Pages that link to "Q37095372"
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The following pages link to The bile acid receptor TGR5 does not interact with β-arrestins or traffic to endosomes but transmits sustained signals from plasma membrane rafts. (Q37095372):
Displaying 42 items.
- Transactivation of Epidermal Growth Factor Receptor by G Protein-Coupled Receptors: Recent Progress, Challenges and Future Research (Q26771727) (← links)
- Can residence time offer a useful strategy to target agonist drugs for sustained GPCR responses? (Q26801424) (← links)
- Unravelling the molecular complexity of GPCR-mediated EGFR transactivation using functional genomics approaches (Q27008332) (← links)
- Understanding the Functional Plasticity in Neural Networks of the Basal Ganglia in Cocaine Use Disorder: A Role for Allosteric Receptor-Receptor Interactions in A2A-D2 Heteroreceptor Complexes (Q28071958) (← links)
- HEK-293 cells expressing the cystic fibrosis transmembrane conductance regulator (CFTR): a model for studying regulation of Cl- transport (Q30609337) (← links)
- Using nanoBRET and CRISPR/Cas9 to monitor proximity to a genome-edited protein in real-time (Q33784472) (← links)
- A GPBAR1 (TGR5) small molecule agonist shows specific inhibitory effects on myeloid cell activation in vitro and reduces experimental autoimmune encephalitis (EAE) in vivo. (Q33812525) (← links)
- Endothelin-converting enzyme 1 and β-arrestins exert spatiotemporal control of substance P-induced inflammatory signals (Q33931244) (← links)
- Allosteric modulation of M1 muscarinic acetylcholine receptor internalization and subcellular trafficking (Q34076059) (← links)
- Bile acid dysregulation, gut dysbiosis, and gastrointestinal cancer (Q34425909) (← links)
- Signalling profiles of H3 relaxin, H2 relaxin and R3(BΔ23-27)R/I5 acting at the relaxin family peptide receptor 3 (RXFP3) (Q34570793) (← links)
- PRESTO-Tango as an open-source resource for interrogation of the druggable human GPCRome. (Q35582384) (← links)
- Impaired Itching Perception in Murine Models of Cholestasis Is Supported by Dysregulation of GPBAR1 Signaling (Q35855133) (← links)
- Protein Kinase D and Gβγ Subunits Mediate Agonist-evoked Translocation of Protease-activated Receptor-2 from the Golgi Apparatus to the Plasma Membrane (Q35974569) (← links)
- The G-Protein-Coupled Bile Acid Receptor Gpbar1 (TGR5) Inhibits Gastric Inflammation Through Antagonizing NF-κB Signaling Pathway (Q36363512) (← links)
- Prognostic and mechanistic potential of progesterone sulfates in intrahepatic cholestasis of pregnancy and pruritus gravidarum (Q36908093) (← links)
- Mutations of Vasopressin Receptor 2 Including Novel L312S Have Differential Effects on Trafficking. (Q37137434) (← links)
- Membrane bile acid receptor TGR5 predicts good prognosis in ampullary adenocarcinoma patients with hyperbilirubinemia (Q37255472) (← links)
- GPBA: a GPCR for bile acids and an emerging therapeutic target for disorders of digestion and sensation (Q37635259) (← links)
- Chenodeoxycholic acid activates NLRP3 inflammasome and contributes to cholestatic liver fibrosis (Q37708915) (← links)
- Neuro-humoral signalling by bile acids and the TGR5 receptor in the gastrointestinal tract (Q38194905) (← links)
- TGR5 in inflammation and cardiovascular disease (Q38197305) (← links)
- Biased signaling of protease-activated receptors (Q38214613) (← links)
- Chenodeoxycholic acid requires activation of EGFR, EPAC, and Ca2+ to stimulate CFTR-dependent Cl- secretion in human colonic T84 cells (Q38734395) (← links)
- TGR5 contributes to hepatic cystogenesis in rodents with polycystic liver diseases through cyclic adenosine monophosphate/Gαs signaling (Q38764445) (← links)
- Cardiac GPCR-Mediated EGFR Transactivation: Impact and Therapeutic Implications (Q38774086) (← links)
- TGR5 is essential for bile acid-dependent cholangiocyte proliferation in vivo and in vitro (Q38831400) (← links)
- GPCR Signaling and Trafficking: The Long and Short of It. (Q39020873) (← links)
- Structural assemblies of the di- and oligomeric G-protein coupled receptor TGR5 in live cells: an MFIS-FRET and integrative modelling study. (Q39197767) (← links)
- The Viral G Protein-Coupled Receptor ORF74 Hijacks β-Arrestins for Endocytic Trafficking in Response to Human Chemokines (Q39806707) (← links)
- The bile acid receptor TGR5 activates the TRPA1 channel to induce itch in mice. (Q39877298) (← links)
- Neutrophil Elastase Activates Protease-activated Receptor-2 (PAR2) and Transient Receptor Potential Vanilloid 4 (TRPV4) to Cause Inflammation and Pain. (Q41298460) (← links)
- Antagonism of the proinflammatory and pronociceptive actions of canonical and biased agonists of protease-activated receptor-2. (Q41341638) (← links)
- Molecular determinants of orexin receptor-arrestin-ubiquitin complex formation (Q41895362) (← links)
- Cathepsin S causes inflammatory pain via biased agonism of PAR2 and TRPV4. (Q42915550) (← links)
- Bile acid-microbiota crosstalk in gastrointestinal inflammation and carcinogenesis (Q47668921) (← links)
- The actions of relaxin family peptides on signal transduction pathways activated by the relaxin family peptide receptor RXFP4. (Q47868139) (← links)
- Signal transduction pathways activated by insulin-like peptide 5 at the relaxin family peptide RXFP4 receptor. (Q48012894) (← links)
- TUDCA: An Agonist of the Bile Acid Receptor GPBAR1/TGR5 With Anti-Inflammatory Effects in Microglial Cells. (Q48392414) (← links)
- In Silico Workflow for the Discovery of Natural Products Activating the G Protein-Coupled Bile Acid Receptor 1. (Q55710753) (← links)
- The exercise-inducible bile acid receptor Tgr5 improves skeletal muscle function in mice (Q57116409) (← links)
- The Effect of Cell Surface Expression and Linker Sequence on the Recruitment of Arrestin to the GIP Receptor (Q99237829) (← links)