Pages that link to "Q40459805"
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The following pages link to Proteasome inhibition alters glucose-stimulated (pro)insulin secretion and turnover in pancreatic {beta}-cells. (Q40459805):
Displaying 34 items.
- Excessive islet NO generation in type 2 diabetic GK rats coincides with abnormal hormone secretion and is counteracted by GLP-1. (Q33334446) (← links)
- Gene expression profiles in Atlantic salmon adipose-derived stromo-vascular fraction during differentiation into adipocytes (Q33524582) (← links)
- Misfolded proinsulin affects bystander proinsulin in neonatal diabetes (Q33581459) (← links)
- Degradation of an intramitochondrial protein by the cytosolic proteasome (Q33636568) (← links)
- The regulation and turnover of mitochondrial uncoupling proteins. (Q33936374) (← links)
- PERK (EIF2AK3) regulates proinsulin trafficking and quality control in the secretory pathway (Q34024253) (← links)
- Substrate-favored lysosomal and proteasomal pathways participate in the normal balance control of insulin precursor maturation and disposal in β-cells (Q34079350) (← links)
- Phosphorylation and degradation of tomosyn-2 de-represses insulin secretion (Q34139171) (← links)
- Withaferin A induces proteasome inhibition, endoplasmic reticulum stress, the heat shock response and acquisition of thermotolerance (Q34316709) (← links)
- Proteasome dysfunction mediates high glucose-induced apoptosis in rodent beta cells and human islets (Q35123951) (← links)
- Retinal proteins associated with redox regulation and protein folding play central roles in response to high glucose conditions (Q35535076) (← links)
- Investigational agents that protect pancreatic islet beta-cells from failure (Q36268360) (← links)
- Opposite effects of proteasome inhibitors in the adrenergic induction of arylalkylamine N-acetyltransferase in rat pinealocytes (Q36474512) (← links)
- Degradation of cAMP-responsive element-binding protein by the ubiquitin-proteasome pathway contributes to glucotoxicity in beta-cells and human pancreatic islets (Q37163495) (← links)
- Emerging roles for the ubiquitin-proteasome system and autophagy in pancreatic beta-cells (Q37277467) (← links)
- Genetics of type 2 diabetes in East Asian populations (Q38044865) (← links)
- Mitochondrial oxidative metabolism and uncoupling proteins in the failing heart (Q38246367) (← links)
- High glucose-induced proteome alterations in retinal pigmented epithelium cells and its possible relevance to diabetic retinopathy (Q39262816) (← links)
- Putative association between UBE2E2 polymorphisms and the risk of gestational diabetes mellitus (Q39383022) (← links)
- Activation of PERK kinase in neural cells by proteasome inhibitor treatment (Q39782919) (← links)
- Protective role for nitric oxide during the endoplasmic reticulum stress response in pancreatic beta-cells. (Q40177195) (← links)
- Genetic variations in the PSMA3, PSMA6 and PSMC6 genes are associated with type 1 diabetes in Latvians and with expression level of number of UPS-related and T1DM-susceptible genes in HapMap individuals (Q40217776) (← links)
- Endoplasmic reticulum stress and the unfolded protein response regulate genomic cystic fibrosis transmembrane conductance regulator expression (Q40229391) (← links)
- Essential role of ubiquitin-proteasome system in normal regulation of insulin secretion (Q40303586) (← links)
- Proteasome inhibitors, including curcumin, improve pancreatic β-cell function and insulin sensitivity in diabetic mice (Q40945955) (← links)
- Proteins in the insulin-secreting cell line MIN6 bind the imidazoline compound BL11282. (Q44187475) (← links)
- The ubiquitin-proteasome system regulates the stability and activity of the glucose sensor glucokinase in pancreatic β-cells (Q44287398) (← links)
- Intracellular degradation of insulin and crinophagy are maintained by nitric oxide and cyclo-oxygenase 2 activity in isolated pancreatic islets (Q46914949) (← links)
- The immunoproteasome is induced by cytokines and regulates apoptosis in human islets. (Q51037424) (← links)
- A genome-wide association study in the Japanese population identifies susceptibility loci for type 2 diabetes at UBE2E2 and C2CD4A-C2CD4B (Q57249678) (← links)
- PABPN1 polyalanine tract deletion and long expansions modify its aggregation pattern and expression (Q80957829) (← links)
- The intermediate proteasome is constitutively expressed in pancreatic beta cells and upregulated by stimulatory, low concentrations of interleukin 1 β (Q89695487) (← links)
- Functional proteasome complex is required for turnover of islet amyloid polypeptide in pancreatic β-cells (Q90254959) (← links)
- Contribution of the Unfolded Protein Response (UPR) to the Pathogenesis of Proteasome-Associated Autoinflammatory Syndromes (PRAAS) (Q91894003) (← links)