High concentrations of lipopolysaccharide-binding protein in serum of patients with severe sepsis or septic shock inhibit the lipopolysaccharide response in human monocytes (Q24291979)

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English	High concentrations of lipopolysaccharide-binding protein in serum of patients with severe sepsis or septic shock inhibit the lipopolysaccharide response in human monocytes	scientific article

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scholarly article

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High concentrations of lipopolysaccharide-binding protein in serum of patients with severe sepsis or septic shock inhibit the lipopolysaccharide response in human monocytes (English)

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lipopolysaccharide

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Lipopolysaccharide binding protein

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GOA release 2020-03-11

negative regulation of tumor necrosis factor production

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GOA release 2020-03-11

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based on heuristic

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author name string

J Zweigner

1

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H J Gramm

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O C Singer

3

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K Wegscheider

4

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R R Schumann

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language of work or name

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publication date

15 December 2001

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98

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13

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3800-8

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Changes in polymorphonuclear leukocyte surface and plasma bactericidal/permeability-increasing protein and plasma lipopolysaccharide binding protein during endotoxemia or sepsis

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Incidence, risk factors, and outcome of severe sepsis and septic shock in adults. A multicenter prospective study in intensive care units. French ICU Group for Severe Sepsis

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Binding of lipopolysaccharide (LPS) to CHO cells does not correlate with LPS-induced NF-kappaB activation

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The SOFA (Sepsis-related Organ Failure Assessment) score to describe organ dysfunction/failure

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The epidemiology of the systemic inflammatory response

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Correlation between endotoxin-neutralizing capacity of human plasma as tested by the limulus-amebocyte-lysate-test and plasma protein levels

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CDC definitions for nosocomial infections, 1988

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Recognition of gram-negative bacteria and endotoxin by the innate immune system

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Lipopolysaccharide-binding protein is required to combat a murine gram-negative bacterial infection

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A CD14-independent LPS receptor cluster

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Monocyte deactivation in septic patients: restoration by IFN-gamma treatment

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Treating patients with severe sepsis

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Acute-phase proteins and other systemic responses to inflammation

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Innate immunity

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In vivo protection against endotoxin by plasma high density lipoprotein

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Lipopolysaccharide activation of human endothelial and epithelial cells is mediated by lipopolysaccharide-binding protein and soluble CD14

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Mode of action of anti-lipopolysaccharide-binding protein antibodies for prevention of endotoxemic shock in mice.

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Lipopolysaccharide (LPS) signal transduction and clearance. Dual roles for LPS binding protein and membrane CD14.

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Neutralization and transfer of lipopolysaccharide by phospholipid transfer protein

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Soluble CD14 participates in the response of cells to lipopolysaccharide

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Lipopolysaccharide (LPS)-binding protein is carried on lipoproteins and acts as a cofactor in the neutralization of LPS

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Soluble CD14 acts as a shuttle in the neutralization of lipopolysaccharide (LPS) by LPS-binding protein and reconstituted high density lipoprotein

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Antiinflammatory effects of reconstituted high-density lipoprotein during human endotoxemia

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Targeted deletion of the lipopolysaccharide (LPS)-binding protein gene leads to profound suppression of LPS responses ex vivo, whereas in vivo responses remain intact

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Relationship between plasma levels of lipopolysaccharide (LPS) and LPS-binding protein in patients with severe sepsis and septic shock

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Competition between bactericidal/permeability-increasing protein and lipopolysaccharide-binding protein for lipopolysaccharide binding to monocytes

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Lipopolysaccharide (LPS)-Binding Protein In Human Serum Determines The Tumor Necrosis Factor Response Of Monocytes To LPS

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Lipopolysaccharide toxicity-regulating proteins in bacteremia

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American College of Chest Physicians/Society of Critical Care Medicine Consensus Conference

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Low lipid concentrations in critical illness: implications for preventing and treating endotoxemia

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The sirens' songs of confirmatory sepsis trials: selection bias and sampling error.

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Consensus conference definitions for sepsis, septic shock, acute lung injury, and acute respiratory distress syndrome: time for a reevaluation

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CD14, a receptor for complexes of lipopolysaccharide (LPS) and LPS binding protein

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Structure and function of lipopolysaccharide binding protein

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Defective LPS Signaling in C3H/HeJ and C57BL/10ScCr Mice: Mutations in Tlr4 Gene

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Serum amyloid P component prevents high-density lipoprotein-mediated neutralization of lipopolysaccharide.

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The lipopolysaccharide-binding protein is a secretory class 1 acute-phase protein whose gene is transcriptionally activated by APRF/STAT/3 and other cytokine-inducible nuclear proteins

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Production of the acute-phase protein lipopolysaccharide-binding protein by respiratory type II epithelial cells: implications for local defense to bacterial endotoxins

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Secretory products of macrophages

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Divergent response to LPS and bacteria in CD14-deficient murine macrophages.

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10.1182/BLOOD.V98.13.3800

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