Pages that link to "Q36412518"
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The following pages link to The Na+ channel inactivation gate is a molecular complex: a novel role of the COOH-terminal domain. (Q36412518):
Displaying 50 items.
- Phosphorylation of the A-kinase-anchoring protein Yotiao contributes to protein kinase A regulation of a heart potassium channel (Q24307360) (← links)
- Solution NMR structure of Apo-calmodulin in complex with the IQ motif of human cardiac sodium channel NaV1.5 (Q24317459) (← links)
- Cell membrane expression of cardiac sodium channel Na(v)1.5 is modulated by alpha-actinin-2 interaction (Q24322255) (← links)
- Sodium channel dysfunction in intractable childhood epilepsy with generalized tonic-clonic seizures (Q24544180) (← links)
- A C-terminal skeletal muscle sodium channel mutation associated with myotonia disrupts fast inactivation (Q24544300) (← links)
- Molecular basis of ranolazine block of LQT-3 mutant sodium channels: evidence for site of action (Q24671796) (← links)
- The hitchhiker's guide to the voltage-gated sodium channel galaxy (Q26771787) (← links)
- Protein assemblies of sodium and inward rectifier potassium channels control cardiac excitability and arrhythmogenesis (Q26830051) (← links)
- Voltage-gated sodium channel-associated proteins and alternative mechanisms of inactivation and block (Q26991734) (← links)
- Solution Structure of the NaV1.2 C-terminal EF-hand Domain (Q27653359) (← links)
- Crystal Structure of a Fibroblast Growth Factor Homologous Factor (FHF) Defines a Conserved Surface on FHFs for Binding and Modulation of Voltage-gated Sodium Channels (Q27655335) (← links)
- Crystallographic basis for calcium regulation of sodium channels (Q27677258) (← links)
- Cardiac sodium channel Na(v)1.5 and interacting proteins: Physiology and pathophysiology (Q28258096) (← links)
- Post-translational modifications of the cardiac Na channel: contribution of CaMKII-dependent phosphorylation to acquired arrhythmias (Q28292930) (← links)
- Fibroblast growth factor homologous factors control neuronal excitability through modulation of voltage-gated sodium channels (Q28588873) (← links)
- Pathogenic mechanism of recurrent mutations of SCN8A in epileptic encephalopathy (Q29347550) (← links)
- The importance of serine 161 in the sodium channel beta3 subunit for modulation of Na(V)1.2 gating (Q30430912) (← links)
- Inherited and acquired vulnerability to ventricular arrhythmias: cardiac Na+ and K+ channels (Q33152183) (← links)
- Distinct functional defect of three novel Brugada syndrome related cardiac sodium channel mutations (Q33157184) (← links)
- A common SCN5A polymorphism modulates the biophysical defects of SCN5A mutations (Q33159495) (← links)
- Na+ channel regulation by Ca2+/calmodulin and Ca2+/calmodulin-dependent protein kinase II in guinea-pig ventricular myocytes (Q33573637) (← links)
- Cardiac Na Channels: Structure to Function (Q33583786) (← links)
- Sodium channel carboxyl-terminal residue regulates fast inactivation (Q33726588) (← links)
- Calcium-dependent regulation of the voltage-gated sodium channel hH1: intrinsic and extrinsic sensors use a common molecular switch (Q34574396) (← links)
- Mechanisms of a human skeletal myotonia produced by mutation in the C-terminus of NaV1.4: is Ca2+ regulation defective? (Q35063865) (← links)
- Functional characterization of a novel frameshift mutation in the C-terminus of the Nav1.5 channel underlying a Brugada syndrome with variable expression in a Spanish family (Q35074338) (← links)
- Post-transcriptional silencing of SCN1B and SCN2B genes modulates late sodium current in cardiac myocytes from normal dogs and dogs with chronic heart failure (Q35395971) (← links)
- Arrhythmogenic Biophysical Phenotype for SCN5A Mutation S1787N Depends upon Splice Variant Background and Intracellular Acidosis (Q35553358) (← links)
- Determining the Advantages, Costs, and Trade-Offs of a Novel Sodium Channel Mutation in the Copepod Acartia hudsonica to Paralytic Shellfish Toxins (PST). (Q35663867) (← links)
- Novel heterozygous mutation c.4282G>T in the SCN5A gene in a family with Brugada syndrome (Q35754059) (← links)
- Sodium channel inactivation goes with the flow (Q35822660) (← links)
- Computational biology in the study of cardiac ion channels and cell electrophysiology. (Q36015209) (← links)
- A novel C-terminal truncation SCN5A mutation from a patient with sick sinus syndrome, conduction disorder and ventricular tachycardia (Q36178742) (← links)
- Sodium channel inactivation: molecular determinants and modulation (Q36267779) (← links)
- Perturbation of sodium channel structure by an inherited Long QT Syndrome mutation. (Q36455413) (← links)
- Sodium channel inactivation in heart: a novel role of the carboxy-terminal domain (Q36474163) (← links)
- Modulation of cardiac Na(+) and Ca(2+) currents by CaM and CaMKII. (Q36474167) (← links)
- Late sodium current in failing heart: friend or foe? (Q36496273) (← links)
- Multiple pore conformations driven by asynchronous movements of voltage sensors in a eukaryotic sodium channel (Q36582161) (← links)
- Voltage-gated sodium channels: action players with many faces (Q36652537) (← links)
- Role of sodium channels in propagation in heart muscle: how subtle genetic alterations result in major arrhythmic disorders (Q36822944) (← links)
- Late sodium current is a new therapeutic target to improve contractility and rhythm in failing heart (Q36954069) (← links)
- Pathophysiology of the cardiac late Na current and its potential as a drug target (Q37276416) (← links)
- The external pore loop interacts with S6 and S3-S4 linker in domain 4 to assume an essential role in gating control and anticonvulsant action in the Na(+) channel (Q37280509) (← links)
- Late Na+ current produced by human cardiac Na+ channel isoform Nav1.5 is modulated by its beta1 subunit. (Q37347227) (← links)
- NaChBac: the long lost sodium channel ancestor (Q37902637) (← links)
- Biology of cardiac sodium channel Nav1.5 expression (Q37936683) (← links)
- Voltage-gated sodium channels: biophysics, pharmacology, and related channelopathies. (Q38026557) (← links)
- Seeing the forest through the trees: towards a unified view on physiological calcium regulation of voltage-gated sodium channels (Q38071198) (← links)
- Cardiac sodium channelopathy associated with SCN5A mutations: electrophysiological, molecular and genetic aspects (Q38118751) (← links)