Pages that link to "Q37566211"
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The following pages link to Mimicking the BH3 domain to kill cancer cells (Q37566211):
Displaying 50 items.
- Small-Molecule Bcl2 BH4 Antagonist for Lung Cancer Therapy (Q24337757) (← links)
- Novel Targeted Agents in Hodgkin and Non-Hodgkin Lymphoma Therapy (Q26782270) (← links)
- Targeted Therapies in Adult B-Cell Malignancies (Q26783195) (← links)
- The role of BH3-mimetic drugs in the treatment of pediatric hepatoblastoma (Q26827928) (← links)
- Targeting the Bcl-2 family for cancer therapy (Q26866936) (← links)
- Ion channels and apoptosis in cancer (Q27005947) (← links)
- Targeting Mcl-1 for the therapy of cancer (Q27011870) (← links)
- Mitochondrial apoptosis and BH3 mimetics (Q28069552) (← links)
- Metabolic reprogramming in cancer cells: glycolysis, glutaminolysis, and Bcl-2 proteins as novel therapeutic targets for cancer (Q28072669) (← links)
- Alternative Treatments For Melanoma: Targeting BCL-2 Family Members to De-Bulk and Kill Cancer Stem Cells (Q28084243) (← links)
- Novel non-AR therapeutic targets in castrate resistant prostate cancer (Q28383663) (← links)
- Alterations in glutathione levels and apoptotic regulators are associated with acquisition of arsenic trioxide resistance in multiple myeloma (Q28484542) (← links)
- Ultra-High-Throughput Screening of Natural Product Extracts to Identify Proapoptotic Inhibitors of Bcl-2 Family Proteins. (Q30397851) (← links)
- HA14-1, but not the BH3 mimetic ABT-737, causes Ca2+ dysregulation in platelets and human cell lines. (Q30454476) (← links)
- MET-independent lung cancer cells evading EGFR kinase inhibitors are therapeutically susceptible to BH3 mimetic agents (Q30502341) (← links)
- Apoptotic regulation of epithelial cellular extrusion (Q30638314) (← links)
- Expanding the range of 'druggable' targets with natural product-based libraries: an academic perspective (Q33536938) (← links)
- Sorafenib and HDAC inhibitors synergize with TRAIL to kill tumor cells (Q33576187) (← links)
- Ionizing radiation sensitizes breast cancer cells to Bcl-2 inhibitor, ABT-737, through regulating Mcl-1 (Q33924886) (← links)
- PINT: Pathways INtegration Tool. (Q33957511) (← links)
- The BH3 mimetic ABT-737 increases treatment efficiency of paclitaxel against hepatoblastoma (Q33997669) (← links)
- Maturation stage of T-cell acute lymphoblastic leukemia determines BCL-2 versus BCL-XL dependence and sensitivity to ABT-199 (Q34135298) (← links)
- Expression of Bim, Noxa, and Puma in non-small cell lung cancer (Q34334492) (← links)
- Rapid detection of an ABT-737-sensitive primed for death state in cells using microplate-based respirometry (Q34374169) (← links)
- Anti-tumor activity of splice-switching oligonucleotides (Q34401490) (← links)
- Molecular interactions of prodiginines with the BH3 domain of anti-apoptotic Bcl-2 family members (Q34608671) (← links)
- Novel small-molecule inhibitors of Bcl-XL to treat lung cancer. (Q34793542) (← links)
- A urokinase receptor-Bim signaling axis emerges during EGFR inhibitor resistance in mutant EGFR glioblastoma (Q34979703) (← links)
- SNF5 reexpression in malignant rhabdoid tumors regulates transcription of target genes by recruitment of SWI/SNF complexes and RNAPII to the transcription start site of their promoters (Q35125250) (← links)
- Distribution of Bim determines Mcl-1 dependence or codependence with Bcl-xL/Bcl-2 in Mcl-1-expressing myeloma cells (Q35153790) (← links)
- Limited mitochondrial permeabilization causes DNA damage and genomic instability in the absence of cell death (Q35156829) (← links)
- Regulation of the DNA damage response by ubiquitin conjugation (Q35161624) (← links)
- Hox and a newly identified E2F co-repress cell death in Caenorhabditis elegans (Q35221460) (← links)
- An integrated approach to dissecting oncogene addiction implicates a Myb-coordinated self-renewal program as essential for leukemia maintenance (Q35237394) (← links)
- Isocitrate dehydrogenase 1 and 2 mutations induce BCL-2 dependence in acute myeloid leukemia (Q35524250) (← links)
- BH3 Profiling Reveals Selectivity by Herpesviruses for Specific Bcl-2 Proteins To Mediate Survival of Latently Infected Cells. (Q35641404) (← links)
- Use of multifunctional sigma-2 receptor ligand conjugates to trigger cancer-selective cell death signaling (Q35652832) (← links)
- Pancreatic Cancer Combination Therapy Using a BH3 Mimetic and a Synthetic Tetracycline. (Q35676278) (← links)
- Selective peptide inhibitors of antiapoptotic cellular and viral Bcl-2 proteins lead to cytochrome c release during latent Kaposi's sarcoma-associated herpesvirus infection. (Q35802945) (← links)
- Anoikis evasion in inflammatory breast cancer cells is mediated by Bim-EL sequestration (Q35829855) (← links)
- MYC pathway activation in triple-negative breast cancer is synthetic lethal with CDK inhibition (Q35894198) (← links)
- The tipping point for combination therapy: cancer vaccines with radiation, chemotherapy, or targeted small molecule inhibitors (Q35975387) (← links)
- AT-101 enhances gefitinib sensitivity in non-small cell lung cancer with EGFR T790M mutations (Q36081033) (← links)
- Role of intratumoural heterogeneity in cancer drug resistance: molecular and clinical perspectives (Q36386428) (← links)
- Structure-based discovery of BM-957 as a potent small-molecule inhibitor of Bcl-2 and Bcl-xL capable of achieving complete tumor regression (Q36390326) (← links)
- Lapatinib and obatoclax kill breast cancer cells through reactive oxygen species-dependent endoplasmic reticulum stress (Q36412836) (← links)
- Co-targeting of Bcl-2 and mTOR pathway triggers synergistic apoptosis in BH3 mimetics resistant acute lymphoblastic leukemia (Q36544778) (← links)
- Apoptosis Activation in Human Lung Cancer Cell Lines by a Novel Synthetic Peptide Derived from Conus californicus Venom (Q36638317) (← links)
- Nanotechnology inspired tools for mitochondrial dysfunction related diseases (Q36703351) (← links)
- MEK inhibition enhances ABT-737-induced leukemia cell apoptosis via prevention of ERK-activated MCL-1 induction and modulation of MCL-1/BIM complex (Q36706340) (← links)