Pages that link to "Q28144446"
Jump to navigation
Jump to search
The following pages link to Aspartyl beta -hydroxylase (Asph) and an evolutionarily conserved isoform of Asph missing the catalytic domain share exons with junctin (Q28144446):
Displaying 50 items.
- Aspartate-beta-hydroxylase (Q21499084) (← links)
- Binding of proprotein convertase subtilisin/kexin type 9 to epidermal growth factor-like repeat A of low density lipoprotein receptor decreases receptor recycling and increases degradation (Q24303660) (← links)
- Substrate requirements of the oxygen-sensing asparaginyl hydroxylase factor-inhibiting hypoxia-inducible factor (Q24305504) (← links)
- Absence of triadin, a protein of the calcium release complex, is responsible for cardiac arrhythmia with sudden death in human (Q24307681) (← links)
- Junctin and the histidine-rich Ca2+ binding protein: potential roles in heart failure and arrhythmogenesis (Q24320070) (← links)
- Junctate is a Ca2+-sensing structural component of Orai1 and stromal interaction molecule 1 (STIM1) (Q24322166) (← links)
- Collagen and calcium-binding EGF domains 1 is frequently inactivated in ovarian cancer by aberrant promoter hypermethylation and modulates cell migration and survival (Q24655625) (← links)
- Junctate is a key element in calcium entry induced by activation of InsP3 receptors and/or calcium store depletion (Q24684783) (← links)
- Antisense oligodeoxynucleotides directed against aspartyl (asparaginyl) β-hydroxylase suppress migration of cholangiocarcinoma cells (Q28201030) (← links)
- Role of the aspartyl-asparaginyl-beta-hydroxylase gene in neuroblastoma cell motility (Q28214662) (← links)
- Expression of aspartyl beta-hydroxylase and its clinicopathological significance in hepatocellular carcinoma (Q28286169) (← links)
- Absence of post-translational aspartyl beta-hydroxylation of epidermal growth factor domains in mice leads to developmental defects and an increased incidence of intestinal neoplasia (Q28509997) (← links)
- Genetic and phenotypic analysis of Tcm, a mutation affecting early eye development (Q28594664) (← links)
- Distribution and prediction of catalytic domains in 2-oxoglutarate dependent dioxygenases (Q30525690) (← links)
- Differential growth factor regulation of aspartyl-(asparaginyl)-beta-hydroxylase family genes in SH-Sy5y human neuroblastoma cells (Q33265938) (← links)
- Upstream stimulatory factors are involved in the P1 promoter directed transcription of the A beta H-J-J locus (Q33393444) (← links)
- Development of a novel anti-human aspartyl-(asparaginyl) β-hydroxylase monoclonal antibody with diagnostic and therapeutic potential (Q33599455) (← links)
- Myocyte enhancer factor 2 activates promoter sequences of the human AbetaH-J-J locus, encoding aspartyl-beta-hydroxylase, junctin, and junctate (Q33724398) (← links)
- Prognostic value of aspartyl (asparaginyl)-beta-hydroxylase/humbug expression in non-small cell lung carcinoma (Q33944587) (← links)
- Ethanol inhibition of aspartyl-asparaginyl-beta-hydroxylase in fetal alcohol spectrum disorder: potential link to the impairments in central nervous system neuronal migration (Q33944605) (← links)
- Glycosylation regulates Notch signalling. (Q34271410) (← links)
- Triadin/Junctin double null mouse reveals a differential role for Triadin and Junctin in anchoring CASQ to the jSR and regulating Ca(2+) homeostasis (Q34329037) (← links)
- Role of aspartyl-(asparaginyl)-β-hydroxylase mediated notch signaling in cerebellar development and function (Q34335246) (← links)
- Loss of muscleblind-like 1 results in cardiac pathology and persistence of embryonic splice isoforms (Q34466715) (← links)
