Pages that link to "Q49165175"
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The following pages link to Calpains are activated in necrotic fibers from mdx dystrophic mice (Q49165175):
Displaying 50 items.
- Hypernitrosylated ryanodine receptor calcium release channels are leaky in dystrophic muscle (Q24308714) (← links)
- The pathogenic activation of calpain: a marker and mediator of cellular toxicity and disease states (Q24643397) (← links)
- Myosin accumulation and striated muscle myopathy result from the loss of muscle RING finger 1 and 3 (Q24673171) (← links)
- Emerging role of protein kinase C in energy homeostasis: A brief overview (Q27022044) (← links)
- Rescue of dystrophic skeletal muscle by PGC-1α involves a fast to slow fiber type shift in the mdx mouse (Q27310125) (← links)
- Genetic evidence in the mouse solidifies the calcium hypothesis of myofiber death in muscular dystrophy (Q28085320) (← links)
- Calpains and muscular dystrophies (Q28144209) (← links)
- Alteration of excitation-contraction coupling mechanism in extensor digitorum longus muscle fibres of dystrophic mdx mouse and potential efficacy of taurine (Q28354192) (← links)
- Calpain/calpastatin activities and substrate depletion patterns during hindlimb unweighting and reweighting in skeletal muscle (Q28570069) (← links)
- Mice lacking Homer 1 exhibit a skeletal myopathy characterized by abnormal transient receptor potential channel activity (Q28593398) (← links)
- Characteristics of skeletal muscle in mdx mutant mice (Q33644573) (← links)
- Enhanced Ca²⁺ influx from STIM1-Orai1 induces muscle pathology in mouse models of muscular dystrophy. (Q33784460) (← links)
- Understanding dystrophinopathies: an inventory of the structural and functional consequences of the absence of dystrophin in muscles of the mdx mouse (Q33836366) (← links)
- Stable expression of calpain 3 from a muscle transgene in vivo: immature muscle in transgenic mice suggests a role for calpain 3 in muscle maturation. (Q34066168) (← links)
- Pharmacologic management of Duchenne muscular dystrophy: target identification and preclinical trials (Q34152280) (← links)
- Bowman-Birk inhibitor attenuates dystrophic pathology in mdx mice. (Q34305281) (← links)
- Altered cross-bridge properties in skeletal muscle dystrophies (Q34335939) (← links)
- A proteasome inhibitor fails to attenuate dystrophic pathology in mdx mice (Q34367192) (← links)
- Expression of three calpain isoform genes in human skeletal muscles (Q34465707) (← links)
- Long-term blocking of calcium channels in mdx mice results in differential effects on heart and skeletal muscle (Q34469185) (← links)
- Thimerosal-induced apoptosis in mouse C2C12 myoblast cells occurs through suppression of the PI3K/Akt/survivin pathway (Q34473965) (← links)
- Mitigation of muscular dystrophy in mice by SERCA overexpression in skeletal muscle (Q34627089) (← links)
- Apoptosis and muscle fibre loss in neuromuscular disorders (Q34808580) (← links)
- Effects of Dantrolene Therapy on Disease Phenotype in Dystrophin Deficient mdx Mice. (Q35051963) (← links)
- Regulation of the calpain and ubiquitin-proteasome systems in a canine model of muscular dystrophy. (Q35219135) (← links)
- Elevated subsarcolemmal Ca2+ in mdx mouse skeletal muscle fibers detected with Ca2+-activated K+ channels. (Q35700706) (← links)
- Characterization of monoclonal antibodies to calpain 3 and protein expression in muscle from patients with limb-girdle muscular dystrophy type 2A (Q35753488) (← links)
- Troponin T cross-linking in human apoptotic cardiomyocytes (Q35764793) (← links)
- Proteasome inhibitor (MG-132) treatment of mdx mice rescues the expression and membrane localization of dystrophin and dystrophin-associated proteins (Q35792179) (← links)
- Eosinophilia of dystrophin-deficient muscle is promoted by perforin-mediated cytotoxicity by T cell effectors (Q35810072) (← links)
- Contractile function, sarcolemma integrity, and the loss of dystrophin after skeletal muscle eccentric contraction-induced injury (Q35811023) (← links)
- The muscular dystrophies: from genes to therapies (Q35834674) (← links)
- Regulation of cytosolic calcium in skeletal muscle cells of the mdx mouse under conditions of stress (Q35873884) (← links)
- Increased resting intracellular calcium modulates NF-κB-dependent inducible nitric-oxide synthase gene expression in dystrophic mdx skeletal myotubes (Q36033118) (← links)
- Progressive muscular dystrophy in alpha-sarcoglycan-deficient mice (Q36276730) (← links)
- A nitric oxide synthase transgene ameliorates muscular dystrophy in mdx mice (Q36294012) (← links)
- A novel mechanism of myocyte degeneration involving the Ca2+-permeable growth factor-regulated channel (Q36323554) (← links)
- Vascular-targeted therapies for Duchenne muscular dystrophy (Q36835180) (← links)
- A purine nucleoside phosphorylase (PNP) inhibitor induces apoptosis via caspase-3-like protease activity in MOLT-4 T cells (Q36894793) (← links)
- Leupeptin-based inhibitors do not improve the mdx phenotype. (Q37174711) (← links)
- Myonuclear apoptosis in dystrophic mdx muscle occurs by perforin-mediated cytotoxicity (Q37368276) (← links)
- Calcium influx is sufficient to induce muscular dystrophy through a TRPC-dependent mechanism (Q37419160) (← links)
- Canine models of Duchenne muscular dystrophy and their use in therapeutic strategies (Q37548291) (← links)
- Calpain-mediated proteolysis of tropomodulin isoforms leads to thin filament elongation in dystrophic skeletal muscle (Q37635343) (← links)
- The dystrophin-glycoprotein complex in the prevention of muscle damage (Q37946876) (← links)
- Ryanodine receptor patents (Q38054907) (← links)
- The physiological response of protease inhibition in dystrophic muscle (Q38104503) (← links)
- Exercise and muscular dystrophy: implications and analysis of effects on musculoskeletal and cardiovascular systems (Q38111711) (← links)
- Myoblast attachment and spreading are regulated by different patterns by ubiquitous calpains (Q38315548) (← links)
- What has the mdx mouse model of Duchenne muscular dystrophy contributed to our understanding of this disease? (Q38351283) (← links)