Conversion of p35 to p25 deregulates Cdk5 activity and promotes neurodegeneration (Q28140796)

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scientific article (publication date: 9 December 1999)

Language	Label	Description	Also known as
English	Conversion of p35 to p25 deregulates Cdk5 activity and promotes neurodegeneration	scientific article (publication date: 9 December 1999)

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scholarly article

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Conversion of p35 to p25 deregulates Cdk5 activity and promotes neurodegeneration (English)

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neurodegeneration

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author name string

G N Patrick

1

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L Zukerberg

2

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M Nikolic

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S de la Monte

4

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P Dikkes

5

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L H Tsai

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language of work or name

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publication date

9 December 1999

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402

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6762

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615-22

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A family of human cdc2-related protein kinases

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A brain-specific activator of cyclin-dependent kinase 5.

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Identification of the 23 kDa subunit of tau protein kinase II as a putative activator of cdk5 in bovine brain

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p35 is a neural-specific regulatory subunit of cyclin-dependent kinase 5

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The cdk5/p35 kinase is essential for neurite outgrowth during neuronal differentiation

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Evidence for the participation of the neuron-specific CDK5 activator P35 during laminin-enhanced axonal growth.

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Callosal axon guidance defects in p35(-/-) mice

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A novel disruption of cortical development in p35(-/-) mice distinct from reeler

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Mice lacking p35, a neuronal specific activator of Cdk5, display cortical lamination defects, seizures, and adult lethality

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Targeted disruption of the cyclin-dependent kinase 5 gene results in abnormal corticogenesis, neuronal pathology and perinatal death

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Cyclin-dependent kinase 5-deficient mice demonstrate novel developmental arrest in cerebral cortex

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Phosphorylation of DARPP-32 by Cdk5 modulates dopamine signalling in neurons

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Regulation of exocytosis by cyclin-dependent kinase 5 via phosphorylation of Munc18

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Tau in Alzheimer's disease.

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Tau protein pathology in neurodegenerative diseases

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Precursor of cdk5 activator, the 23 kDa subunit of tau protein kinase II: its sequence and developmental change in brain

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Identification of functional domains in the neuronal Cdk5 activator protein

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The p35/Cdk5 kinase is a neuron-specific Rac effector that inhibits Pak1 activity

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The role of cyclin-dependent kinase 5 and a novel regulatory subunit in regulating muscle differentiation and patterning

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p35, the neuronal-specific activator of cyclin-dependent kinase 5 (Cdk5) is degraded by the ubiquitin-proteasome pathway

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Possible role of tau protein kinases in pathogenesis of Alzheimer's disease.

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Abnormal Alzheimer-like phosphorylation of tau-protein by cyclin-dependent kinases cdk2 and cdk5

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Characterization of tau phosphorylation in glycogen synthase kinase-3beta and cyclin dependent kinase-5 activator (p23) transfected cells

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Phosphorylation by neuronal cdc2-like protein kinase promotes dimerization of Tau protein in vitro

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Alzheimer's disease-like phosphorylation of the microtubule-associated protein tau by glycogen synthase kinase-3 in transfected mammalian cells.

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The phosphorylation of tau: a critical stage in neurodevelopment and neurodegenerative processes.

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Phosphorylation of human high molecular weight neurofilament protein (hNF-H) by neuronal cyclin-dependent kinase 5 (cdk5)

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Phosphorylation of the high molecular weight neurofilament protein (NF-H) by Cdk5 and p35

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Differential cellular phosphorylation of neurofilament heavy side-arms by glycogen synthase kinase-3 and cyclin-dependent kinase-5.

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Tau domains, phosphorylation, and interactions with microtubules.

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Interaction of cyclin-dependent kinase 5 (Cdk5) and neuronal Cdk5 activator in bovine brain.

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Association of cyclin-dependent kinase 5 and its activator p35 with apoptotic cell death

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Increased expression of cyclin-dependent kinase 5 in induced apoptotic neuron death in rat substantia nigra

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Expression of cell cycle-related genes during neuronal apoptosis: is there a distinct pattern?

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Cyclin-dependent kinase 5 is associated with apoptotic cell death during development and tissue remodeling

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Site-specific phosphorylation of synapsin I by mitogen-activated protein kinase and Cdk5 and its effects on physiological functions

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Regulation of Munc-18/syntaxin 1A interaction by cyclin-dependent kinase 5 in nerve endings

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The distribution of tau in the mammalian central nervous system

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A neuronal antigen in the brains of Alzheimer patients

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Overexpression in neurons of human presenilin-1 or a presenilin-1 familial Alzheimer disease mutant does not enhance apoptosis.

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