Pages that link to "Q33744644"
Jump to navigation
Jump to search
The following pages link to NF-kappaB activation in hypothalamic pro-opiomelanocortin neurons is essential in illness- and leptin-induced anorexia (Q33744644):
Displaying 34 items.
- Neuroinflammatory basis of metabolic syndrome (Q26852584) (← links)
- Celastrol, an NF-κB inhibitor, improves insulin resistance and attenuates renal injury in db/db mice (Q27323293) (← links)
- The central role of hypothalamic inflammation in the acute illness response and cachexia (Q28080648) (← links)
- Hypothalamic TLR2 triggers sickness behavior via a microglia-neuronal axis (Q28383973) (← links)
- Phospholipase C-related catalytically inactive protein can regulate obesity, a state of peripheral inflammation (Q33560216) (← links)
- Neuropeptides in the pathophysiology and treatment of cachexia (Q34712204) (← links)
- Hypothalamic microinflammation: a common basis of metabolic syndrome and aging (Q34808934) (← links)
- Hypothalamic inflammation: a double-edged sword to nutritional diseases (Q35318592) (← links)
- TAK1 in brain endothelial cells mediates fever and lethargy (Q35627672) (← links)
- A weighty problem: metabolic perturbations and the obesity-cancer link (Q35689545) (← links)
- Leptin in anorexia and cachexia syndrome (Q35829778) (← links)
- Uric Acid Produces an Inflammatory Response through Activation of NF-κB in the Hypothalamus: Implications for the Pathogenesis of Metabolic Disorders (Q35856483) (← links)
- Increased hypothalamic serotonin turnover in inflammation-induced anorexia (Q36024642) (← links)
- Lipopolysacharide Rapidly and Completely Suppresses AgRP Neuron-Mediated Food Intake in Male Mice (Q36965238) (← links)
- A distinct brain pathway links viral RNA exposure to sickness behavior (Q37110979) (← links)
- Hypothalamic activation is essential for endotoxemia-induced acute muscle wasting (Q37475891) (← links)
- The Leptin Receptor Complex: Heavier Than Expected? (Q37655073) (← links)
- Leptin signaling regulates hypothalamic expression of nescient helix-loop-helix 2 (Nhlh2) through signal transducer and activator 3 (Stat3). (Q37700045) (← links)
- GABAergic signaling by AgRP neurons prevents anorexia via a melanocortin-independent mechanism (Q37826478) (← links)
- Leptin promotes a proinflammatory lipid profile and induces inflammatory pathways in human SZ95 sebocytes (Q38306107) (← links)
- Leptin resistance in diet-induced obesity: the role of hypothalamic inflammation (Q38318601) (← links)
- Leptin signalling pathways in hypothalamic neurons (Q38703387) (← links)
- Hypothalamic and inflammatory basis of hypertension (Q38774292) (← links)
- Renaissance of leptin for obesity therapy (Q38777443) (← links)
- Pain Relief with Wet Cupping Therapy in Rats is Mediated by Heat Shock Protein 70 and ß-Endorphin (Q39152049) (← links)
- Hypothalamic Inflammation and Energy Balance Disruptions: Spotlight on Chemokines (Q41418798) (← links)
- Knocking down the transcript of NF-kappaB modulates the reciprocal regulation of endogenous antioxidants and feeding behavior in phenylpropanolamine-treated rats (Q42726126) (← links)
- Inhibiting neuropeptide Y Y1 receptor modulates melanocortin receptor- and NF-κB-mediated feeding behavior in phenylpropanolamine-treated rats (Q45775341) (← links)
- Hypothalamic POMC expression is required for peripheral insulin action on hepatic gluconeogenesis through regulating STAT3 in sepsis rats (Q48333429) (← links)
- Central dopamine action modulates neuropeptide-controlled appetite via the hypothalamic PI3K/NF-κB-dependent mechanism (Q48582523) (← links)
- The brain splits obesity and hypertension (Q49076219) (← links)
- Nuclear factor κB (NF-κB) suppresses food intake and energy expenditure in mice by directly activating the Pomc promoter. (Q51533764) (← links)
- Hypothalamic Inflammation at a Crossroad of Somatic Diseases (Q58093829) (← links)
- Hypertension in obesity: the role of hypothalamic inflammation (Q85774927) (← links)