Hypoxia and acidosis activate cardiac myocyte death through the Bcl-2 family protein BNIP3. (Q28577311)

5 July 2017

title

Hypoxia and acidosis activate cardiac myocyte death through the Bcl-2 family protein BNIP3. (English)

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5 July 2017

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Lori A Kubasiak

1

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5 July 2017

Olga M Hernandez

series ordinal

2

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5 July 2017

Nanette H Bishopric

series ordinal

3

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5 July 2017

Keith A Webster

series ordinal

4

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5 July 2017

language of work or name

English

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publication date

11 September 2002

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5 July 2017

number of pages

6

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Proceedings of the National Academy of Sciences of the United States of America

inferred from page(s)

published in

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5 July 2017

volume

99

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5 July 2017

issue

20

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5 July 2017

page(s)

12825-12830

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BNIP3 and genetic control of necrosis-like cell death through the mitochondrial permeability transition pore

5 July 2017

cites work

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17 March 2017

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BNIP3 heterodimerizes with Bcl-2/Bcl-X(L) and induces cell death independent of a Bcl-2 homology 3 (BH3) domain at both mitochondrial and nonmitochondrial sites

23 March 2017

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7 April 2017

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29 September 2017

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29 September 2017

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Expression of the gene encoding the proapoptotic Nip3 protein is induced by hypoxia

29 September 2017

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29 September 2017

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29 September 2017

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29 September 2017

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3 June 2018

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Intracellular alkalinization suppresses lovastatin-induced apoptosis in HL-60 cells through the inactivation of a pH-dependent endonuclease

3 June 2018

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Adenovirus E1A products suppress myogenic differentiation and inhibit transcription from muscle-specific promoters

3 June 2018

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Molecular regulation of cardiac myocyte adaptations to chronic hypoxia

3 June 2018

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Effect of vacuolar proton ATPase on pHi, Ca2+, and apoptosis in neonatal cardiomyocytes during metabolic inhibition/recovery

3 June 2018

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Apoptotic and necrotic myocyte cell deaths are independent contributing variables of infarct size in rats

27 November 2018

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Induction and nuclear accumulation of fos and jun proto-oncogenes in hypoxic cardiac myocytes.

27 November 2018

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Role of glycolytic products in damage to ischemic myocardium. Dissociation of adenosine triphosphate levels and recovery of function of reperfused ischemic hearts

27 November 2018

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12 December 2020

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12 December 2020

Apoptosis in myocytes in end-stage heart failure

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12 December 2020

Apoptosis in ischemic and reperfused rat myocardium

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12 December 2020

Myocardial cell death and apoptosis in hibernating myocardium

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12 December 2020

Myocyte cell death in the diseased heart

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12 December 2020

"Apoptotic" myocytes in infarct area in rabbit hearts may be oncotic myocytes with DNA fragmentation: analysis by immunogold electron microscopy combined with In situ nick end-labeling

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12 December 2020

Apoptosis in the failing heart

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12 December 2020

Identifiers

DOI

10.1073/PNAS.202474099

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OpenCitations bibliographic resource ID

5 July 2017

0 references

1277647

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

1 reference

5 July 2017

1 reference