Oncogenic forms of NOTCH1 lacking either the primary binding site for RBP-Jkappa or nuclear localization sequences retain the ability to associate with RBP-Jkappa and activate transcription (Q28235557)

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scientific article (publication date: 25 April 1997)

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English	Oncogenic forms of NOTCH1 lacking either the primary binding site for RBP-Jkappa or nuclear localization sequences retain the ability to associate with RBP-Jkappa and activate transcription	scientific article (publication date: 25 April 1997)

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scholarly article

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Oncogenic forms of NOTCH1 lacking either the primary binding site for RBP-Jkappa or nuclear localization sequences retain the ability to associate with RBP-Jkappa and activate transcription (English)

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Elliott D. Kieff

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object named as

E Kieff

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author name string

J C Aster

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E S Robertson

2

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R P Hasserjian

3

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J R Turner

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J Sklar

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language of work or name

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publication date

25 April 1997

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Journal of Biological Chemistry

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272

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17

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11336-43

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Analysis of DNA surrounding the breakpoints of chromosomal translocations involving the beta T cell receptor gene in human lymphoblastic neoplasms

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TAN-1, the human homolog of the Drosophila Notch gene, is broken by chromosomal translocations in T lymphoblastic neoplasms

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Exclusive development of T cell neoplasms in mice transplanted with bone marrow expressing activated Notch alleles

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Specific truncations of Drosophila Notch define dominant activated and dominant negative forms of the receptor.

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An activated Notch receptor blocks cell-fate commitment in the developing Drosophila eye.

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Expression of an extracellular deletion of Xotch diverts cell fate in Xenopus embryos

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Jagged: A mammalian ligand that activates notch1

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A protein binding to the J kappa recombination sequence of immunoglobulin genes contains a sequence related to the integrase motif

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The recombination signal sequence-binding protein RBP-2N functions as a transcriptional repressor

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Mediation of Epstein-Barr virus EBNA2 transactivation by recombination signal-binding protein J kappa

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The Epstein-Barr virus nuclear antigen 2 transactivator is directed to response elements by the J kappa recombination signal binding protein

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glp-1 and lin-12, genes implicated in distinct cell-cell interactions in C. elegans, encode similar transmembrane proteins

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The suppressor of hairless protein participates in notch receptor signaling

1 reference

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Suppressor of hairless directly activates transcription of enhancer of split complex genes in response to Notch receptor activity

1 reference

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The neurogenic suppressor of hairless DNA-binding protein mediates the transcriptional activation of the enhancer of split complex genes triggered by Notch signaling.

1 reference

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Signalling downstream of activated mammalian Notch

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Truncated mammalian Notch1 activates CBF1/RBPJk-repressed genes by a mechanism resembling that of Epstein-Barr virus EBNA2

1 reference

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Physical interaction between a novel domain of the receptor Notch and the transcription factor RBP-J kappa/Su(H)

1 reference

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Production of high-titer helper-free retroviruses by transient transfection

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Human homologs of a Drosophila Enhancer of split gene product define a novel family of nuclear proteins

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Human von Willebrand factor (vWF): isolation of complementary DNA (cDNA) clones and chromosomal localization

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A novel genetic system to detect protein-protein interactions

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Advanced mammalian gene transfer: high titre retroviral vectors with multiple drug selection markers and a complementary helper-free packaging cell line

1 reference

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The Epstein-Barr virus immortalizing protein EBNA-2 is targeted to DNA by a cellular enhancer-binding protein

1 reference

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Signal transduction by activated mNotch: importance of proteolytic processing and its regulation by the extracellular domain.

1 reference

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T cell leukemia-associated human Notch/translocation-associated Notch homologue has I kappa B-like activity and physically interacts with nuclear factor-kappa B proteins in T cells

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Epstein-Barr virus nuclear protein 2 is a key determinant of lymphocyte transformation

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Masking of the CBF1/RBPJ kappa transcriptional repression domain by Epstein-Barr virus EBNA2.

1 reference

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Antineurogenic phenotypes induced by truncated Notch proteins indicate a role in signal transduction and may point to a novel function for Notch in nuclei

1 reference

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Intrinsic activity of the Lin-12 and Notch intracellular domains in vivo

1 reference

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Notch signaling

1 reference

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21 January 2018

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