Pages that link to "Q38326990"
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The following pages link to Enhanced laminin binding by alpha-dystroglycan after enzymatic deglycosylation (Q38326990):
Displaying 43 items.
- Dystroglycan function requires xylosyl- and glucuronyltransferase activities of LARGE (Q24301093) (← links)
- O-Mannosylation and human disease (Q27014986) (← links)
- Crystal Structure and Cell Surface Anchorage Sites of Laminin 1LG4-5 (Q27643849) (← links)
- Identification of new dystroglycan complexes in skeletal muscle (Q28535211) (← links)
- Brain alpha-dystroglycan displays unique glycoepitopes and preferential binding to laminin-10/11 (Q28589413) (← links)
- Structural basis of laminin binding to the LARGE glycans on dystroglycan (Q31121747) (← links)
- Identification of alpha-dystroglycan binding sequences in the laminin alpha2 chain LG4-5 module (Q33678854) (← links)
- The dystroglycanopathies: the new disorders of O-linked glycosylation (Q33813487) (← links)
- Adeno-associated virus-mediated overexpression of LARGE rescues α-dystroglycan function in dystrophic mice with mutations in the fukutin-related protein (Q34013566) (← links)
- Site mapping and characterization of O-glycan structures on alpha-dystroglycan isolated from rabbit skeletal muscle. (Q34042753) (← links)
- O-mannosyl phosphorylation of alpha-dystroglycan is required for laminin binding. (Q34090655) (← links)
- Glycoproteomic characterization of recombinant mouse α-dystroglycan (Q34126184) (← links)
- Like-acetylglucosaminyltransferase (LARGE)-dependent modification of dystroglycan at Thr-317/319 is required for laminin binding and arenavirus infection. (Q34223029) (← links)
- miRNA-based buffering of the cobblestone-lissencephaly-associated extracellular matrix receptor dystroglycan via its alternative 3'-UTR. (Q34349564) (← links)
- Phosphorylation within the cysteine-rich region of dystrophin enhances its association with β-dystroglycan and identifies a potential novel therapeutic target for skeletal muscle wasting (Q34575694) (← links)
- Mutational and functional analysis of Large in a novel CHO glycosylation mutant (Q34983110) (← links)
- Glycomic analyses of mouse models of congenital muscular dystrophy (Q35065134) (← links)
- Increased sialylation as a phenomenon in accommodation of the parasitic nematode Trichinella spiralis (Owen, 1835) in skeletal muscle fibres (Q35776470) (← links)
- Absence of post-phosphoryl modification in dystroglycanopathy mouse models and wild-type tissues expressing non-laminin binding form of α-dystroglycan (Q35841883) (← links)
- Old World and clade C New World arenaviruses mimic the molecular mechanism of receptor recognition used by alpha-dystroglycan's host-derived ligands (Q35857739) (← links)
- Dystroglycan Depletion Impairs Actin-Dependent Functions of Differentiated Kasumi-1 Cells (Q35858815) (← links)
- Mechanisms of disease: congenital muscular dystrophies-glycosylation takes center stage (Q36577280) (← links)
- The o-mannosylation pathway: glycosyltransferases and proteins implicated in congenital muscular dystrophy (Q36666131) (← links)
- ISPD produces CDP-ribitol used by FKTN and FKRP to transfer ribitol phosphate onto α-dystroglycan (Q36919737) (← links)
- Dystroglycan glycosylation and muscular dystrophy. (Q37261423) (← links)
- Muscular dystrophies due to glycosylation defects (Q37329563) (← links)
- Deciphering the glycosylome of dystroglycanopathies using haploid screens for lassa virus entry (Q37572562) (← links)
- Integrin and dystroglycan compensate each other to mediate laminin-dependent basement membrane assembly and epiblast polarization. (Q37596648) (← links)
- Vertebrate protein glycosylation: diversity, synthesis and function (Q37601831) (← links)
- Protein O-mannosylation in animal development and physiology: from human disorders to Drosophila phenotypes (Q37723603) (← links)
- Biological role of dystroglycan in Schwann cell function and its implications in peripheral nervous system diseases (Q37772080) (← links)
- Lissencephaly with brainstem and cerebellar hypoplasia and congenital cataracts (Q38122354) (← links)
- Mammalian O-mannosylation pathway: glycan structures, enzymes, and protein substrates (Q38208557) (← links)
- Finding the sweet spot: assembly and glycosylation of the dystrophin-associated glycoprotein complex (Q38240078) (← links)
- Matriglycan: a novel polysaccharide that links dystroglycan to the basement membrane. (Q38424785) (← links)
- Disease mutations in CMP-sialic acid transporter SLC35A1 result in abnormal α-dystroglycan O-mannosylation, independent from sialic acid. (Q38924048) (← links)
- Initiation of mammalian O-mannosylation in vivo is independent of a consensus sequence and controlled by peptide regions within and upstream of the alpha-dystroglycan mucin domain (Q39985380) (← links)
- Dystroglycan and protein O-mannosyltransferases 1 and 2 are required to maintain integrity of Drosophila larval muscles. (Q40724004) (← links)
- Alterations of dystrophin-associated glycoproteins in the heart lacking dystrophin or dystrophin and utrophin (Q41919730) (← links)
- LARGE enzyme activity deciphered: a new therapeutic target for muscular dystrophies (Q42125324) (← links)
- Characterization of site-specific O-glycan structures within the mucin-like domain of alpha-dystroglycan from human skeletal muscle (Q43052323) (← links)
- O-glycosylation of the non-canonical T-cadherin from rabbit skeletal muscle by single mannose residues (Q43528757) (← links)
- HNK-1 Sulfotransferase modulates α-dystroglycan glycosylation by 3-O-sulfation of glucuronic acid on matriglycan (Q90154126) (← links)