Interleukin-18 induces rheumatoid arthritis synovial fibroblast CXC chemokine production through NFkappaB activation (Q24291791)

Human chemokines: an update

GOA release 2020-03-11

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J C Morel

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C C Park

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P Kumar

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A E Koch

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4

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language of work or name

English

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publication date

October 2001

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published in

Laboratory Investigation

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volume

81

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issue

10

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page(s)

1371-83

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A proinflammatory role for IL-18 in rheumatoid arthritis

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Combined Effects of IL-12 and IL-18 on the Induction of Collagen-Induced Arthritis

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Human keratinocytes constitutively express interleukin-18 and secrete biologically active interleukin-18 after treatment with pro-inflammatory mediators and dinitrochlorobenzene.

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Interleukin-18 (IFNgamma-inducing factor) induces IL-8 and IL-1beta via TNFalpha production from non-CD14+ human blood mononuclear cells

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Manipulation of distinct NFkappaB proteins alters interleukin-1beta-induced human rheumatoid synovial fibroblast prostaglandin E2 formation

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Effects of Th1 and Th2 cytokines on cytokine production and ICAM-1 expression on synovial fibroblasts

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Interleukin 12 acts directly on CD4+ T cells to enhance priming for interferon gamma production and diminishes interleukin 4 inhibition of such priming

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Interleukin 18 stimulates HIV type 1 in monocytic cells

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Thrombin receptor mediated signals induce expressions of interleukin 6 and granulocyte colony stimulating factor via NF-kappa B activation in synovial fibroblasts

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Interleukin-18 activates NF-kappaB in murine T helper type 1 cells

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Opposite effects of interleukin-13 and interleukin-12 on the release of inflammatory cytokines, cytokine inhibitors and prostaglandin E from synovial fibroblasts and blood mononuclear cells

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