Pages that link to "Q48292540"
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The following pages link to Tau binding to microtubules does not directly affect microtubule-based vesicle motility (Q48292540):
Displaying 45 items.
- Tau and tauopathies (Q27027415) (← links)
- Inhibition of fast axonal transport by pathogenic SOD1 involves activation of p38 MAP kinase (Q28533850) (← links)
- The amino terminus of tau inhibits kinesin-dependent axonal transport: implications for filament toxicity. (Q30490027) (← links)
- Phosphorylation in the amino terminus of tau prevents inhibition of anterograde axonal transport. (Q30504709) (← links)
- Heat shock protein 70 prevents both tau aggregation and the inhibitory effects of preexisting tau aggregates on fast axonal transport. (Q30519474) (← links)
- Axonal transport rates in vivo are unaffected by tau deletion or overexpression in mice (Q33623043) (← links)
- The role of tau kinases in Alzheimer's disease (Q34138413) (← links)
- Reduced axonal transport and increased excitotoxic retinal ganglion cell degeneration in mice transgenic for human mutant P301S tau. (Q34230291) (← links)
- The nucleotide-binding state of microtubules modulates kinesin processivity and the ability of Tau to inhibit kinesin-mediated transport (Q35604721) (← links)
- Pathogenic forms of tau inhibit kinesin-dependent axonal transport through a mechanism involving activation of axonal phosphotransferases (Q36082071) (← links)
- Tau Hyperphosphorylation and Oxidative Stress, a Critical Vicious Circle in Neurodegenerative Tauopathies? (Q36241505) (← links)
- Single-molecule motility: statistical analysis and the effects of track length on quantification of processive motion (Q36939757) (← links)
- Tau pathology-mediated presynaptic dysfunction (Q36951523) (← links)
- Combined optical micromanipulation and interferometric topography (COMMIT). (Q37057619) (← links)
- Dissociation of tau toxicity and phosphorylation: role of GSK-3beta, MARK and Cdk5 in a Drosophila model (Q37102130) (← links)
- Quantitative and functional analyses of spastin in the nervous system: implications for hereditary spastic paraplegia (Q37219413) (← links)
- Global axonal transport rates are unaltered in htau mice in vivo (Q37284948) (← links)
- Phosphorylated Tau interacts with c-Jun N-terminal kinase-interacting protein 1 (JIP1) in Alzheimer disease (Q37344062) (← links)
- Axonal transport defects in neurodegenerative diseases (Q37614744) (← links)
- Brief update on different roles of tau in neurodegeneration (Q37893588) (← links)
- Axonal degeneration in Alzheimer's disease: when signaling abnormalities meet the axonal transport system. (Q38020618) (← links)
- Axonal transport deficits and neurodegenerative diseases (Q38077679) (← links)
- MAPping out distribution routes for kinesin couriers (Q38116595) (← links)
- Transport and diffusion of Tau protein in neurons (Q38200941) (← links)
- Protein phosphorylation in neurodegeneration: friend or foe? (Q38214594) (← links)
- Axonal transport defects in Alzheimer's disease (Q38232821) (← links)
- Effect of Pin1 or microtubule binding on dephosphorylation of FTDP-17 mutant Tau (Q38354586) (← links)
- Mathematical models of α-synuclein transport in axons (Q38981601) (← links)
- Levels of kinesin light chain and dynein intermediate chain are reduced in the frontal cortex in Alzheimer's disease: implications for axoplasmic transport. (Q39441258) (← links)
- Is abnormal axonal transport a cause, a contributing factor or a consequence of the neuronal pathology in Alzheimer's disease? (Q39909606) (← links)
- Pseudophosphorylation of tau at S422 enhances SDS-stable dimer formation and impairs both anterograde and retrograde fast axonal transport (Q41484754) (← links)
- Effects of eribulin, vincristine, paclitaxel and ixabepilone on fast axonal transport and kinesin-1 driven microtubule gliding: implications for chemotherapy-induced peripheral neuropathy (Q42112858) (← links)
- Analysis of isoform-specific tau aggregates suggests a common toxic mechanism involving similar pathological conformations and axonal transport inhibition (Q42714396) (← links)
- Axonal Degeneration in Tauopathies: Disease Relevance and Underlying Mechanisms (Q46985283) (← links)
- The axonal transport motor kinesin-2 navigates microtubule obstacles via protofilament switching (Q48155017) (← links)
- Effect of taxoid and nontaxoid site microtubule-stabilizing agents on axonal transport of mitochondria in untransfected and ECFP-htau40-transfected rat cortical neurons in culture (Q48801607) (← links)
- Biosensing MAPs as "roadblocks": kinesin-based functional analysis of tau protein isoforms and mutants using suspended microtubules (sMTs). (Q50896489) (← links)
- Mutant spastin proteins promote deficits in axonal transport through an isoform-specific mechanism involving casein kinase 2 activation. (Q51063673) (← links)
- Vesicular Axonal Transport is Modified In Vivo by Tau Deletion or Overexpression in Drosophila. (Q51735877) (← links)
- Important neuronal toxicity of microtubule-bound Tau in vivo in Drosophila. (Q52723359) (← links)
- A coupled model of fast axonal transport of organelles and slow axonal transport of tau protein (Q57435821) (← links)
- Cytoskeletal changes in diseases of the nervous system (Q58488843) (← links)
- Tau isoform-specific modulation of kinesin-driven microtubule gliding rates and trajectories as determined with tau-stabilized microtubules (Q82910022) (← links)
- Tau and Axonal Transport Misregulation in Tauopathies (Q89871454) (← links)
- Ablation of cytoskeletal scaffolding proteins, Band 4.1B and Whirlin, leads to cerebellar purkinje axon pathology and motor dysfunction (Q93185930) (← links)