Pages that link to "Q37394402"

The following pages link to Ts1Cje, a partial trisomy 16 mouse model for Down syndrome, exhibits learning and behavioral abnormalities (Q37394402):

Displaying 50 items.

• Primary and secondary transcriptional effects in the developing human Down syndrome brain and heart (Q21184170) ‎ (← links)
• Gene-dosage effects in Down syndrome and trisomic mouse models (Q21194863) ‎ (← links)
• The sequence of human chromosome 21 and implications for research into Down syndrome (Q21194900) ‎ (← links)
• Down syndrome: searching for the genetic culprits (Q22241596) ‎ (← links)
• On the promise of pharmacotherapies targeted at cognitive and neurodegenerative components of Down syndrome (Q22241936) ‎ (← links)
• Prospects for Improving Brain Function in Individuals with Down Syndrome (Q22252566) ‎ (← links)
• Molecular genetic analysis of Down syndrome (Q22252694) ‎ (← links)
• C21orf5, a novel human chromosome 21 gene, has a Caenorhabditis elegans ortholog (pad-1) required for embryonic patterning (Q24290120) ‎ (← links)
• Novel human BTB/POZ domain-containing zinc finger protein ZNF295 is directly associated with ZFP161 (Q24337190) ‎ (← links)
• Dissecting Alzheimer disease in Down syndrome using mouse models (Q26777254) ‎ (← links)
• Behavioral and Genetic Evidence for GIRK Channels in the CNS: Role in Physiology, Pathophysiology, and Drug Addiction (Q26783948) ‎ (← links)
• Mouse models of Down syndrome as a tool to unravel the causes of mental disabilities (Q26863005) ‎ (← links)
• Down Syndrome Cognitive Phenotypes Modeled in Mice Trisomic for All HSA 21 Homologues (Q27300997) ‎ (← links)
• Chromosome 21: from sequence to applications (Q28191044) ‎ (← links)
• The differentially expressed C21orf5 gene in the medial temporal-lobe system could play a role in mental retardation in Down syndrome and transgenic mice (Q28206602) ‎ (← links)
• Chromosome 21 and down syndrome: from genomics to pathophysiology (Q28290257) ‎ (← links)
• Animal models of intellectual disability: towards a translational approach (Q28389500) ‎ (← links)
• App gene dosage modulates endosomal abnormalities of Alzheimer's disease in a segmental trisomy 16 mouse model of down syndrome (Q28511479) ‎ (← links)
• Dosage of the Abcg1-U2af1 region modifies locomotor and cognitive deficits observed in the Tc1 mouse model of Down syndrome (Q28543589) ‎ (← links)
• Down syndrome cell adhesion molecule DSCAM mediates homophilic intercellular adhesion (Q28611501) ‎ (← links)
• The use of mouse models to understand and improve cognitive deficits in Down syndrome (Q30474023) ‎ (← links)
• Behavioral validation of the Ts65Dn mouse model for Down syndrome of a genetic background free of the retinal degeneration mutation Pde6b(rd1). (Q30477664) ‎ (← links)
• Alzheimer disease models and human neuropathology: similarities and differences (Q30499574) ‎ (← links)
• PCP4 (PEP19) overexpression induces premature neuronal differentiation associated with Ca(2+) /calmodulin-dependent kinase II-δ activation in mouse models of Down syndrome (Q30668603) ‎ (← links)
• The mouse brain transcriptome by SAGE: differences in gene expression between P30 brains of the partial trisomy 16 mouse model of Down syndrome (Ts65Dn) and normals (Q30976629) ‎ (← links)
• Trisomy for the Down syndrome 'critical region' is necessary but not sufficient for brain phenotypes of trisomic mice. (Q31104439) ‎ (← links)
• An additional human chromosome 21 causes suppression of neural fate of pluripotent mouse embryonic stem cells in a teratoma model (Q33307796) ‎ (← links)
• Impairments in motor coordination without major changes in cerebellar plasticity in the Tc1 mouse model of Down syndrome (Q33404261) ‎ (← links)
• A chromosome 21 critical region does not cause specific Down syndrome phenotypes. (Q33605833) ‎ (← links)
• Gene network disruptions and neurogenesis defects in the adult Ts1Cje mouse model of Down syndrome (Q33641252) ‎ (← links)
• Maternal choline supplementation improves spatial learning and adult hippocampal neurogenesis in the Ts65Dn mouse model of Down syndrome (Q33643589) ‎ (← links)
• Mouse autosomal trisomy: two's company, three's a crowd. (Q33650529) ‎ (← links)
• Meiotic behavior of aneuploid chromatin in mouse models of Down syndrome. (Q33767425) ‎ (← links)
• Gene expression profiling in a mouse model identifies fetal liver- and placenta-derived potential biomarkers for Down Syndrome screening. (Q33886427) ‎ (← links)
• Meta-analysis of heterogeneous Down Syndrome data reveals consistent genome-wide dosage effects related to neurological processes (Q33898516) ‎ (← links)
• Segmental trisomy of chromosome 17: a mouse model of human aneuploidy syndromes (Q33927713) ‎ (← links)
• An aneuploid mouse strain carrying human chromosome 21 with Down syndrome phenotypes (Q33990273) ‎ (← links)
• Functional transcriptome analysis of the postnatal brain of the Ts1Cje mouse model for Down syndrome reveals global disruption of interferon-related molecular networks (Q34012056) ‎ (← links)
• Brain phenotype of transgenic mice overexpressing cystathionine β-synthase (Q34130338) ‎ (← links)
• An extra human chromosome 21 reduces mlc-2a expression in chimeric mice and Down syndrome (Q34135319) ‎ (← links)
• Too much of a good thing: mechanisms of gene action in Down syndrome. (Q34137795) ‎ (← links)
• Genotype-phenotype correlations in Down syndrome identified by array CGH in 30 cases of partial trisomy and partial monosomy chromosome 21 (Q34325983) ‎ (← links)
• The complex SNP and CNV genetic architecture of the increased risk of congenital heart defects in Down syndrome. (Q34352054) ‎ (← links)
• Murine models for Down syndrome (Q34375884) ‎ (← links)
• Engineering chromosomal rearrangements in mice (Q34389499) ‎ (← links)
• Controlled somatic and germline copy number variation in the mouse model (Q34479642) ‎ (← links)
• NFAT dysregulation by increased dosage of DSCR1 and DYRK1A on chromosome 21. (Q34504363) ‎ (← links)
• Emerging roles for G protein-gated inwardly rectifying potassium (GIRK) channels in health and disease. (Q34643934) ‎ (← links)
• Animal models of mental retardation: from gene to cognitive function. (Q35121791) ‎ (← links)
• Understanding mental retardation in Down's syndrome using trisomy 16 mouse models (Q35204307) ‎ (← links)