Pages that link to "Q57331400"
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The following pages link to Mice Deficient in Complement Receptors 1 and 2 Lack a Tissue Injury-Inducing Subset of the Natural Antibody Repertoire (Q57331400):
Displaying 50 items.
- Pathogenic natural antibodies recognizing annexin IV are required to develop intestinal ischemia-reperfusion injury (Q33646877) (← links)
- Intestinal lipid alterations occur prior to antibody-induced prostaglandin E2 production in a mouse model of ischemia/reperfusion (Q33678586) (← links)
- B cells limit repair after ischemic acute kidney injury (Q33748553) (← links)
- Membrane lipid interactions in intestinal ischemia/reperfusion-induced Injury (Q33902557) (← links)
- Inhibition of rat gut reperfusion injury with an agent developed for the mouse. Evidence that amplification of injury by innate immunity is conserved between two animal species (Q33917428) (← links)
- Targeted complement inhibition by C3d recognition ameliorates tissue injury without apparent increase in susceptibility to infection (Q33925353) (← links)
- Complement and its role in innate and adaptive immune responses (Q34088142) (← links)
- Platelet-associated CD40/CD154 mediates remote tissue damage after mesenteric ischemia/reperfusion injury (Q34182418) (← links)
- Role of the complement system in rejection. (Q34263683) (← links)
- Immunogenicity to biologics: mechanisms, prediction and reduction (Q34296526) (← links)
- Altered renal tubular expression of the complement inhibitor Crry permits complement activation after ischemia/reperfusion. (Q34315819) (← links)
- Attenuation of the effects of rat hemorrhagic shock with a reperfusion injury-inhibiting agent specific to mice (Q34357587) (← links)
- Domain V Peptides Inhibit β2-Glycoprotein I-Mediated Mesenteric Ischemia/Reperfusion-Induced Tissue Damage and Inflammation (Q34401715) (← links)
- Helicobacter infection alters MyD88 and Trif signalling in response to intestinal ischaemia-reperfusion (Q34504559) (← links)
- CR2+ marginal zone B cell production of pathogenic natural antibodies is C3 independent (Q34507790) (← links)
- Sca-1 influences the innate immune response during skeletal muscle regeneration (Q34597576) (← links)
- Oxidative stress sensitizes retinal pigmented epithelial (RPE) cells to complement-mediated injury in a natural antibody-, lectin pathway-, and phospholipid epitope-dependent manner (Q34620560) (← links)
- Synergistic induction of interferon α through TLR-3 and TLR-9 agonists identifies CD21 as interferon α receptor for the B cell response (Q34629175) (← links)
- TLR2 modulates antibodies required for intestinal ischemia/reperfusion-induced damage and inflammation (Q34980207) (← links)
- A new mouse anti-mouse complement receptor type 2 and 1 (CR2/CR1) monoclonal antibody as a tool to study receptor involvement in chronic models of immune responses and disease (Q35002158) (← links)
- B cell subsets contribute to renal injury and renal protection after ischemia/reperfusion (Q35097477) (← links)
- The complement system as a therapeutic target in autoimmunity (Q35153653) (← links)
- Complement and autoimmunity (Q35538114) (← links)
- Ischaemia-reperfusion is an event triggered by immune complexes and complement (Q35597392) (← links)
- B-cell extrinsic CR1/CR2 promotes natural antibody production and tolerance induction of anti-alphaGAL-producing B-1 cells (Q35628964) (← links)
- Innate response to self-antigen significantly exacerbates burn wound depth (Q35676741) (← links)
- Pathogenic natural antibodies propagate cerebral injury following ischemic stroke in mice (Q35685695) (← links)
- Challenging the role of adaptive immunity in neurotrauma: Rag1(-/-) mice lacking mature B and T cells do not show neuroprotection after closed head injury. (Q35886721) (← links)
- The complement system in regulation of adaptive immunity (Q35904390) (← links)
- The Fifth Domain of Beta 2 Glycoprotein I Protects from Natural IgM Mediated Cardiac Ischaemia Reperfusion Injury (Q35974974) (← links)
- Complement receptors and the shaping of the natural antibody repertoire (Q35991469) (← links)
- IgM exacerbates glomerular disease progression in complement-induced glomerulopathy (Q36016859) (← links)
- Innate autoimmunity (Q36037749) (← links)
- Identification of the target self-antigens in reperfusion injury (Q36227933) (← links)
- Small β2-glycoprotein I peptides protect from intestinal ischemia reperfusion injury (Q36374030) (← links)
- TLR9 is dispensable for intestinal ischemia/reperfusion-induced tissue damage (Q36431855) (← links)
- The role of complement in danger sensing and transmission (Q36499763) (← links)
- Natural antibodies, autoantibodies and complement activation in tissue injury (Q36572982) (← links)
- Triggers of inflammation after renal ischemia/reperfusion (Q36634596) (← links)
- IgM contributes to glomerular injury in FSGS (Q36638347) (← links)
- The role of complement and Toll-like receptors in organ transplantation (Q36817201) (← links)
- Natural IgM-mediated innate autoimmunity: a new target for early intervention of ischemia-reperfusion injury (Q36960513) (← links)
- The classical complement pathway in transplantation: unanticipated protective effects of C1q and role in inductive antibody therapy (Q36984472) (← links)
- Identification of a specific self-reactive IgM antibody that initiates intestinal ischemia/reperfusion injury (Q37095150) (← links)
- The innate immune response in ischemic acute kidney injury (Q37105485) (← links)
- Molecular structure and expression of anthropic, ovine, and murine forms of complement receptor type 2. (Q37133741) (← links)
- The role of the complement system in acute kidney injury (Q37275159) (← links)
- Lymphocytes and ischemia-reperfusion injury (Q37332688) (← links)
- The role of complement in the antiphospholipid syndrome-associated pathology (Q37344788) (← links)
- TLR4-mediated Cox-2 expression increases intestinal ischemia/reperfusion-induced damage (Q37362932) (← links)