Pages that link to "Q43002476"
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The following pages link to Bax oligomerization in mitochondrial membranes requires tBid (caspase-8-cleaved Bid) and a mitochondrial protein. (Q43002476):
Displaying 50 items.
- Use of human cancer cell lines mitochondria to explore the mechanisms of BH3 peptides and ABT-737-induced mitochondrial membrane permeabilization (Q21562635) (← links)
- Regulation of the mitochondrial apoptosis-induced channel, MAC, by BCL-2 family proteins (Q21564051) (← links)
- Interaction with a membrane surface triggers a reversible conformational change in Bax normally associated with induction of apoptosis (Q24296781) (← links)
- Membrane remodeling induced by the dynamin-related protein Drp1 stimulates Bax oligomerization (Q24300125) (← links)
- Proapoptotic Bak is sequestered by Mcl-1 and Bcl-xL, but not Bcl-2, until displaced by BH3-only proteins (Q24302494) (← links)
- MAP-1 is a mitochondrial effector of Bax. (Q24337383) (← links)
- Mitochondrial carrier homolog 2 is a target of tBID in cells signaled to die by tumor necrosis factor alpha (Q24529078) (← links)
- Loss of Bif-1 suppresses Bax/Bak conformational change and mitochondrial apoptosis (Q24534937) (← links)
- Altered Traffic of Cardiolipin during Apoptosis: Exposure on the Cell Surface as a Trigger for "Antiphospholipid Antibodies" (Q26780481) (← links)
- Guards and culprits in the endoplasmic reticulum: glucolipotoxicity and β-cell failure in type II diabetes (Q26992217) (← links)
- The structure of a Bcl-xL/Bim fragment complex: implications for Bim function (Q27642185) (← links)
- Intermembrane space proteome of yeast mitochondria (Q27935768) (← links)
- Activation of the caspase-8/Bid and Bax pathways in aspirin-induced apoptosis in gastric cancer (Q28211131) (← links)
- Bax activation initiates the assembly of a multimeric catalyst that facilitates Bax pore formation in mitochondrial outer membranes (Q28483986) (← links)
- Spatial and temporal changes in Bax subcellular localization during anoikis (Q28507532) (← links)
- Aberrant Apoptotic Response of Colorectal Cancer Cells to Novel Nucleoside Analogues (Q28548303) (← links)
- Mcl-1 downregulation by pro-inflammatory cytokines and palmitate is an early event contributing to β-cell apoptosis (Q28571868) (← links)
- MTCH2/MIMP is a major facilitator of tBID recruitment to mitochondria (Q28594262) (← links)
- Active fragments from pro- and antiapoptotic BCL-2 proteins have distinct membrane behavior reflecting their functional divergence (Q28749082) (← links)
- Mitochondrial apoptosis is amplified through gap junctions (Q30491150) (← links)
- Involvement of VDAC, Bax and ceramides in the efflux of AIF from mitochondria during curcumin-induced apoptosis (Q33495179) (← links)
- Elucidating cytochrome C release from mitochondria: insights from an in silico three-dimensional model (Q34306872) (← links)
- Bax channel inhibitors prevent mitochondrion-mediated apoptosis and protect neurons in a model of global brain ischemia. (Q34458933) (← links)
- Is MAC the knife that cuts cytochrome c from mitochondria during apoptosis? (Q34522154) (← links)
- TOM22, a core component of the mitochondria outer membrane protein translocation pore, is a mitochondrial receptor for the proapoptotic protein Bax. (Q34580706) (← links)
- The effects of erdosteine and N-acetylcysteine on apoptotic and antiapoptotic markers in pulmonary epithelial cells in sepsis (Q34666878) (← links)
- Mitochondria: releasing power for life and unleashing the machineries of death (Q35071387) (← links)
- A retention factor keeps death at bay. (Q35096245) (← links)
- Mitochondrial membrane permeabilisation by Bax/Bak (Q35120714) (← links)
- Utilizing mitochondrial events as biomarkers for imaging apoptosis (Q35202281) (← links)
- Bax modulates neuronal survival while p53 is unaltered after Cytochrome C induced oxidative stress in the adult olfactory bulb in vivo (Q35540814) (← links)
- Identification of Bax-voltage-dependent anion channel 1 complexes in digitonin-solubilized cerebellar granule neurons (Q35552682) (← links)
- The Bcl-2 family: roles in cell survival and oncogenesis. (Q35591695) (← links)
- Molecular mechanisms behind progressing chronic inflammatory dilated cardiomyopathy (Q35608214) (← links)
- Role of mitochondrial membrane permeabilization in apoptosis and cancer (Q35743123) (← links)
- Unlocking Pandora's box: personalising cancer cell death in non-small cell lung cancer (Q36173303) (← links)
- Minute kinetics of proapoptotic proteins: BAX and Smac/DIABLO in living tumor cells revealed by homeostatic confocal microscopy (Q36256139) (← links)
- How the Bcl-2 family of proteins interact to regulate apoptosis. (Q36394994) (← links)
- BCL2 family of apoptosis-related genes: functions and clinical implications in cancer (Q36419013) (← links)
- RRR-gamma-tocopherol induces human breast cancer cells to undergo apoptosis via death receptor 5 (DR5)-mediated apoptotic signaling (Q36489164) (← links)
- The Bax pore in liposomes, Biophysics (Q36501189) (← links)
- Role of cardiolipin in cytochrome c release from mitochondria (Q36790564) (← links)
- Characterization of MUDENG, a novel anti-apoptotic protein (Q37096238) (← links)
- Mitochondrial outer membrane proteins assist Bid in Bax-mediated lipidic pore formation (Q37158414) (← links)
- Assembly of the mitochondrial apoptosis-induced channel, MAC. (Q37169502) (← links)
- Molecular participants in mitochondrial cell death channel formation during neuronal ischemia (Q37430520) (← links)
- Bax transmembrane domain interacts with prosurvival Bcl-2 proteins in biological membranes (Q37589942) (← links)
- MAC and Bcl-2 family proteins conspire in a deadly plot (Q37677213) (← links)
- Permeabilization of the Outer Mitochondrial Membrane by Bcl-2 Proteins (Q37778022) (← links)
- Apoptosis-induced changes in mitochondrial lipids (Q37796181) (← links)