Pages that link to "Q37998609"
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The following pages link to Genetic architecture in autism spectrum disorder (Q37998609):
Displaying 50 items.
- Lamarckian evolution explains human brain evolution and psychiatric disorders (Q21129357) (← links)
- Hyperbaric oxygen therapy for autism spectrum disorder (ASD) in children and adults (Q24201426) (← links)
- SHANK1 Deletions in Males with Autism Spectrum Disorder (Q24313471) (← links)
- A SWI/SNF-related autism syndrome caused by de novo mutations in ADNP (Q24567998) (← links)
- A novel computational biostatistics approach implies impaired dephosphorylation of growth factor receptors as associated with severity of autism (Q24612710) (← links)
- Whole Genome Sequencing as a Genetic Test for Autism Spectrum Disorder: From Bench to Bedside and then Back Again (Q24658630) (← links)
- Analysis of conditional heterozygous STXBP1 mutations in human neurons (Q26269829) (← links)
- Recent Advances in Autism Spectrum Disorders: Applications of Whole Exome Sequencing Technology (Q26744368) (← links)
- Modeling psychiatric disorders for developing effective treatments (Q26784269) (← links)
- Understanding the molecular basis of autism in a dish using hiPSCs-derived neurons from ASD patients (Q26784399) (← links)
- Channelopathy pathogenesis in autism spectrum disorders (Q26864726) (← links)
- Is autism curable? (Q27015272) (← links)
- Therapeutic approaches for shankopathies (Q27024976) (← links)
- Defective control of pre-messenger RNA splicing in human disease (Q28077564) (← links)
- A familial heterozygous null mutation of MET in autism spectrum disorder (Q28241623) (← links)
- DUF1220 copy number is associated with schizophrenia risk and severity: implications for understanding autism and schizophrenia as related diseases (Q28271073) (← links)
- Major channels involved in neuropsychiatric disorders and therapeutic perspectives (Q28290639) (← links)
- Letting a typical mouse judge whether mouse social interactions are atypical (Q28383456) (← links)
- Mosaic epigenetic dysregulation of ectodermal cells in autism spectrum disorder (Q28386912) (← links)
- Translational Mouse Models of Autism: Advancing Toward Pharmacological Therapeutics (Q28394524) (← links)
- Using Gene Ontology to describe the role of the neurexin-neuroligin-SHANK complex in human, mouse and rat and its relevance to autism (Q28645671) (← links)
- Protein interaction networks reveal novel autism risk genes within GWAS statistical noise (Q28651845) (← links)
- Individual common variants exert weak effects on the risk for autism spectrum disorders (Q28943296) (← links)
- The Changing Epidemiology of Autism Spectrum Disorders (Q29227439) (← links)
- Chromosomal microarray analysis of consecutive individuals with autism spectrum disorders or learning disability presenting for genetic services (Q30009531) (← links)
- An Overview of Autism Spectrum Disorder, Heterogeneity and Treatment Options. (Q30235878) (← links)
- Common Polymorphisms in the Age of Research Domain Criteria (RDoC): Integration and Translation. (Q30366614) (← links)
- Targeting Glia with N-Acetylcysteine Modulates Brain Glutamate and Behaviors Relevant to Neurodevelopmental Disorders in C57BL/6J Mice. (Q30395471) (← links)
- Using whole-exome sequencing to identify inherited causes of autism (Q30418126) (← links)
- Towards identification of individual etiologies by resolving genomic and biological conundrums in patients with autism spectrum disorders (Q30451842) (← links)
- Identification of rare X-linked neuroligin variants by massively parallel sequencing in males with autism spectrum disorder (Q30461208) (← links)
- Common genetic variants, acting additively, are a major source of risk for autism (Q30536405) (← links)
- The autism sequencing consortium: large-scale, high-throughput sequencing in autism spectrum disorders (Q30559475) (← links)
- Phenotypic and functional analysis of SHANK3 stop mutations identified in individuals with ASD and/or ID. (Q30653240) (← links)
- Uncovering the etiology of autism spectrum disorders: genomics, bioinformatics, environment, data collection and exploration, and future possibilities (Q30706007) (← links)
- Oxytocin receptor gene variations predict neural and behavioral response to oxytocin in autism. (Q30845236) (← links)
- DAWN: a framework to identify autism genes and subnetworks using gene expression and genetics (Q33592044) (← links)
- Serum glycopattern and Maackia amurensis lectin-II binding glycoproteins in autism spectrum disorder (Q33651921) (← links)
- De novo mutation in the dopamine transporter gene associates dopamine dysfunction with autism spectrum disorder (Q33715513) (← links)
- Association of rare missense variants in the second intracellular loop of NaV1.7 sodium channels with familial autism (Q33790438) (← links)
- An inside job: how endosomal Na(+)/H(+) exchangers link to autism and neurological disease (Q33791880) (← links)
- Convergence of genes and cellular pathways dysregulated in autism spectrum disorders (Q33794546) (← links)
- Lithium ameliorates autistic-like behaviors induced by neonatal isolation in rats. (Q33810377) (← links)
- Towards a molecular characterization of autism spectrum disorders: an exome sequencing and systems approach. (Q33840498) (← links)
- A randomized controlled study of a social skills training for preadolescent children with autism spectrum disorders: generalization of skills by training parents and teachers? (Q33907228) (← links)
- Whole exome sequencing reveals inherited and de novo variants in autism spectrum disorder: a trio study from Saudi families (Q33916084) (← links)
- A CTNNA3 compound heterozygous deletion implicates a role for αT-catenin in susceptibility to autism spectrum disorder (Q33925250) (← links)
- Gene × Environment interactions in autism spectrum disorders: role of epigenetic mechanisms. (Q33997473) (← links)
- Family-based clinical associations and functional characterization of the serotonin 2A receptor gene (HTR2A) in autism spectrum disorder (Q34052727) (← links)
- Most genetic risk for autism resides with common variation (Q34063817) (← links)