Opposing roles of netrin-1 and the dependence receptor DCC in cancer cell invasion, tumor growth and metastasis (Q28291163)

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O De Wever

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E Bruyneel

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R J Rooney

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C Gespach

Netrin-1 and its receptors in tumorigenesis

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language of work or name

English

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publication date

16 August 2007

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published in

Oncogene

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volume

26

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issue

38

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page(s)

5615-25

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cites work

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Netrin-1-mediated axon outgrowth and cAMP production requires interaction with adenosine A2b receptor

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Netrin-1-mediated axon outgrowth requires deleted in colorectal cancer-dependent MAPK activation

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Ras-associated protein-1 regulates extracellular signal-regulated kinase activation and migration in melanoma cells: two processes important to melanoma tumorigenesis and metastasis.

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Brain meets pancreas: netrin, an axon guidance molecule, controls epithelial cell migration

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Cortactin underpins CD44-promoted invasion and adhesion of breast cancer cells to bone marrow endothelial cells

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A ligand-gated association between cytoplasmic domains of UNC5 and DCC family receptors converts netrin-induced growth cone attraction to repulsion

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Mammalian homologs of seven in absentia regulate DCC via the ubiquitin-proteasome pathway

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Epac mediates a cAMP-to-PKC signaling in inflammatory pain: an isolectin B4(+) neuron-specific mechanism.

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Netrin induces down-regulation of its receptor, Deleted in Colorectal Cancer, through the ubiquitin-proteasome pathway in the embryonic cortical neuron

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Inhibition by platelet-activating factor of Src- and hepatocyte growth factor-dependent invasiveness of intestinal and kidney epithelial cells. Phosphatidylinositol 3'-kinase is a critical mediator of tumor invasion

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Does integrin-mediated cell death confer tissue tropism in metastasis?

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Netrin-1 controls colorectal tumorigenesis by regulating apoptosis

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Commutators of PAR-1 signaling in cancer cell invasion reveal an essential role of the Rho-Rho kinase axis and tumor microenvironment

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The netrins define a family of axon outgrowth-promoting proteins homologous to C. elegans UNC-6.

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The netrin-1 receptor DCC promotes filopodia formation and cell spreading by activating Cdc42 and Rac1.

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Adenosine downregulates DPPIV on HT-29 colon cancer cells by stimulating protein tyrosine phosphatase(s) and reducing ERK1/2 activity via a novel pathway

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Netrins promote developmental and therapeutic angiogenesis.

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