Pages that link to "Q28586023"
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The following pages link to Dachshund homologues play a conserved role in islet cell development (Q28586023):
Displaying 15 items.
- Dachshund family transcription factor 1 (Q21981328) (← links)
- The retinal determination gene network: from developmental regulator to cancer therapeutic target (Q26747218) (← links)
- Concise review: in search of unlimited sources of functional human pancreatic beta cells (Q26999928) (← links)
- Analysis of beta-cell gene expression reveals inflammatory signaling and evidence of dedifferentiation following human islet isolation and culture (Q31047685) (← links)
- Specific control of pancreatic endocrine β- and δ-cell mass by class IIa histone deacetylases HDAC4, HDAC5, and HDAC9 (Q35405305) (← links)
- Conserved role for the Dachshund protein with Drosophila Pax6 homolog Eyeless in insulin expression (Q35787038) (← links)
- Age-Dependent Pancreatic Gene Regulation Reveals Mechanisms Governing Human β Cell Function (Q36892940) (← links)
- Familial young-onset diabetes, pre-diabetes and cardiovascular disease are associated with genetic variants of DACH1 in Chinese (Q37495697) (← links)
- Decreased DACH1 expression in glomerulopathy is associated with disease progression and severity (Q37699281) (← links)
- DACH1 stimulates shear stress-guided endothelial cell migration and coronary artery growth through the CXCL12-CXCR4 signaling axis (Q38639773) (← links)
- Hepatocyte DACH1 Is Increased in Obesity via Nuclear Exclusion of HDAC4 and Promotes Hepatic Insulin Resistance (Q41493159) (← links)
- The transcription factor Dach1 is essential for podocyte function. (Q51736678) (← links)
- The regulation of cytokine signaling by retinal determination gene network pathway in cancer (Q57483942) (← links)
- Identification of the neurotransmitter profile of AmFoxP expressing neurons in the honeybee brain using double-label in situ hybridization (Q58605605) (← links)
- Genetic deletion of miR-204 improves glycemic control despite obesity in db/db mice (Q99574874) (← links)