- Glial vascular degeneration in CADASIL. (Q34482302) (← links)
- Genetic analysis of complex traits in the emerging Collaborative Cross (Q35145475) (← links)
- Aspartyl-(asparaginyl) beta-hydroxylase, hypoxia-inducible factor-alpha and Notch cross-talk in regulating neuronal motility (Q35157948) (← links)
- Aspartate β-Hydroxylase expression promotes a malignant pancreatic cellular phenotype (Q35176052) (← links)
- Regulation of Aspartyl-(Asparaginyl)-β-Hydroxylase Protein Expression and Function by Phosphorylation in Hepatocellular Carcinoma Cells. (Q35653763) (← links)
- Potential Role of Phosphorylation as a Regulator of Aspartyl-(asparaginyl)-β-hydroxylase: Relevance to Infiltrative Spread of Human Hepatocellular Carcinoma (Q36170624) (← links)
- Novel sarco(endo)plasmic reticulum proteins and calcium homeostasis in striated muscles. (Q36227308) (← links)
- Optimized Expression and Purification of Humbug in Pichia pastoris and Its Monoclonal Antibody Preparation (Q36495723) (← links)
- Conservation of intrinsic disorder in protein domains and families: II. functions of conserved disorder (Q36869604) (← links)
- Regulatory roles of junctin in sarcoplasmic reticulum calcium cycling and myocardial function (Q36995841) (← links)
- siRNA inhibition of aspartyl-asparaginyl β-hydroxylase expression impairs cell motility, Notch signaling, and fetal growth (Q37023897) (← links)
- Minor sarcoplasmic reticulum membrane components that modulate excitation-contraction coupling in striated muscles (Q37464475) (← links)
- Aspartate-β-hydroxylase (ASPH): A potential therapeutic target in human malignant gliomas (Q37485171) (← links)
- Transcriptional activity and Sp 1/3 transcription factor binding to the P1 promoter sequences of the human AbetaH-J-J locus (Q38299426) (← links)
- Gamma interferon (IFN-γ) receptor restricts systemic dengue virus replication and prevents paralysis in IFN-α/β receptor-deficient mice (Q39279894) (← links)
- The tallysomycin biosynthetic gene cluster from Streptoalloteichus hindustanus E465-94 ATCC 31158 unveiling new insights into the biosynthesis of the bleomycin family of antitumor antibiotics (Q42608593) (← links)
- Overexpression of aspartyl-(asparaginyl)-beta-hydroxylase in hepatocellular carcinoma is associated with worse surgical outcome (Q43005656) (← links)
- Junctate, an inositol 1,4,5-triphosphate receptor associated protein, is present in rodent sperm and binds TRPC2 and TRPC5 but not TRPC1 channels. (Q46695331) (← links)
- Ethanol-Induced White Matter Atrophy Is Associated with Impaired Expression of Aspartyl-Asparaginyl-β-Hydroxylase (ASPH) and Notch Signaling in an Experimental Rat Model (Q47331366) (← links)
- Phosphorylation Modulates Aspartyl-(Asparaginyl)-β Hydroxylase Protein Expression, Catalytic Activity and Migration in Human Immature Neuronal Cerebellar Cells. (Q53092252) (← links)
- Aspartyl-asparagyl beta hydroxylase over-expression in human hepatoma is linked to activation of insulin-like growth factor and notch signaling mechanisms. (Q54590341) (← links)
- Hydroxylase Activity of ASPH Promotes Hepatocellular Carcinoma Metastasis Through Epithelial-to-Mesenchymal Transition Pathway. (Q55378664) (← links)
- Recent advances in research on aspartate β-hydroxylase (ASPH) in pancreatic cancer: A brief update (Q59329484) (← links)
- Overexpression of junctate induces cardiac hypertrophy and arrhythmia via altered calcium handling (Q80915724) (← links)
- Prometastatic Secretome Trafficking via Exosomes Initiates Pancreatic Cancer Pulmonary Metastasis (Q90127696) (← links)
- Aspartate β-hydroxylase as a target for cancer therapy (Q98564461) (← links)