Pages that link to "Q39447996"

The following pages link to The BH3 domain of Bcl-x(S) is required for inhibition of the antiapoptotic function of Bcl-x(L). (Q39447996):

Displaying 23 items.

• MCL-1S, a splicing variant of the antiapoptotic BCL-2 family member MCL-1, encodes a proapoptotic protein possessing only the BH3 domain (Q22254196) (← links)
• Identification of small-molecule inhibitors of interaction between the BH3 domain and Bcl-xL (Q28200040) (← links)
• The functional differences between pro-survival and pro-apoptotic B cell lymphoma 2 (Bcl-2) proteins depend on structural differences in their Bcl-2 homology 3 (BH3) domains. (Q30368405) (← links)
• Effect of endogenous androgens on 17beta-estradiol-mediated protection after spinal cord injury in male rats (Q33841455) (← links)
• Deubiquitinase OTUD6B Isoforms Are Important Regulators of Growth and Proliferation (Q34545092) (← links)
• Killing of sarcoma cells by proapoptotic Bcl-X(S): role of the BH3 domain and regulation by Bcl-X(L). (Q34781607) (← links)
• Bcl-xL is necessary for neurite outgrowth in hippocampal neurons (Q34802591) (← links)
• Mechanisms of cytochrome c release by proapoptotic BCL-2 family members (Q35120707) (← links)
• Protein kinase C-dependent control of Bcl-x alternative splicing (Q36316498) (← links)
• Gene expression in osteoblast cells treated with submicron to nanometer hydroxyapatite-mullite eluate particles (Q39406843) (← links)
• Bax activation by engagement with, then release from, the BH3 binding site of Bcl-xL. (Q39617756) (← links)
• Hydrogen peroxide-induced apoptosis in human gingival fibroblasts (Q39991644) (← links)
• Akt and Bcl-xL promote growth factor-independent survival through distinct effects on mitochondrial physiology (Q40816815) (← links)
• Bcl-XL expression correlates with primary macrophage differentiation, activation of functional competence, and survival and results from synergistic transcriptional activation by Ets2 and PU.1. (Q40817271) (← links)
• Bcl-x(S) can form homodimers and heterodimers and its apoptotic activity requires localization of Bcl-x(S) to the mitochondria and its BH3 and loop domains (Q43722383) (← links)
• Bcl-x(S) induces an NGF-inhibitable cytochrome c release (Q44948703) (← links)
• Neuron-specific Bcl-2 homology 3 domain-only splice variant of Bak is anti-apoptotic in neurons, but pro-apoptotic in non-neuronal cells (Q48933231) (← links)
• Down-regulation of Bcl-2 and Bcl-xL expression with bispecific antisense treatment in glioblastoma cell lines induce cell death. (Q55473040) (← links)
• The BH4 domain of Bcl-XL rescues astrocyte degeneration in amyotrophic lateral sclerosis by modulating intracellular calcium signals (Q57732159) (← links)
• BCL-2 family isoforms in apoptosis and cancer (Q64118328) (← links)
• The Structural Biology of Bcl-xL. (Q64897378) (← links)
• Bak but not Bax is essential for Bcl-xS-induced apoptosis (Q81699206) (← links)
• BCL-XL expression is essential for human erythropoiesis and engraftment of hematopoietic stem cells (Q92453289) (← links)