Defective axonal transport in a transgenic mouse model of amyotrophic lateral sclerosis (Q24306886)

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English	Defective axonal transport in a transgenic mouse model of amyotrophic lateral sclerosis	scientific article

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scholarly article

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title

Defective axonal transport in a transgenic mouse model of amyotrophic lateral sclerosis (English)

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main subject

amyotrophic lateral sclerosis

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Neurofilament heavy

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stated in

GOA release 2020-03-11

regulation of organelle transport along microtubule

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stated in

GOA release 2020-03-11

author name string

J F Collard

series ordinal

1

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F Côté

series ordinal

2

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J P Julien

series ordinal

3

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language of work or name

English

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publication date

4 May 1995

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published in

Nature

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volume

375

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issue

6526

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page(s)

61-4

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cites work

Progressive neuronopathy in transgenic mice expressing the human neurofilament heavy gene: a mouse model of amyotrophic lateral sclerosis

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stated in

Crossref

reference URL

https://api.crossref.org/works/10.1038%2F375061A0

retrieved

7 January 2021

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inferred from DOI database lookup

Increased expression of neurofilament subunit NF-L produces morphological alterations that resemble the pathology of human motor neuron disease

1 reference

stated in

Crossref

reference URL

https://api.crossref.org/works/10.1038%2F375061A0

retrieved

7 January 2021

based on heuristic

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A mutant neurofilament subunit causes massive, selective motor neuron death: implications for the pathogenesis of human motor neuron disease

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stated in

Crossref

reference URL

https://api.crossref.org/works/10.1038%2F375061A0

retrieved

7 January 2021

based on heuristic

inferred from DOI database lookup

Variants of the heavy neurofilament subunit are associated with the development of amyotrophic lateral sclerosis

1 reference

stated in

Crossref

reference URL

https://api.crossref.org/works/10.1038%2F375061A0

retrieved

7 January 2021

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The slow component of axonal transport. Identification of major structural polypeptides of the axon and their generality among mammalian neurons

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stated in

Crossref

reference URL

https://api.crossref.org/works/10.1038%2F375061A0

retrieved

7 January 2021

based on heuristic

inferred from DOI database lookup

Neurofibrillary axonal swellings and amyotrophic lateral sclerosis

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stated in

Crossref

reference URL

https://api.crossref.org/works/10.1038%2F375061A0

retrieved

7 January 2021

based on heuristic

inferred from DOI database lookup

Slow Axonal Transport of Neurofilament Proteins: Impairment of β,β′-Iminodipropionitrile Administration

1 reference

stated in

Crossref

reference URL

https://api.crossref.org/works/10.1038%2F375061A0

retrieved

7 January 2021

based on heuristic

inferred from DOI database lookup

Axonal transport in neurological disease

1 reference

stated in

Crossref

reference URL

https://api.crossref.org/works/10.1038%2F375061A0

retrieved

7 January 2021

based on heuristic

inferred from DOI database lookup

Cytotypic differences in the protein composition of the axonally transported cytoskeleton in mammalian neurons

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Crossref

reference URL

https://api.crossref.org/works/10.1038%2F375061A0

retrieved

7 January 2021

based on heuristic

inferred from DOI database lookup

Retardation in the slow axonal transport of cytoskeletal elements during maturation and aging

1 reference

stated in

Crossref

reference URL

https://api.crossref.org/works/10.1038%2F375061A0

retrieved

7 January 2021

based on heuristic

inferred from DOI database lookup

Motor neuron degeneration in mice that express a human Cu,Zn superoxide dismutase mutation

1 reference

stated in

Crossref

reference URL

https://api.crossref.org/works/10.1038%2F375061A0

retrieved

7 January 2021

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inferred from DOI database lookup

Identifiers

DOI

10.1038/375061A0

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stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

3544472

Dimensions Publication ID

1015455449

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OpenCitations bibliographic resource ID

3544472

1 reference

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

3544472

PubMed publication ID

7536898

1 reference

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

3544472

 

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