Pages that link to "Q28609805"
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The following pages link to Dual function for poly(ADP-ribose) synthesis in response to DNA strand breakage (Q28609805):
Displaying 50 items.
- A cellular defense pathway regulating transcription through poly(ADP-ribosyl)ation in response to DNA damage (Q24289566) (← links)
- XRCC1 is specifically associated with poly(ADP-ribose) polymerase and negatively regulates its activity following DNA damage (Q24522502) (← links)
- Second pathway for completion of human DNA base excision-repair: reconstitution with purified proteins and requirement for DNase IV (FEN1). (Q24532243) (← links)
- TFIIH-mediated nucleotide excision repair and initiation of mRNA transcription in an optimized cell-free DNA repair and RNA transcription assay (Q24548165) (← links)
- Bench-to-bedside review: Cytopathic hypoxia (Q24807180) (← links)
- DNA excision repair and DNA damage-induced apoptosis are linked to Poly(ADP-ribosyl)ation but have different requirements for p53 (Q28115289) (← links)
- Mice lacking ADPRT and poly(ADP-ribosyl)ation develop normally but are susceptible to skin disease (Q28298870) (← links)
- Depletion of the 110-kilodalton isoform of poly(ADP-ribose) glycohydrolase increases sensitivity to genotoxic and endotoxic stress in mice (Q28508144) (← links)
- Niacin deficiency alters p53 expression and impairs etoposide-induced cell cycle arrest and apoptosis in rat bone marrow cells (Q28566331) (← links)
- PolyADP-ribosylation is involved in neurotrophic activity (Q28567748) (← links)
- PARP inhibitor treatment in ovarian and breast cancer (Q28741688) (← links)
- Poly(ADP-ribosyl)ation reactions in the regulation of nuclear functions (Q29617672) (← links)
- PARP inhibition: PARP1 and beyond (Q29619617) (← links)
- Solution structures of a duplex containing an adenine opposite a gap (absence of one nucleotide). An NMR study and molecular dynamic simulations with explicit water molecules (Q30580635) (← links)
- Cytopathic hypoxia. Mitochondrial dysfunction as mechanism contributing to organ dysfunction in sepsis (Q32060202) (← links)
- Competent transcription initiation by RNA polymerase II in cell-free extracts from xeroderma pigmentosum groups B and D in an optimized RNA transcription assay (Q32148632) (← links)
- Involvement of poly(ADP-ribose) polymerase in base excision repair (Q33601042) (← links)
- Mammalian X-ray-sensitive mutants which are defective in non-homologous (illegitimate) DNA double-strand break repair (Q33601058) (← links)
- Functional Aspects of PARP1 in DNA Repair and Transcription (Q33649584) (← links)
- XRCC1 keeps DNA from getting stranded (Q33839295) (← links)
- Functions of poly(ADP-ribose) polymerase (PARP) in DNA repair, genomic integrity and cell death (Q33948501) (← links)
- Alkylation DNA damage in combination with PARP inhibition results in formation of S-phase-dependent double-strand breaks. (Q34037211) (← links)
- Role of nicotinamide in DNA damage, mutagenesis, and DNA repair (Q34042309) (← links)
- The complexes of mammalian target of rapamycin. (Q34116818) (← links)
- Niacin, poly(ADP-ribose) polymerase-1 and genomic stability (Q34213961) (← links)
- Cytopathic hypoxia. Is oxygen use impaired in sepsis as a result of an acquired intrinsic derangement in cellular respiration? (Q34575280) (← links)
- DNA repair defect in poly(ADP-ribose) polymerase-deficient cell lines (Q34668455) (← links)
- A role for poly(ADP-ribosyl)ation in DNA methylation (Q35191562) (← links)
- Is poly(ADP-ribose) polymerase involved in bovine placental retention? (Q35214805) (← links)
- XRCC1 and DNA strand break repair (Q35216366) (← links)
- Pharmacological Modulation of Poly(ADP-ribose) Polymerase-Mediated Cell Death: Exploitation in Cancer Chemotherapy (Q35565981) (← links)
- Efficient retroviral infection of mammalian cells is blocked by inhibition of poly(ADP-ribose) polymerase activity (Q35861994) (← links)
- MeCP2 binds to nucleosome free (linker DNA) regions and to H3K9/H3K27 methylated nucleosomes in the brain (Q35888788) (← links)
- Genetic and epigenetic features in radiation sensitivity Part I: cell signalling in radiation response (Q36013284) (← links)
- Regulation of chromatin structure by poly(ADP-ribosyl)ation (Q36204365) (← links)
- Absence of stimulation of poly(ADP-ribose) polymerase activity in patients predisposed to colon cancer (Q36292102) (← links)
- PARP inhibition during alkylation-induced genotoxic stress signals a cell cycle checkpoint response mediated by ATM. (Q37392187) (← links)
- Poly(ADP-ribose) polymerases covalently modify strand break termini in DNA fragments in vitro. (Q37401119) (← links)
- Structural Implications for Selective Targeting of PARPs (Q37405386) (← links)
- Manipulation of indoleamine 2,3 dioxygenase; a novel therapeutic target for treatment of diseases (Q37522460) (← links)
- Poly(adenosine diphosphate-ribose) polymerase as therapeutic target: lessons learned from its inhibitors (Q38692308) (← links)
- Beet root juice protects against doxorubicin toxicity in cardiomyocytes while enhancing apoptosis in breast cancer cells (Q38749854) (← links)
- Dorzolamide synergizes the antitumor activity of mitomycin C against Ehrlich's carcinoma grown in mice: role of thioredoxin-interacting protein. (Q38844369) (← links)
- Reduced ADP-ribosylation by PARP1 natural polymorphism V762A and by PARP1 inhibitors enhance Hepatitis B virus replication (Q39116332) (← links)
- Shiga toxin 1, as DNA repair inhibitor, synergistically potentiates the activity of the anticancer drug, mafosfamide, on raji cells (Q39190558) (← links)
- Base excision repair is efficient in cells lacking poly(ADP-ribose) polymerase 1. (Q39584636) (← links)
- DNA damage and repair in tumour and non-tumour tissues of mice induced by nicotinamide (Q39997202) (← links)
- Poly(ADP-ribose) polymerase 1 accelerates single-strand break repair in concert with poly(ADP-ribose) glycohydrolase (Q40124096) (← links)
- Biochemical properties and function of poly(ADP-ribose) glycohydrolase (Q40424492) (← links)
- Enzymes acting at strand interruptions in DNA. (Q40522912) (← links)