Induction of activator protein-1 through reactive oxygen species by crystalline silica in JB6 cells (Q23918461)

HELD

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25 September 2023

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15 October 2024

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29 March 2020

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29 March 2020

reactive oxygen species

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8

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Val Vallyathan

9

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Chuanshu Huang

C. Huang

4

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James M. Antonini

7

J. Antonini

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Xianglin Shi

2

X. Shi

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Jenny R. Roberts

6

J. Roberts

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Min Ding

1

M. Ding

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Stephen S. Leonard

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S. Leonard

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author name string

Y. Lu

full work available at URL

3

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language of work or name

English

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publication date

23 March 2001

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http://www.jbc.org/cgi/content/full/276/12/9108

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29 March 2020

Journal of Biological Chemistry

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volume

276

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issue

12

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page(s)

9108-9114

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on focus list of Wikimedia project

Wikimedia–NIOSH collaboration

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Freshly fractured crystalline silica induces activator protein-1 activation through ERKs and p38 MAPK

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AP1/jun function is differentially induced in promotion-sensitive and resistant JB6 cells.

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Transgenic mice demonstrate AP-1 (activator protein-1) transactivation is required for tumor promotion

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Reduction in mitochondrial potential constitutes an early irreversible step of programmed lymphocyte death in vivo.

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Sequential reduction of mitochondrial transmembrane potential and generation of reactive oxygen species in early programmed cell death

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Enhanced Generation of Free Radicals from Phagocytes Induced by Mineral Dusts

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Ultraviolet B-induced activated protein-1 activation does not require epidermal growth factor receptor but is blocked by a dominant negative PKClambda/iota

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