Synaptic scaling mediated by glial TNF-alpha (Q29616816)

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scientific article (publication date: 20 April 2006)

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Language	Label	Description	Also known as
English	Synaptic scaling mediated by glial TNF-alpha	scientific article (publication date: 20 April 2006)

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instance of

scholarly article

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title

Synaptic scaling mediated by glial TNF-alpha (English)

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stated in

PubMed

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=16547515

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30 April 2017

main subject

TNF

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glial cell-neuron signaling

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stated in

Gene Ontology release 2020-05-02

Gene Ontology ID

GO:0150098

regulation of synaptic scaling

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stated in

Gene Ontology release 2020-05-02

Gene Ontology ID

GO:0150092

Tumor necrosis factor

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stated in

GOA release 2020-03-11

author name string

Stellwagen D

series ordinal

1

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PubMed

reference URL

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30 April 2017

Malenka RC

series ordinal

2

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PubMed

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30 April 2017

language of work or name

English

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publication date

20 April 2006

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published in

Nature

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PubMed

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volume

440

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PubMed

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issue

7087

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PubMed

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page(s)

1054-9

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cites work

Homeostatic plasticity in the developing nervous system

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Synaptic gain control and homeostasis.

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LTP and LTD

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Activity-dependent modulation of synaptic AMPA receptor accumulation

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Postsynaptic expression of homeostatic plasticity at neocortical synapses.

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Activity deprivation reduces miniature IPSC amplitude by decreasing the number of postsynaptic GABA(A) receptors clustered at neocortical synapses.

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Control of synaptic strength by glial TNFalpha

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Differential regulation of AMPA receptor and GABA receptor trafficking by tumor necrosis factor-alpha

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Rapid redistribution of glutamate receptors contributes to long-term depression in hippocampal cultures

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Immune and inflammatory responses in TNF alpha-deficient mice: a critical requirement for TNF alpha in the formation of primary B cell follicles, follicular dendritic cell networks and germinal centers, and in the maturation of the humoral immune re

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Activity-dependent regulation of dendritic synthesis and trafficking of AMPA receptors

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CXCR4-activated astrocyte glutamate release via TNFalpha: amplification by microglia triggers neurotoxicity

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Crossref

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Activity-dependent scaling of quantal amplitude in neocortical neurons

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Crossref

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A cytotoxicity assay for tumor necrosis factor employing a multiwell fluorescence scanner

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Postsynaptic depolarization scales quantal amplitude in cortical pyramidal neurons.

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GABA itself promotes the developmental switch of neuronal GABAergic responses from excitation to inhibition.

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BDNF has opposite effects on the quantal amplitude of pyramidal neuron and interneuron excitatory synapses

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Rapid spine delivery and redistribution of AMPA receptors after synaptic NMDA receptor activation

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Bidirectional synaptic plasticity regulated by phosphorylation of stargazin-like TARPs.

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Driving AMPA receptors into synapses by LTP and CaMKII: requirement for GluR1 and PDZ domain interaction

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Glial cells and neurotransmission: an inclusive view of synaptic function

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Neuronal activity regulates correlated network properties of spontaneous calcium transients in astrocytes in situ.

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Deriving the glutamate clearance time course from transporter currents in CA1 hippocampal astrocytes: transmitter uptake gets faster during development.

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Crossref

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Synaptic signaling between neurons and glia.

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Thrombospondins are astrocyte-secreted proteins that promote CNS synaptogenesis

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Crossref

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Control of synapse number by glia

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Crossref

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7 January 2021

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Functional requirement for class I MHC in CNS development and plasticity

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Crossref

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A simple method for organotypic cultures of nervous tissue

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Crossref

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7 January 2021

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Identifiers

DOI

10.1038/NATURE04671

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PubMed

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=16547515

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30 April 2017

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

1354669

Dimensions Publication ID

1000381986

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OpenCitations bibliographic resource ID

1354669

1 reference

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

1354669

PubMed publication ID

16547515

2 references

stated in

PubMed

reference URL

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=16547515

retrieved

30 April 2017

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

1354669

ResearchGate publication ID

7230480

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