links)
- The role of Jun, Fos and the AP-1 complex in cell-proliferation and transformation (Q29400446) (← links)
- TGF- Biology in Mammary Development and Breast Cancer (Q33681686) (← links)
- Ionizing radiation regulates expression of the c-jun protooncogene (Q33709934) (← links)
- Pretranscriptional regulation of Tgf-beta1 by PI polyamide prevents scarring and accelerates wound healing of the cornea after exposure to alkali (Q33731237) (← links)
- Role of Ras and Mapks in TGFbeta signaling (Q33857574) (← links)
- TIEG proteins join the Smads as TGF-beta-regulated transcription factors that control pancreatic cell growth. (Q33890489) (← links)
- TGF-beta induces fibronectin synthesis through a c-Jun N-terminal kinase-dependent, Smad4-independent pathway (Q33890533) (← links)
- Autocrine induction of tumor protease production and invasion by a metallothionein-regulated TGF-beta 1 (Ser223, 225) (Q33937674) (← links)
- Transcriptional regulation of c-jun gene expression by arabinofuranosylcytosine in human myeloid leukemia cells (Q34263164) (← links)
- Jun, the oncoprotein. (Q34279331) (← links)
- JunB mediates basal- and TGFβ1-induced smooth muscle cell contractility (Q34545069) (← links)
- Defective repression of c-myc in breast cancer cells: A loss at the core of the transforming growth factor beta growth arrest program (Q34637234) (← links)
- What's new in the role of cytokines on osteoblast proliferation and differentiation? (Q35221338) (← links)
- Induction of junB expression, but not c-jun, by granulocyte colony-stimulating factor or macrophage colony-stimulating factor in the proliferative response of human myeloid leukemia cells (Q35602784) (← links)
- Dual role of SnoN in mammalian tumorigenesis (Q35641680) (← links)
- Reconstitution of TGFBR2-Mediated Signaling Causes Upregulation of GDF-15 in HCT116 Colorectal Cancer Cells (Q35675385) (← links)
- Process of hepatic metastasis from pancreatic cancer: biology with clinical significance (Q35739397) (← links)
- Neutralizing antibodies against transforming growth factor beta potentiate the proliferation of Ki-1 positive lymphoma cells. Further evidence for negative autocrine regulation by transforming growth factor beta (Q35830680) (← links)
- Localization of Fos and Jun proteins in rat aortic smooth muscle cells after vascular injury. (Q35832425) (← links)
- JunB contributes to Id2 repression and the epithelial-mesenchymal transition in response to transforming growth factor-β. (Q35839788) (← links)
- The role of ras gene expression and transforming growth factor α production in the etiology and progression of rodent and human breast cancer (Q36486913) (← links)
- Enhanced VEGF production and decreased immunogenicity induced by TGF-beta 1 promote liver metastasis of pancreatic cancer (Q36623590) (← links)
- Control of junB and extracellular matrix protein expression by transforming growth factor-beta 1 is independent of simian virus 40 T antigen-sensitive growth-sensitive growth-inhibitory events (Q36684664) (← links)
- Regulation of proto-oncogene expression in adult and developing lungs. (Q36684750) (← links)
- Cloning from a mouse osteoblastic cell line of a set of transforming-growth-factor-beta 1-regulated genes, one of which seems to encode a follistatin-related polypeptide (Q36720977) (← links)
- Induction of the proto-oncogene c-jun by angiotensin II. (Q36729875) (← links)
- Production of hydrogen peroxide by transforming growth factor-beta 1 and its involvement in induction of egr-1 in mouse osteoblastic cells (Q36736153) (← links)
- Transcriptional regulatory elements downstream of the JunB gene (Q36736293) (← links)
- A highly conserved intronic sequence is involved in transcriptional regulation of the alpha 1(I) collagen gene. (Q36737357) (← links)
- Cloning of a growth arrest-specific and transforming growth factor beta-regulated gene, TI 1, from an epithelial cell line (Q36738990) (← links)
- Autoinduction of transforming growth factor beta 1 is mediated by the AP-1 complex (Q36755274) (← links)
- Mutations in the Jun delta region suggest an inverse correlation between transformation and transcriptional activation (Q36790815) (← links)
- Response of bone marrow stromal cells to adipogenic antagonists (Q36794262) (← links)
- Evidence for the involvement of protein kinase activity in transforming growth factor-beta signal transduction (Q36808476) (← links)
- Role of protein kinase C and transcription factor AP-1 in the acid-induced increase in Na/H antiporter activity (Q37051106) (← links)
- Rapid induction of the c-jun protooncogene in the avian oviduct by the antiestrogen tamoxifen (Q37387061) (← links)
- Recent advances in our understanding of tubal function (Q37768723) (← links)
- TGF-beta receptors and TGF-beta binding proteoglycans: recent progress in identifying their functional properties (Q37886466) (← links)
- Transcriptional control of expression of the TGF-betas (Q37943210) (← links)
- c-Jun, at the crossroad of the signaling network (Q37969687) (← links)
- TAK1 activation of the mouse JunB promoter is mediated through a CCAAT box and NF-Y. (Q38295793) (← links)
- Smad3/AP-1 interactions control transcriptional responses to TGF-beta in a promoter-specific manner (Q38299529) (← links)
- Activation of Ito cells involves regulation of AP-1 binding proteins and induction of type I collagen gene expression (Q38301298) (← links)