Counterbalance between RB inactivation and miR-17-92 overexpression in reactive oxygen species and DNA damage induction in lung cancers (Q28251838)

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2 February 2020

title

Counterbalance between RB inactivation and miR-17-92 overexpression in reactive oxygen species and DNA damage induction in lung cancers (English)

1 reference

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2 February 2020

0 references

1 reference

reactive oxygen species

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1

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2 February 2020

Yasushi Yatabe

5

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2 February 2020

Takashi Takahashi

10

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2 February 2020

author name string

T Sato

2

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2 February 2020

N Sugito

3

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2 February 2020

Y Hosono

4

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2 February 2020

Y Matsuyama

6

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2 February 2020

T Yamaguchi

7

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2 February 2020

H Osada

8

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2 February 2020

M Suzuki

9

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2 February 2020

language of work or name

English

0 references

publication date

13 July 2009

1 reference

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2 February 2020

1 reference

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2 February 2020

volume

28

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2 February 2020

issue

38

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2 February 2020

page(s)

3371-3379

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A polycistronic microRNA cluster, miR-17-92, is overexpressed in human lung cancers and enhances cell proliferation

2 February 2020

cites work

1 reference

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RAS is regulated by the let-7 microRNA family

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Transgenic over-expression of the microRNA miR-17-92 cluster promotes proliferation and inhibits differentiation of lung epithelial progenitor cells

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Apoptosis induction by antisense oligonucleotides against miR-17-5p and miR-20a in lung cancers overexpressing miR-17-92

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c-Myc-regulated microRNAs modulate E2F1 expression

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E2F1-regulated microRNAs impair TGFbeta-dependent cell-cycle arrest and apoptosis in gastric cancer

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miR-17 and miR-20a temper an E2F1-induced G1 checkpoint to regulate cell cycle progression

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Identification of hypoxia-inducible factor-1 alpha as a novel target for miR-17-92 microRNA cluster

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Reduced expression of the let-7 microRNAs in human lung cancers in association with shortened postoperative survival

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let-7 regulates Dicer expression and constitutes a negative feedback loop

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Targeted deletion reveals essential and overlapping functions of the miR-17 through 92 family of miRNA clusters

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Alterations of the PPP2R1B gene in human lung and colon cancer

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DNA damage response as a candidate anti-cancer barrier in early human tumorigenesis

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Activation of the DNA damage checkpoint and genomic instability in human precancerous lesions

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gamma-H2AX dephosphorylation by protein phosphatase 2A facilitates DNA double-strand break repair

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53BP1 functions in an ATM-dependent checkpoint pathway that is constitutively activated in human cancer

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Novel NBS1 heterozygous germ line mutation causing MRE11-binding domain loss predisposes to common types of cancer

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An oncogene-induced DNA damage model for cancer development

21 January 2018

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MicroRNA-17-92 down-regulates expression of distinct targets in different B-cell lymphoma subtypes

21 January 2018

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Identification of decatenation G2 checkpoint impairment independently of DNA damage G2 checkpoint in human lung cancer cell lines.

21 January 2018

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Rb inactivation leads to E2F1-mediated DNA double-strand break accumulation

21 January 2018

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E2F1 death pathways as targets for cancer therapy

21 January 2018

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Molecular genetics of lung cancer

21 January 2018

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E2F1 and c-Myc potentiate apoptosis through inhibition of NF-kappaB activity that facilitates MnSOD-mediated ROS elimination

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Retinoblastoma pathway defects show differential ability to activate the constitutive DNA damage response in human tumorigenesis

21 January 2018

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Complex engagement of DNA damage response pathways in human cancer and in lung tumor progression

21 January 2018

1 reference

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31 March 2020

Identifiers

DOI

10.1038/ONC.2009.201

2 references

Consolidated OpenCitations Corpus – April 2017

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:19597473%20AND%20SRC:MED&resulttype=core&format=json

Dimensions Publication ID

2 February 2020

1037331347

0 references

1 reference

Consolidated OpenCitations Corpus – April 2017

2 references

Consolidated OpenCitations Corpus – April 2017