Voltage-dependent potentiation of L-type Ca2+ channels in skeletal muscle cells requires anchored cAMP-dependent protein kinase (Q24561617)

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English	Voltage-dependent potentiation of L-type Ca2+ channels in skeletal muscle cells requires anchored cAMP-dependent protein kinase	scientific article

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instance of

scholarly article

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title

Voltage-dependent potentiation of L-type Ca2+ channels in skeletal muscle cells requires anchored cAMP-dependent protein kinase (English)

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main subject

skeletal muscle cell

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based on heuristic

inferred from title

voltage

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author name string

B D Johnson

series ordinal

1

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T Scheuer

series ordinal

2

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W A Catterall

series ordinal

3

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language of work or name

English

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publication date

22 November 1994

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published in

Proceedings of the National Academy of Sciences of the United States of America

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volume

91

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issue

24

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page(s)

11492-6

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cites work

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Purified skeletal muscle 1,4-dihydropyridine receptor forms phosphorylation-dependent oligomeric calcium channels in planar bilayers.

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Voltage sensor of excitation-contraction coupling in skeletal muscle

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Ca2+ and activation mechanisms in skeletal muscle

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Excitation-contraction coupling in vertebrate skeletal muscle: a tale of two calcium channels

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Co-localization of the dihydropyridine receptor and the cyclic AMP-binding subunit of an intrinsic protein kinase to the junctional membrane of the transverse tubules of skeletal muscle

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Novel mechanism of voltage-dependent gating in L-type calcium channels

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Decrease in the size of tetanic responses produced by nitrendipine or by extracellular calcium ion removal without blocking twitches or action potentials in skeletal muscle

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Conditional immortalization of normal and dysgenic mouse muscle cells by the SV40 large T antigen under the vimentin promoter control

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Anchoring of protein kinase A is required for modulation of AMPA/kainate receptors on hippocampal neurons

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Dihydropyridine receptors in muscle are voltage-dependent but most are not functional calcium channels

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Co2+, low Ca2+, and verapamil reduce mechanical activity in rat skeletal muscles

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The stimulatory G protein of adenylyl cyclase, Gs, also stimulates dihydropyridine-sensitive Ca2+ channels. Evidence for direct regulation independent of phosphorylation by cAMP-dependent protein kinase or stimulation by a dihydropyridine agonist

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The purified Ca2+ antagonist receptor from skeletal muscle: subunit structure, photoaffinity labeling and endogenous protein kinase activity

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Cardiac-type excitation-contraction coupling in dysgenic skeletal muscle injected with cardiac dihydropyridine receptor cDNA

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Demonstration of the phosphorylation of dihydropyridine-sensitive calcium channels in chick skeletal muscle and the resultant activation of the channels after reconstitution

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Charge movement and calcium currents in skeletal muscle fibers are enhanced by GTP gamma S

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Double-pulse calcium channel current facilitation in adult rat sympathetic neurones

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Voltage-dependent phosphorylation may recruit Ca2+ current facilitation in chromaff in cells

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Voltage-dependent potentiation of L-type Ca2+ channels due to phosphorylation by cAMP-dependent protein kinase

1 reference

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PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/7972090

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Voltage-dependent potentiation of the slow inward current in frog atrium

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Identifiers

DOI

10.1073/PNAS.91.24.11492

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ADS bibcode

1994PNAS...9111492J

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PMC publication ID

45257

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PubMed publication ID

7972090

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ResearchGate publication ID

15227330

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