Reactive oxygen species have a causal role in multiple forms of insulin resistance (Q29619831)

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reactive oxygen species

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3

Lander ES

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Houstis N

1

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Rosen ED

series ordinal

2

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language of work or name

1 reference

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publication date

13 April 2006

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volume

440

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issue

7086

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page(s)

944-8

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Tumor necrosis factor-alpha- and hyperglycemia-induced insulin resistance. Evidence for different mechanisms and different effects on insulin signaling

30 April 2017

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Prolonged oxidative stress impairs insulin-induced GLUT4 translocation in 3T3-L1 adipocytes

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R-alpha-lipoic acid action on cell redox status, the insulin receptor, and glucose uptake in 3T3-L1 adipocytes

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Reactive oxygen species promote TNFalpha-induced death and sustained JNK activation by inhibiting MAP kinase phosphatases

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A role for sphingolipids in producing the common features of type 2 diabetes, metabolic syndrome X, and Cushing's syndrome

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Ceramide Interaction with the Respiratory Chain of Heart Mitochondria†

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Regulation of insulin-stimulated glucose transporter GLUT4 translocation and Akt kinase activity by ceramide.

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=16612386

Identifiers

2 references

30 April 2017

Consolidated OpenCitations Corpus – April 2017

Dimensions Publication ID

1030208897

0 references

1 reference

Consolidated OpenCitations Corpus – April 2017

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=16612386

PubMed publication ID

2 references

30 April 2017

Consolidated OpenCitations Corpus – April 2017