Pages that link to "Q48214725"

The following pages link to Abnormal expression of SNS/PN3 sodium channel in cerebellar Purkinje cells following loss of myelin in the taiep rat. (Q48214725):

Displaying 24 items.

• Functional reciprocity between Na+ channel Nav1.6 and beta1 subunits in the coordinated regulation of excitability and neurite outgrowth (Q33719761) (← links)
• Reduced thermal sensitivity and Nav1.8 and TRPV1 channel expression in sensory neurons of aged mice (Q33739335) (← links)
• The neuron as a dynamic electrogenic machine: modulation of sodium-channel expression as a basis for functional plasticity in neurons (Q33867569) (← links)
• Sodium channels and their genes: dynamic expression in the normal nervous system, dysregulation in disease states(1). (Q34105765) (← links)
• Transcriptional channelopathies: an emerging class of disorders (Q34354397) (← links)
• Expression and distribution of voltage-gated sodium channels in the cerebellum (Q34535787) (← links)
• Sensory neuron-specific sodium channel SNS is abnormally expressed in the brains of mice with experimental allergic encephalomyelitis and humans with multiple sclerosis (Q35325861) (← links)
• Cerebellar dysfunction in multiple sclerosis: evidence for an acquired channelopathy (Q36015265) (← links)
• An emerging role for voltage-gated Na+ channels in cellular migration: regulation of central nervous system development and potentiation of invasive cancers (Q36049612) (← links)
• Axonal conduction and injury in multiple sclerosis: the role of sodium channels (Q36657811) (← links)
• Organotypic cultures of cerebellar slices as a model to investigate demyelinating disorders (Q39437479) (← links)
• Glial-derived neurotrophic factor upregulates expression of functional SNS and NaN sodium channels and their currents in axotomized dorsal root ganglion neurons. (Q40907012) (← links)
• A channelopathy contributes to cerebellar dysfunction in a model of multiple sclerosis. (Q43773926) (← links)
• Mutant Taiep rats exhibit an increase in D1 binding in basal ganglia (Q44212251) (← links)
• Sodium channels contribute to microglia/macrophage activation and function in EAE and MS. (Q45076768) (← links)
• Abnormal Purkinje cell activity in vivo in experimental allergic encephalomyelitis (Q47670118) (← links)
• Temporal Course of Upregulation of Nav1.8 in Purkinje Neurons Parallels the Progression of Clinical Deficit in Experimental Allergic Encephalomyelitis (Q48177322) (← links)
• Tetrodotoxin-resistant sodium channels Na(v)1.8/SNS and Na(v)1.9/NaN in afferent neurons innervating urinary bladder in control and spinal cord injured rats (Q48397699) (← links)
• Expression of Nav1.8 sodium channels perturbs the firing patterns of cerebellar Purkinje cells (Q48422070) (← links)
• Axotomy does not up-regulate expression of sodium channel Na(v)1.8 in Purkinje cells (Q48607314) (← links)
• Reactive microglia in dysmyelination and demyelination (Q48912211) (← links)
• Selective expression of a persistent tetrodotoxin-resistant Na+ current and NaV1.9 subunit in myenteric sensory neurons. (Q51300821) (← links)
• Changes in expression of two tetrodotoxin-resistant sodium channels and their currents in dorsal root ganglion neurons after sciatic nerve injury but not rhizotomy. (Q51377717) (← links)
• Paranodal interactions regulate expression of sodium channel subtypes and provide a diffusion barrier for the node of Ranvier. (Q52551533) (← links)