Polypeptide GalNAc-transferase T3 and familial tumoral calcinosis. Secretion of fibroblast growth factor 23 requires O-glycosylation (Q24321247)

Polypeptide N-acetylgalactosaminyltransferase 3

GOA release 2020-03-11

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GOA release 2020-03-11

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Bettina Lorenz-Depiereux

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object named as

Bettina Lorenz-Depiereux

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Tim M Strom

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object named as

Tim M Strom

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author name string

Kentaro Kato

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Charlotte Jeanneau

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Mads Agervig Tarp

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Anna Benet-Pagès

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Bettina Lorenz-Depiereux

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Eric Paul Bennett

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Ulla Mandel

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Henrik Clausen

Journal of Biological Chemistry

9

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language of work or name

English

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publication date

7 July 2006

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published in

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volume

281

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issue

27

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page(s)

18370-7

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cites work

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Why are proteins O-glycosylated?

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Autosomal dominant hypophosphataemic rickets is associated with mutations in FGF23

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Mutations in GALNT3, encoding a protein involved in O-linked glycosylation, cause familial tumoral calcinosis

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Cloning, expression and gene organization of a human Neu5Ac alpha 2-3Gal beta 1-3GalNAc alpha 2,6-sialyltransferase: hST6GalNAcIV

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Autosomal-dominant hypophosphatemic rickets (ADHR) mutations stabilize FGF-23

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Cloning and characterization of FGF23 as a causative factor of tumor-induced osteomalacia

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T-cell-specific deletion of a polypeptide N-acetylgalactosaminyl-transferase gene by site-directed recombination

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cDNA cloning and expression of a novel human UDP-N-acetyl-alpha-D-galactosamine. Polypeptide N-acetylgalactosaminyltransferase, GalNAc-t3

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Cloning and characterization of a close homologue of human UDP-N-acetyl-alpha-D-galactosamine:Polypeptide N-acetylgalactosaminyltransferase-T3, designated GalNAc-T6. Evidence for genetic but not functional redundancy

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Characteristics and structural requirements of apical sorting of the rat growth hormone through the O-glycosylated stalk region of intestinal sucrase-isomaltase

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Cloning and expression of human core 1 beta1,3-galactosyltransferase

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Mucin-like domain of enteropeptidase directs apical targeting in Madin-Darby canine kidney cells

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Molecular cloning and characterization of a novel UDP-GlcNAc:GalNAc-peptide beta1,3-N-acetylglucosaminyltransferase (beta 3Gn-T6), an enzyme synthesizing the core 3 structure of O-glycans

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Cloning and characterization of a new human UDP-N-acetyl-alpha-D-galactosamine:polypeptide N-acetylgalactosaminyltransferase, designated pp-GalNAc-T13, that is specifically expressed in neurons and synthesizes GalNAc alpha-serine/threonine antigen

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Furin directly cleaves proMMP-2 in the trans-Golgi network resulting in a nonfunctioning proteinase

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Proprotein convertases [corrected] are responsible for proteolysis and inactivation of endothelial lipase

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Three types of low density lipoprotein receptor-deficient mutant have pleiotropic defects in the synthesis of N-linked, O-linked, and lipid-linked carbohydrate chains

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The O-glycosylated stalk domain is required for apical sorting of neurotrophin receptors in polarized MDCK cells

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Expression of polypeptide GalNAc-transferases in stratified epithelia and squamous cell carcinomas: immunohistological evaluation using monoclonal antibodies to three members of the GalNAc-transferase family

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Chemoenzymatically synthesized multimeric Tn/STn MUC1 glycopeptides elicit cancer-specific anti-MUC1 antibody responses and override tolerance.

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An FGF23 missense mutation causes familial tumoral calcinosis with hyperphosphatemia.

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7 January 2021

Molecular cloning and genomic analysis of mouse GalNAc alpha2, 6-sialyltransferase (ST6GalNAc I)

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Abnormal intracellular sorting of O-linked carbohydrate-deficient interleukin-2 receptors

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Familial tumoral calcinosis and testicular microlithiasis associated with a new mutation of GALNT3 in a white family.

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Role of the human ST6GalNAc-I and ST6GalNAc-II in the synthesis of the cancer-associated sialyl-Tn antigen

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Mutant FGF-23 responsible for autosomal dominant hypophosphatemic rickets is resistant to proteolytic cleavage and causes hypophosphatemia in vivo.

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Fibroblast growth factor-23 mutants causing familial tumoral calcinosis are differentially processed.

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The roles of specific genes implicated as circulating factors involved in normal and disordered phosphate homeostasis: frizzled related protein-4, matrix extracellular phosphoglycoprotein, and fibroblast growth factor 23.

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A novel GALNT3 mutation in a pseudoautosomal dominant form of tumoral calcinosis: evidence that the disorder is autosomal recessive

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A novel mutation in fibroblast growth factor 23 gene as a cause of tumoral calcinosis

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O-glycosylation is essential for intracellular targeting of synaptotagmins I and II in non-neuronal specialized secretory cells.

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UDP-N-acetyl-D-galactosamine: polypeptide N-acetylgalactosaminyltransferase-6 as a new immunohistochemical breast cancer marker.

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Immunohistochemical detection of FGF-23 protein in tumors that cause oncogenic osteomalacia

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Identifiers

DOI

10.1074/JBC.M602469200

1 reference

Consolidated OpenCitations Corpus – April 2017

1 reference

Consolidated OpenCitations Corpus – April 2017

PubMed publication ID

16638743

1 reference

Consolidated OpenCitations Corpus – April 2017