Toll-like receptor (TLR) 2 and TLR5, but not TLR4, are required for Helicobacter pylori-induced NF-kappa B activation and chemokine expression by epithelial cells (Q28179206)

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scientific article (publication date: 29 August 2003)

Language	Label	Description	Also known as
English	Toll-like receptor (TLR) 2 and TLR5, but not TLR4, are required for Helicobacter pylori-induced NF-kappa B activation and chemokine expression by epithelial cells	scientific article (publication date: 29 August 2003)

Statements

scholarly article

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Toll-like receptor (TLR) 2 and TLR5, but not TLR4, are required for Helicobacter pylori-induced NF-kappa B activation and chemokine expression by epithelial cells (English)

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Helicobacter pylori

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toll-like receptor

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1 reference

based on heuristic

inferred from title

author name string

Michael F Smith

1

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Anastasia Mitchell

2

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Guolian Li

3

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Song Ding

4

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Ann Marie Fitzmaurice

5

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Kieran Ryan

6

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Sheila Crowe

7

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Joanna B Goldberg

8

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language of work or name

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publication date

29 August 2003

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Journal of Biological Chemistry

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278

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35

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32552-60

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Helicobacter pylori activates mitogen-activated protein kinase cascades and induces expression of the proto-oncogenes c-fos and c-jun.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M305536200

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Helicobacter pylori infection activates NF-kappa B in gastric epithelial cells

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M305536200

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Expression of interleukin 8 and CD54 by human gastric epithelium after Helicobacter pylori infection in vitro

1 reference

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The Toll receptor family and microbial recognition

1 reference

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Differential roles of TLR2 and TLR4 in recognition of gram-negative and gram-positive bacterial cell wall components

1 reference

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Toll-like receptor-2 mediates mycobacteria-induced proinflammatory signaling in macrophages.

1 reference

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Defective LPS Signaling in C3H/HeJ and C57BL/10ScCr Mice: Mutations in Tlr4 Gene

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A Toll-like receptor recognizes bacterial DNA

1 reference

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Toll receptors: a central element in innate immune responses

1 reference

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Summary and comparison of the signaling mechanisms of the Toll/interleukin-1 receptor family

1 reference

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Type I Helicobacter pylori lipopolysaccharide stimulates toll-like receptor 4 and activates mitogen oxidase 1 in gastric pit cells

1 reference

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Cutting edge: repurification of lipopolysaccharide eliminates signaling through both human and murine toll-like receptor 2.

1 reference

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Toll-like receptor 2 and 4 (TLR2 and TLR4) agonists differentially regulate secretory interleukin-1 receptor antagonist gene expression in macrophages.

1 reference

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Structural characterization of the lipid A component of Helicobacter pylori rough- and smooth-form lipopolysaccharides

1 reference

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Signaling by toll-like receptor 2 and 4 agonists results in differential gene expression in murine macrophages.

1 reference

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Human Toll-like receptor 4 recognizes host-specific LPS modifications.

1 reference

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Cell-free pool of CD14 mediates activation of transcription factor NF-kappa B by lipopolysaccharide in human endothelial cells

1 reference

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CD14 enhances cellular responses to endotoxin without imparting ligand-specific recognition

1 reference

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Helicobacter pylori uses motility for initial colonization and to attain robust infection

1 reference

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TLR4, but not TLR2, mediates IFN-beta-induced STAT1alpha/beta-dependent gene expression in macrophages.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M305536200

21 January 2018

Cag pathogenicity island-specific responses of gastric epithelial cells to Helicobacter pylori infection

1 reference

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21 January 2018

SHP-2 tyrosine phosphatase as an intracellular target of Helicobacter pylori CagA protein

1 reference

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21 January 2018

Identifiers

10.1074/JBC.M305536200

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Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

OpenCitations bibliographic resource ID

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Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

PubMed publication ID

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

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