The anti-inflammatory agents aspirin and salicylate inhibit the activity of I(kappa)B kinase-beta (Q29617188)

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scientific article (publication date: 5 November 1998)

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English	The anti-inflammatory agents aspirin and salicylate inhibit the activity of I(kappa)B kinase-beta	scientific article (publication date: 5 November 1998)

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title

The anti-inflammatory agents aspirin and salicylate inhibit the activity of I(kappa)B kinase-beta (English)

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PubMed

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=9817203

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30 April 2017

main subject

aspirin

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inferred from title

salicylic acid

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author name string

Yin MJ

series ordinal

1

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PubMed

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Yamamoto Y

series ordinal

2

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PubMed

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Gaynor RB

series ordinal

3

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PubMed

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language of work or name

English

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publication date

5 November 1998

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published in

Nature

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PubMed

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volume

396

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issue

6706

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PubMed

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page(s)

77-80

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cites work

NF-kappa B: ten years after

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https://api.crossref.org/works/10.1038%2F23948

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7 January 2021

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The NF-kappa B and I kappa B proteins: new discoveries and insights

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Control of I kappa B-alpha proteolysis by site-specific, signal-induced phosphorylation

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7 January 2021

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Signal-induced site-specific phosphorylation targets I kappa B alpha to the ubiquitin-proteasome pathway

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7 January 2021

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A cytokine-responsive IkappaB kinase that activates the transcription factor NF-kappaB

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Identification and characterization of an IkappaB kinase

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https://api.crossref.org/works/10.1038%2F23948

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7 January 2021

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IKK-1 and IKK-2: cytokine-activated IkappaB kinases essential for NF-kappaB activation

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https://api.crossref.org/works/10.1038%2F23948

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7 January 2021

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The IkappaB kinase complex (IKK) contains two kinase subunits, IKKalpha and IKKbeta, necessary for IkappaB phosphorylation and NF-kappaB activation

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https://api.crossref.org/works/10.1038%2F23948

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7 January 2021

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IkappaB kinase-beta: NF-kappaB activation and complex formation with IkappaB kinase-alpha and NIK

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Crossref

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https://api.crossref.org/works/10.1038%2F23948

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7 January 2021

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MAP3K-related kinase involved in NF-kappaB induction by TNF, CD95 and IL-1

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https://api.crossref.org/works/10.1038%2F23948

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7 January 2021

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HTLV-I Tax protein binds to MEKK1 to stimulate IkappaB kinase activity and NF-kappaB activation

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https://api.crossref.org/works/10.1038%2F23948

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7 January 2021

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Inhibition of prostaglandin synthesis as a mechanism of action for aspirin-like drugs

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https://api.crossref.org/works/10.1038%2F23948

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7 January 2021

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Towards a better aspirin

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7 January 2021

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Inhibition of NF-kappa B by sodium salicylate and aspirin

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7 January 2021

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Neuroprotection by aspirin and sodium salicylate through blockade of NF-kappaB activation.

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7 January 2021

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TRAF2-mediated activation of NF-kappa B by TNF receptor 2 and CD40

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7 January 2021

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Immunosuppression by glucocorticoids: inhibition of NF-kappa B activity through induction of I kappa B synthesis

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7 January 2021

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Role of transcriptional activation of I kappa B alpha in mediation of immunosuppression by glucocorticoids

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7 January 2021

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ERKs: a family of protein-serine/threonine kinases that are activated and tyrosine phosphorylated in response to insulin and NGF

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Reconstitution of mitogen-activated protein kinase phosphorylation cascades in bacteria. Efficient synthesis of active protein kinases

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MEKK1 binds directly to the c-Jun N-terminal kinases/stress-activated protein kinases

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The stress-activated protein kinase subfamily of c-Jun kinases

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7 January 2021

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Selectivity of nonsteroidal antiinflammatory drugs as inhibitors of constitutive and inducible cyclooxygenase

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7 January 2021

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Aspirin-like molecules that covalently inactivate cyclooxygenase-2.

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7 January 2021

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Identifiers

DOI

10.1038/23948

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PubMed

reference URL

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=9817203

retrieved

30 April 2017

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

932641

Dimensions Publication ID

1049122030

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OpenCitations bibliographic resource ID

932641

1 reference

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

932641

PubMed publication ID

9817203

2 references

stated in

PubMed

reference URL

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=9817203

retrieved

30 April 2017

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

932641

 

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