Pages that link to "Q73304613"
Jump to navigation
Jump to search
The following pages link to Advanced glycation end products activate Smad signaling via TGF-beta-dependent and independent mechanisms: implications for diabetic renal and vascular disease (Q73304613):
Displaying 50 items.
- Mechanisms, Pathophysiology, and Therapy of Arterial Stiffness (Q22306395) (← links)
- The Role of AGE/RAGE Signaling in Diabetes-Mediated Vascular Calcification (Q26740003) (← links)
- Vascular Fibrosis in Aging and Hypertension: Molecular Mechanisms and Clinical Implications (Q26749488) (← links)
- TGF-β/Smad signaling in renal fibrosis (Q26851198) (← links)
- Smads as therapeutic targets for chronic kidney disease (Q26865998) (← links)
- Atherosclerosis and restenosis: is there a role for RAGE? (Q28303607) (← links)
- A cationic-independent mannose 6-phosphate receptor inhibitor (PXS64) ameliorates kidney fibrosis by inhibiting activation of transforming growth factor-β1 (Q28543256) (← links)
- A thrombospondin-1 antagonist of transforming growth factor-beta activation blocks cardiomyopathy in rats with diabetes and elevated angiotensin II (Q28583200) (← links)
- Advanced glycation end products induce tubular epithelial-myofibroblast transition through the RAGE-ERK1/2 MAP kinase signaling pathway (Q30445160) (← links)
- Ultrasound-microbubble-mediated gene transfer of inducible Smad7 blocks transforming growth factor-beta signaling and fibrosis in rat remnant kidney. (Q30499402) (← links)
- RAGE signaling in inflammation and arterial aging (Q33346824) (← links)
- S100A4 and bone morphogenetic protein-2 codependently induce vascular smooth muscle cell migration via phospho-extracellular signal-regulated kinase and chloride intracellular channel 4 (Q33636296) (← links)
- Advanced glycation end-products induce tubular CTGF via TGF-beta-independent Smad3 signaling (Q33711306) (← links)
- Kidney dysfunction in patients with pulmonary arterial hypertension (Q33738621) (← links)
- Clusterin/apolipoprotein J attenuates angiotensin II-induced renal fibrosis (Q34078756) (← links)
- Resveratrol inhibits renal fibrosis in the obstructed kidney: potential role in deacetylation of Smad3 (Q34086829) (← links)
- miR-192 mediates TGF-beta/Smad3-driven renal fibrosis (Q34124178) (← links)
- Vasohibin-1 deficiency enhances renal fibrosis and inflammation after unilateral ureteral obstruction (Q34399941) (← links)
- Advanced glycation end products activate a chymase-dependent angiotensin II-generating pathway in diabetic complications (Q34448452) (← links)
- The protective role of Smad7 in diabetic kidney disease: mechanism and therapeutic potential (Q34527184) (← links)
- Essential role of Smad3 in angiotensin II-induced vascular fibrosis (Q34575283) (← links)
- Angiotensin-(1-7) abolishes AGE-induced cellular hypertrophy and myofibroblast transformation via inhibition of ERK1/2. (Q34623871) (← links)
- Proinflammatory protein CARD9 is essential for infiltration of monocytic fibroblast precursors and cardiac fibrosis caused by Angiotensin II infusion (Q35114753) (← links)
- Chaihuang-Yishen granule inhibits diabetic kidney disease in rats through blocking TGF-β/Smad3 signaling. (Q35124896) (← links)
- Role of Cell Division Autoantigen 1 (CDA1) in Cell Proliferation and Fibrosis (Q35141843) (← links)
- Inhibition of p38 mitogen-activated protein kinase and transforming growth factor-beta1/Smad signaling pathways modulates the development of fibrosis in adriamycin-induced nephropathy. (Q35607297) (← links)
- AMP-activated protein kinase (AMPK) activation inhibits nuclear translocation of Smad4 in mesangial cells and diabetic kidneys (Q35622481) (← links)
- Blockade of endothelial-mesenchymal transition by a Smad3 inhibitor delays the early development of streptozotocin-induced diabetic nephropathy (Q35757154) (← links)
- Therapeutic Effects of Tangshen Formula on Diabetic Nephropathy in Rats (Q35903165) (← links)
- Increased urinary Smad3 is significantly correlated with glomerular hyperfiltration and a reduced glomerular filtration rate and is a new urinary biomarker for diabetic nephropathy (Q36142232) (← links)
- Circulating TGF-β1, glycation, and oxidation in children with diabetes mellitus type 1. (Q36294234) (← links)
- Mechanism Investigation of the Improvement of Chang Run Tong on the Colonic Remodeling in Streptozotocin-Induced Diabetic Rats (Q36454380) (← links)
- Smad7 suppresses renal fibrosis via altering expression of TGF-β/Smad3-regulated microRNAs (Q36673667) (← links)
- C-Reactive Protein Promotes Diabetic Kidney Disease in db/db Mice via the CD32b-Smad3-mTOR signaling Pathway. (Q36934223) (← links)
- Blockade of KCa3.1 ameliorates renal fibrosis through the TGF-β1/Smad pathway in diabetic mice (Q37030987) (← links)
- Specific local cardiovascular changes of Nepsilon-(carboxymethyl)lysine, vascular endothelial growth factor, and Smad2 in the developing embryos coincide with maternal diabetes-induced congenital heart defects (Q37163557) (← links)
- Genetic deletion of cell division autoantigen 1 retards diabetes-associated renal injury (Q37260211) (← links)
- Blockade of advanced glycation end product formation attenuates bleomycin-induced pulmonary fibrosis in rats (Q37266352) (← links)
- Is transforming growth factor-β signaling activated in human hypertrophied prostate treated by 5-alpha reductase inhibitor? (Q37338238) (← links)
- Podocytes: the Weakest Link in Diabetic Kidney Disease? (Q37340690) (← links)
- Endothelial-myofibroblast transition contributes to the early development of diabetic renal interstitial fibrosis in streptozotocin-induced diabetic mice. (Q37362038) (← links)
- Proteinase-activated receptor-2 transactivation of epidermal growth factor receptor and transforming growth factor-β receptor signaling pathways contributes to renal fibrosis. (Q37415689) (← links)
- Advanced glycation end products and C-peptide-modulators in diabetic vasculopathy and atherogenesis (Q37433970) (← links)
- Aldosterone and myocardial extracellular matrix expansion in type 2 diabetes mellitus (Q37643007) (← links)
- Diverse roles of TGF-β/Smads in renal fibrosis and inflammation (Q37934555) (← links)
- Chromatin modifications associated with diabetes (Q38013734) (← links)
- Molecular targets for treatment of kidney fibrosis (Q38062404) (← links)
- Molecular mechanisms in the pathogenesis of diabetic nephropathy: an update (Q38073540) (← links)
- Role of the endothelial-to-mesenchymal transition in renal fibrosis of chronic kidney disease (Q38083596) (← links)
- Pathophysiology of the diabetic kidney. (Q38111707) (← links)