Pages that link to "Q39486053"
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The following pages link to The transcription factor FOXM1 is a cellular target of the natural product thiostrepton (Q39486053):
Displaying 50 items.
- Cyclisation mechanisms in the biosynthesis of ribosomally synthesised and post-translationally modified peptides (Q26738949) (← links)
- Thiopeptide antibiotics: retrospective and recent advances (Q26852567) (← links)
- FOXM1: an emerging master regulator of DNA damage response and genotoxic agent resistance (Q26864654) (← links)
- Expanding the number of 'druggable' targets: non-enzymes and protein-protein interactions (Q27003966) (← links)
- Disabling Mitochondrial Peroxide Metabolism via Combinatorial Targeting of Peroxiredoxin 3 as an Effective Therapeutic Approach for Malignant Mesothelioma (Q27306784) (← links)
- Cell fate control by pioneer transcription factors (Q28079637) (← links)
- ThioFinder: a web-based tool for the identification of thiopeptide gene clusters in DNA sequences (Q28483962) (← links)
- FOXM1 is an oncogenic mediator in Ewing Sarcoma (Q28485291) (← links)
- Peroxiredoxin 3 is a redox-dependent target of thiostrepton in malignant mesothelioma cells (Q28728015) (← links)
- Identification of neuron selective androgen receptor inhibitors (Q33711488) (← links)
- Prognostic value of FOXM1 in solid tumors: a systematic review and meta-analysis (Q33762091) (← links)
- MELK is an oncogenic kinase essential for mitotic progression in basal-like breast cancer cells. (Q33762222) (← links)
- Binding induced RNA conformational changes control substrate recognition and catalysis by the thiostrepton resistance methyltransferase (Tsr). (Q34249082) (← links)
- Pioneer factors in hormone-dependent cancers (Q34272580) (← links)
- Ribosomally synthesized and post-translationally modified peptide natural products: overview and recommendations for a universal nomenclature (Q34312732) (← links)
- Down-regulation of FoxM1 by thiostrepton or small interfering RNA inhibits proliferation, transformation ability and angiogenesis, and induces apoptosis of nasopharyngeal carcinoma cells (Q34371119) (← links)
- Overexpression of FOXM1 is associated with metastases of nasopharyngeal carcinoma (Q34589178) (← links)
- Translational synthetic chemistry (Q34626958) (← links)
- Suppression of the FOXM1 transcriptional programme via novel small molecule inhibition (Q34649774) (← links)
- Targeting of mutant p53-induced FoxM1 with thiostrepton induces cytotoxicity and enhances carboplatin sensitivity in cancer cells (Q34956879) (← links)
- Identification of candidate B-lymphoma genes by cross-species gene expression profiling (Q35018604) (← links)
- ROS inhibitor N-acetyl-L-cysteine antagonizes the activity of proteasome inhibitors (Q35065984) (← links)
- Identification of FOXM1 as a therapeutic target in B-cell lineage acute lymphoblastic leukaemia (Q35197045) (← links)
- Glioblastoma multiforme formation and EMT: role of FoxM1 transcription factor (Q35234273) (← links)
- Unravelling the genomic targets of small molecules using high-throughput sequencing (Q35328171) (← links)
- A FOXM1 Dependent Mesenchymal-Epithelial Transition in Retinal Pigment Epithelium Cells (Q35677149) (← links)
- Combination with bortezomib enhances the antitumor effects of nanoparticle-encapsulated thiostrepton (Q35911674) (← links)
- MEIS2 is essential for neuroblastoma cell survival and proliferation by transcriptional control of M-phase progression (Q35966255) (← links)
- FoxM1 Directs STAT3 Expression Essential for Human Endometrial Stromal Decidualization (Q36022421) (← links)
- Thiostrepton interacts covalently with Rpt subunits of the 19S proteasome and proteasome substrates (Q36057485) (← links)
- Prognostic significance of FOXM1 expression and antitumor effect of FOXM1 inhibition in synovial sarcomas (Q36083264) (← links)
- FOXM1 is overexpressed in B-acute lymphoblastic leukemia (B-ALL) and its inhibition sensitizes B-ALL cells to chemotherapeutic drugs (Q36094104) (← links)
- Elucidating and engineering thiopeptide biosynthesis (Q36369930) (← links)
- FOXM1 is a downstream target of LPA and YAP oncogenic signaling pathways in high grade serous ovarian cancer (Q36413873) (← links)
- Expression of Forkhead box M1 in soft tissue leiomyosarcoma: Clinicopathologic and in vitro study using a newly established cell line. (Q36495922) (← links)
- Novel functions of FoxM1: from molecular mechanisms to cancer therapy (Q36657633) (← links)
- Recombinant thiopeptides containing noncanonical amino acids (Q36770573) (← links)
- Saturation mutagenesis of TsrA Ala4 unveils a highly mutable residue of thiostrepton A. (Q36781888) (← links)
- FOXM1 is a therapeutic target for high-risk multiple myeloma (Q36783627) (← links)
- FOXM1 is a molecular determinant of the mitogenic and invasive phenotype of anaplastic thyroid carcinoma (Q36799818) (← links)
- Thiostrepton Variants Containing a Contracted Quinaldic Acid Macrocycle Result from Mutagenesis of the Second Residue (Q36843584) (← links)
- Genome-wide mapping of FOXM1 binding reveals co-binding with estrogen receptor alpha in breast cancer cells (Q36872440) (← links)
- Englerin A Inhibits EWS-FLI1 DNA Binding in Ewing Sarcoma Cells (Q36876146) (← links)
- Co-targeting of Cyclooxygenase-2 and FoxM1 is a viable strategy in inducing anticancer effects in colorectal cancer cells (Q36883777) (← links)
- The dual inhibitory effect of thiostrepton on FoxM1 and EWS/FLI1 provides a novel therapeutic option for Ewing's sarcoma (Q37209489) (← links)
- Pathological Ace2-to-Ace enzyme switch in the stressed heart is transcriptionally controlled by the endothelial Brg1-FoxM1 complex (Q37281630) (← links)
- The Forkhead Box M1 protein regulates BRIP1 expression and DNA damage repair in epirubicin treatment (Q37417960) (← links)
- FOXM1 evokes 5-fluorouracil resistance in colorectal cancer depending on ABCC10. (Q37702756) (← links)
- Radical S-adenosylmethionine enzymes (Q37727698) (← links)
- Biosynthesis of thiopeptide antibiotics and their pathway engineering (Q38068336) (← links)