Factor I is required for the development of membranoproliferative glomerulonephritis in factor H-deficient mice (Q24651118)

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Factor I is required for the development of membranoproliferative glomerulonephritis in factor H–deficient mice

Language	Label	Description	Also known as
English	Factor I is required for the development of membranoproliferative glomerulonephritis in factor H-deficient mice	scientific article	Factor I is required for the development of membranoproliferative glomerulonephritis in factor H–deficient mice

Statements

scholarly article

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Factor I is required for the development of membranoproliferative glomerulonephritis in factor H-deficient mice (English)

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Complement component factor i

1 reference

GOA release 2020-03-11

glomerulonephritis

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based on heuristic

inferred from title

membranoproliferative glomerulonephritis

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based on heuristic

inferred from title

11

object named as

Marina Botto

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Matthew C Pickering

object named as

Matthew C Pickering

12

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object named as

Mark J Walport

9

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object named as

Steven H Sacks

8

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author name string

Kirsten L Rose

1

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Danielle Paixao-Cavalcante

2

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Jennifer Fish

3

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Anthony P Manderson

4

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Talat H Malik

5

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Anne E Bygrave

6

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Tao Lin

7

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H Terence Cook

10

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Marina Botto

11

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language of work or name

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publication date

February 2008

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Journal of Clinical Investigation

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118

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2

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608-18

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Deletion of Lys224 in regulatory domain 4 of Factor H reveals a novel pathomechanism for dense deposit disease (MPGN II)

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

7 April 2017

Control of the amplification convertase of complement by the plasma protein beta1H

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

7 April 2017

Human complement C3b inactivator: isolation, characterization, and demonstration of an absolute requirement for the serum protein beta1H for cleavage of C3b and C4b in solution

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

7 April 2017

Complement factor I deficiency associated with recurrent meningitis coinciding with menstruation

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

7 April 2017

Complement factor I deficiency in a family with recurrent infections

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

7 April 2017

Hereditary complement factor I deficiency

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

7 April 2017

Dense deposit disease: a variant of membranoproliferative glomerulonephritis

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

7 April 2017

Formation of the initial C3 convertase of the alternative complement pathway. Acquisition of C3b-like activities by spontaneous hydrolysis of the putative thioester in native C3

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

7 April 2017

Primary glomerulonephritis with isolated C3 deposits: a new entity which shares common genetic risk factors with haemolytic uraemic syndrome

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

28 September 2017

The simple design of complement factor H: Looks can be deceiving

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

28 September 2017

Prevention of C5 activation ameliorates spontaneous and experimental glomerulonephritis in factor H-deficient mice

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

28 September 2017

Deficiency of C4 from donor or recipient mouse fails to prevent renal allograft rejection

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

28 September 2017

Human factor H-related protein 5 has cofactor activity, inhibits C3 convertase activity, binds heparin and C-reactive protein, and associates with lipoprotein.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

28 September 2017

Membranoproliferative glomerulonephritis type II (dense deposit disease): an update

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

28 September 2017

The molecular basis for hereditary porcine membranoproliferative glomerulonephritis type II: point mutations in the factor H coding sequence block protein secretion

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

28 September 2017

Systemic lupus erythematosus, complement deficiency, and apoptosis

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

28 September 2017

Control of the complement system.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

28 September 2017

Extensive complement activation in hereditary porcine membranoproliferative glomerulonephritis type II (porcine dense deposit disease)

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

28 September 2017

Hereditary porcine membranoproliferative glomerulonephritis type II is caused by factor H deficiency

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

28 September 2017

Neoantigen of the polymerized ninth component of complement. Characterization of a monoclonal antibody and immunohistochemical localization in renal disease

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

28 September 2017

Metabolic Studies of the Third Component of Complement and the Glycine-Rich Beta Glycoprotein in Patients with Hypocomplementemia

