Cloning of a bcl-2 homologue by interaction with adenovirus E1B 19K (Q28299861)

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J H White

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I Martinou

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T Raven

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K T Pun

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C J Grinham

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J C Martinou

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R Brown

Regulation of Apoptosis by the Transforming Genes of the DNA Tumor Virus Adenovirus

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language of work or name

English

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publication date

20 April 1995

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published in

Nature

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volume

374

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issue

6524

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page(s)

731-3

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cites work

1 reference

7 January 2021

Activation of Epstein-Barr virus latent genes protects human B cells from death by apoptosis

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Epstein-Barr virus-coded BHRF1 protein, a viral homologue of Bcl-2, protects human B cells from programmed cell death

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An evolutionary perspective on apoptosis.

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Control of programmed cell death by the baculovirus genes p35 and iap.

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The adenovirus E1A proteins induce apoptosis, which is inhibited by the E1B 19-kDa and Bcl-2 proteins

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The E1b oncogene of adenovirus confers cellular resistance to cytotoxicity of tumor necrosis factor and monoclonal anti-Fas antibody

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Viral proteins E1B19K and p35 protect sympathetic neurons from cell death induced by NGF deprivation

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BH1 and BH2 domains of Bcl-2 are required for inhibition of apoptosis and heterodimerization with Bax.

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Bcl-2 and the regulation of programmed cell death

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Cloning of the chromosome breakpoint of neoplastic B cells with the t(14;18) chromosome translocation

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MCL1, a gene expressed in programmed myeloid cell differentiation, has sequence similarity to BCL2

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C. elegans cell survival gene ced-9 encodes a functional homolog of the mammalian proto-oncogene bcl-2.

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Molecular regulation of apoptosis: genetic controls on cell death.

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bcl-x, a bcl-2-related gene that functions as a dominant regulator of apoptotic cell death

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The retinoblastoma protein associates with the protein phosphatase type 1 catalytic subunit

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A novel genetic system to detect protein-protein interactions

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Specific disruption of intermediate filaments and the nuclear lamina by the 19-kDa product of the adenovirus E1B oncogene

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Prevention of programmed cell death of sympathetic neurons by the bcl-2 proto-oncogene

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Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death

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Identification of immunosuppressant-induced apoptosis in a murine B-cell line and its prevention by bcl-x but not bcl-2.

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Wild-type p53 mediates apoptosis by E1A, which is inhibited by E1B.

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Bcl-2 blocks p53-dependent apoptosis

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Single-step purification of shuttle vectors from yeast for high frequency back-transformation into E. coli

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