Pages that link to "Q41411411"
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The following pages link to Induction of tyrosine phosphorylation of Vav and expression of Pim-1 correlates with Jak2-mediated growth signaling from the erythropoietin receptor (Q41411411):
Displaying 25 items.
- Janus kinase 2 (Q14914518) (← links)
- Regulatory and signaling properties of the Vav family (Q24554307) (← links)
- Mice deficient for all PIM kinases display reduced body size and impaired responses to hematopoietic growth factors (Q24563610) (← links)
- Direct association with and dephosphorylation of Jak2 kinase by the SH2-domain-containing protein tyrosine phosphatase SHP-1 (Q24647567) (← links)
- Analysis of receptor signaling pathways by mass spectrometry: identification of vav-2 as a substrate of the epidermal and platelet-derived growth factor receptors (Q24649799) (← links)
- p95vav associates with the nuclear protein Ku-70 (Q24650938) (← links)
- Crystal Structure of Pim1 Kinase in Complex with a Pyrido[4,3-D]Pyrimidine Derivative Suggests a Unique Binding Mode (Q27679530) (← links)
- AP1 regulation of proliferation and initiation of apoptosis in erythropoietin-dependent erythroid cells (Q33774503) (← links)
- Cooperation of Spi-1/PU.1 with an activated erythropoietin receptor inhibits apoptosis and Epo-dependent differentiation in primary erythroblasts and induces their Kit ligand-dependent proliferation (Q33887496) (← links)
- Cell and gene therapy for severe heart failure patients: the time and place for Pim-1 kinase (Q34075071) (← links)
- Testing electromagnetic fields for potential carcinogenic activity: a critical review of animal models (Q34639888) (← links)
- Developmental stage-specific effects of Pim-1 dysregulation on murine bone marrow B cell development (Q36048889) (← links)
- Signals emanating from the membrane proximal region of the thrombopoietin receptor (mpl) support hematopoietic stem cell self-renewal (Q36113963) (← links)
- Erythropoietin induces activation of Stat5 through association with specific tyrosines on the receptor that are not required for a mitogenic response. (Q36558498) (← links)
- Intrinsic resistance to PIM kinase inhibition in AML through p38α-mediated feedback activation of mTOR signaling (Q37437166) (← links)
- Peginesatide and erythropoietin stimulate similar erythropoietin receptor-mediated signal transduction and gene induction events (Q37700912) (← links)
- Why Target PIM1 for Cancer Diagnosis and Treatment? (Q37797472) (← links)
- For better or for worse: the role of Pim oncogenes in tumorigenesis (Q37820056) (← links)
- JAK2 inhibitors for myeloproliferative neoplasms: what is next? (Q38684435) (← links)
- Selective JAK2 inhibition specifically decreases Hodgkin lymphoma and mediastinal large B-cell lymphoma growth in vitro and in vivo (Q39017147) (← links)
- The direct Myc target Pim3 cooperates with other Pim kinases in supporting viability of Myc-induced B-cell lymphomas (Q39529566) (← links)
- Interleukin 2 and erythropoietin activate STAT5/MGF via distinct pathways (Q40806909) (← links)
- Phosphorylation of erythropoietin receptors in the endoplasmic reticulum by pervanadate-mediated inhibition of tyrosine phosphatases (Q41080870) (← links)
- A potential therapeutic target for FLT3-ITD AML: PIM1 kinase (Q41973517) (← links)
- FLT3-ITD induces expression of Pim kinases through STAT5 to confer resistance to the PI3K/Akt pathway inhibitors on leukemic cells by enhancing the mTORC1/Mcl-1 pathway. (Q52366350) (← links)