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

28 September 2017

Activation of the alternative pathway of complement by monoclonal lambda light chains in membranoproliferative glomerulonephritis

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

28 September 2017

Properdin: binding to C3b and stabilization of the C3b-dependent C3 convertase

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

28 September 2017

Immunofluorescence studies of dense deposit disease. The presence of railroad tracks and mesangial rings

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

28 September 2017

Complement C3 convertase: Cell surface restriction of β1H control and generation of restriction on neuraminidase-treated cells

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

28 September 2017

Inherited factor H deficiency and collagen type III glomerulopathy

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

30 May 2018

Relation of putative thioester bond in C3 to activation of the alternative pathway and the binding of C3b to biological targets of complement

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

30 May 2018

Reaction mechanism of the alternative pathway of complement fixation

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

30 May 2018

Post-translational processing of the murine third component of complement

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

30 May 2018

Hypocomplementemic glomerulonephritis in an infant and mother. Evidence for an abnormal form of C3.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

30 May 2018

Complement factor I deficiency associated with recurrent infections, vasculitis and immune complex glomerulonephritis

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

28 November 2018

Ontogeny of complement regulatory proteins - concentrations of factor h, factor I, c4b-binding protein, properdin and vitronectin in healthy children of different ages and in adults

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

28 November 2018

Uncontrolled C3 activation causes membranoproliferative glomerulonephritis in mice deficient in complement factor H

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

28 November 2018

Accelerated nephrotoxic nephritis is exacerbated in C1q-deficient mice

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

28 November 2018

Deficiency of human complement factor I associated with lowered factor H.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

28 November 2018

Heterozygous and homozygous factor H deficiency states in a Dutch family

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2200299

28 November 2018

Glomerulonephritis in a patient with complement factor I deficiency

1 reference

https://pubmed.ncbi.nlm.nih.gov/18202746

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Distinct and separable roles of the complement system in factor H-deficient bone marrow chimeric mice with immune complex disease

1 reference

https://pubmed.ncbi.nlm.nih.gov/18202746

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

The physiological breakdown of the third component of human complement

1 reference

https://pubmed.ncbi.nlm.nih.gov/18202746

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Hypocomplementaemia due to a genetic deficiency of beta 1H globulin

1 reference

https://pubmed.ncbi.nlm.nih.gov/18202746

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

An inherited defect in the C3 convertase, C3b,Bb, associated with glomerulonephritis

1 reference

https://pubmed.ncbi.nlm.nih.gov/18202746

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Composition of nephritic factor-generated glomerular deposits in membranoproliferative glomerulonephritis type 2

1 reference

https://pubmed.ncbi.nlm.nih.gov/18202746

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Inherited complete factor I deficiency associated with systemic lupus erythematosus, higher susceptibility to infection and low levels of factor H

1 reference

https://pubmed.ncbi.nlm.nih.gov/18202746

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Regulatory proteins for the activated third and fourth components of complement (C3b and C4b) in mice. I. Isolation and characterization of factor H: the serum cofactor for the C3b/C4b inactivator (factor I)

1 reference

https://pubmed.ncbi.nlm.nih.gov/18202746

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Effect of nephritic factor on C3 and on the terminal pathway of complement in vivo and in vitro

1 reference

https://pubmed.ncbi.nlm.nih.gov/18202746

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

H deficiency in two brothers with atypical dense intramembranous deposit disease

1 reference

https://pubmed.ncbi.nlm.nih.gov/18202746

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Combined homozygous factor H and heterozygous C2 deficiency in an Italian family

1 reference

https://pubmed.ncbi.nlm.nih.gov/18202746

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Hereditary, complete deficiency of complement factor H associated with recurrent meningococcal disease

1 reference

https://pubmed.ncbi.nlm.nih.gov/18202746

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Secondary membranoproliferative glomerulonephritis

1 reference

https://pubmed.ncbi.nlm.nih.gov/18202746

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1172/JCI32525

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